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Battling Losing Natural Testosterone output while on Paxil
- Thread starter enigma79
- Start date
I've been on paxil for seven years and as far as I'm concerned, it saved my life. I use it to treat both major clinical depression and an anxiety disorder, both of which are genetic in my family.
The trick to paxil, and probably all anti-d's, is to use the absolutely smallest dose possible to get the job done. I started at 30mg ED and took forever to get off, but it didn't matter at the time because I had bigger problems to worry about (suicidal ideation).
Once I got better, after lots of therapy and meds, I was able to start cutting down to the point where today, 10mg ED does the trick with virtually no sides. I tried weaning myself off slowly last year over the course of 4 months, and began having problems again, so it looks like I'm gonna be on it for the rest of my life. Not so bad, considering the alternatives.
I do, however, believe that anti-d's should ONLY be used for physiologically based forms of depression, not situational. If you're bummed because your gf dumped you and is now getting pounded by your best friend, that is not a reason to jump on the meds bandwagon. Take a deep breath, talk to a professional, have a beer or a bong, get through it day-by-day, but don't rely on drugs to do all your work for you. Paxil will not stop you from feeling sadness over the loss of a loved one or general disappointments in life. Docs should stop handing it out like candy.
The trick to paxil, and probably all anti-d's, is to use the absolutely smallest dose possible to get the job done. I started at 30mg ED and took forever to get off, but it didn't matter at the time because I had bigger problems to worry about (suicidal ideation).
Once I got better, after lots of therapy and meds, I was able to start cutting down to the point where today, 10mg ED does the trick with virtually no sides. I tried weaning myself off slowly last year over the course of 4 months, and began having problems again, so it looks like I'm gonna be on it for the rest of my life. Not so bad, considering the alternatives.
I do, however, believe that anti-d's should ONLY be used for physiologically based forms of depression, not situational. If you're bummed because your gf dumped you and is now getting pounded by your best friend, that is not a reason to jump on the meds bandwagon. Take a deep breath, talk to a professional, have a beer or a bong, get through it day-by-day, but don't rely on drugs to do all your work for you. Paxil will not stop you from feeling sadness over the loss of a loved one or general disappointments in life. Docs should stop handing it out like candy.
SCOTTY-TWO-HOTTY
New member
Ever since I started taking Paxil in the Fall of 2002... I've never been able to get my natural Test levels to equalize between cycles. Before that, a short regimen of ancilliaries would bounce me back no prob. I've pretty much been forced to run low-dose HRT between cycles now.
I wouldn't be here without Paxil though... anxiety would have taken my life in short order had I not started on it when I did.
I wouldn't be here without Paxil though... anxiety would have taken my life in short order had I not started on it when I did.
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2sick said:
kbrkbr said:
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=9539303&query_hl=1
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=14764772&query_hl=1
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=15181056&query_hl=2
Here is what Paxil and other SSRI's do to Prolactin.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=9443523&query_hl=3
This is mainly due to direct stimulation of the 5HT2 subtype of the serotonin receptor. Stimulation of this receptor will cause an increase in ACTH, Cortisol, Prolactin and acts as a governor on dopamine release in certain parts of the brain. Lowering prolactin is well known to increase LH receptor sensitivity.
This one is interesting because it shows a direct decrease in the direct metabolite of dopamine HVA which is a marker for decreased dopamine levels.
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=pubmed&dopt=Abstract&list_uids=8274586&query_hl=2
This is also why wellbutrin an NDRI is so popular to add to SSRI treatment.
It would be interesting to see what a combo like dostinex and proviron would do.
