I guess I have to give the evidence for the error of your ways.
First, substrate preference during exercise is not the predictor of weight loss or body composition, it is INTENSITY...which equals calories. The higher the intensity the more calories, the lower the intensity the lower the calories. As I have said before, if you want to burn primarily fat, then don't exercise. Fat is the primary fuel choice at rest, but the caloric demand is nil. At near 100%VO2 Max, glucose (glycogen) is the sole energy source. Who looks better: endurance athletes or sprinters?
Second, intensity levels determine postexercise metabolism, which also plays a part in weight loss. The greater the intensity, the greater the adrenergic response, which elevates metabolism for a longer time.
Also, MS, during exercise, insulin secretion is blunted, since non-insulin dependant glucose transport occurs. Fahey did a study attempting to elevate insulin levels during exercise and this required the consumption of near 2000 cal drinks.
Here are the studies:
Metabolism 1994 Jul;43(7):814-8
"Impact of exercise intensity on body fatness and skeletal muscle metabolism."
Tremblay A, Simoneau JA, Bouchard C.
Physical Activity Sciences Laboratory, Laval University, Ste-Foy, Quebec, Canada.
The impact of two different modes of training on body fatness and skeletal muscle metabolism was investigated in young adults who were subjected to either a 20-week endurance-training (ET) program (eight men and nine women) or a 15-week high-intensity intermittent-training (HIIT) program (five men and five women). The mean estimated total energy cost of the ET program was 120.4 MJ, whereas the corresponding value for the HIIT program was 57.9 MJ. Despite its lower energy cost, the HIIT program induced a more pronounced reduction in subcutaneous adiposity compared with the ET program. When corrected for the energy cost of training, the decrease in the sum of six subcutaneous skinfolds induced by the HIIT program was ninefold greater than by the ET program. Muscle biopsies obtained in the vastus lateralis before and after training showed that both training programs increased similarly the level of the citric acid cycle enzymatic marker. On the other hand, the activity of muscle glycolytic enzymes was increased by the HIIT program, whereas a decrease was observed following the ET program. The enhancing effect of training on muscle 3-hydroxyacyl coenzyme A dehydrogenase (HADH) enzyme activity, a marker of the activity of beta-oxidation, was significantly greater after the HIIT program. In conclusion, these results reinforce the notion that for a given level of energy expenditure, vigorous exercise favors negative energy and lipid balance to a greater extent than exercise of low to moderate intensity. Moreover, the metabolic adaptations taking place in the skeletal muscle in response to the HIIT program appear to favor the process of lipid oxidation.
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Med Sci Sports Exerc 1996 Sep;28(9):1138-43
"Effects of exercise intensity on 24-h energy expenditure and substrate oxidation."
Treuth MS, Hunter GR, Williams M.
USDA/ARS Children's Nutrition Research Center, Department of Pediatrics, Baylor College of Medicine, Houston, TX 77030, USA.
[email protected].
The purpose of this study was to determine: 1) the reliability of 24-h respiratory calorimetry measurements, and 2) the effects of low- versus high-intensity exercise on energy expenditure (EE) and substrate oxidation over a 24-h period. Eight women (age 28 +/- 4.3 yr) were measured for body composition, maximal oxygen consumption while cycling, and EE in three, 24-h calorimeter tests, with identical work output but differing intensity during a 60-min exercise session. Low-intensity (LI) exercise involved continuous cycling at 50% VO2max; whereas high-intensity (HI) exercise involved interval cycling (2 min exercise/recovery) at 100% VO2max. Subjects were randomly assigned to the first two tests at LI or HI, with the third test at the alternate intensity. No differences in EE or respiratory quotient (RQ) during rest, sleep, exercise, or over the 24 h were found between the first two tests (C.V. = 6.0%), demonstrating the reliability of the measurements. The HI protocol elicited significantly higher EE than LI during rest, exercise, and over 24 h, whereas sleeping EE approached significance. No significant differences in RQ during rest, sleep, or over 24 h were found, but 24-h lipid and carbohydrate oxidation were similar in the two protocols. The HI exercise RQ was significantly higher than LI. These findings demonstrated higher 24-h EE in the HI than LI protocol, but similar 24-h substrate oxidation rates.
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Int J Obes Relat Metab Disord 2001 Mar;25(3):332-9
"Impact of high-intensity exercise on energy expenditure, lipid oxidation and body fatness."
Yoshioka M, Doucet E, St-Pierre S, Almeras N, Richard D, Labrie A, Despres JP, Bouchard C, Tremblay A.
Division of Kinesiology, Laval University, Ste-Foy, Quebec, Canada.
OBJECTIVE: Two studies were conducted to assess the potential of an increase in exercise intensity to alter energy and lipid metabolism and body fatness under conditions mimicking real life. METHODS: Study 1 was based on the comparison of adiposity markers obtained in 352 male healthy adults who participated in the Quebec Family Study who either regularly participated in high-intensity physical activities or did not. Study 2 was designed to determine the effects of high-intensity exercise on post-exercise post-prandial energy and lipid metabolism as well as the contribution of beta-adrenergic stimulation to such differences under a real-life setting. RESULTS: Results from Study 1 showed that men who regularly take part in intense physical activities display lower fat percentage and subcutaneous adiposity than men who never perform such activities, and this was true even if the latter group reported a lower energy intake (917 kJ/day, P<0.05). In Study 2, the high-intensity exercise stimulus produced a greater post-exercise post-prandial oxygen consumption as well as fat oxidation than the resting session, an effect which disappeared with the addition of propranolol. In addition, the increase in post-prandial oxygen consumption observed after the high-intensity exercise session was also significantly greater than that promoted by the low-intensity exercise session. CONCLUSION: These results suggest that high-intensity exercise favors a lesser body fat deposition which might be related to an increase in post-exercise energy metabolism that is mediated by beta-adrenergic stimulation.