Turinabol+ Anavar ULTIMATE STACK!

[Mavafanculo]

8 weeks is enought to do serious damage to your liver, especially if stacking orals. Test has other hormonal functioning in the body which no AAS can mimic. This is why test should be your base as NOTHING ELSE CAN REPLACE IT!

on a side note here, assumming ast/alt numbers are low, any problem continuing on with the orals past the 6 or 8 week norm?? I just got clean liver panal back (right in the middle of normal range) after 8 weeks 60 mg BD dbol, and want to continue using dbol with the test/npp (switching to naposims).

 

[Ross]

ALSO....Using TESTOSTERONE(THE PRIMARY SEX HORMONE!!) in supraphysiological dosages is never safe--thus, anytime you use test, you are essentially going through puberty again:and again....and again.

Stick with testosterone derivatives--synthetic androgens that have anabolic/androgenic activity, though not NEARLY as much so as test. Your body has steroid receptors and knows EXACTLY what to do with each of these unique chemicals. Furthermore, the pituiatry and the testes are not as severely influenced with these more mild androgens, so gains are always more permenent.

THO, TEST IS KING--for MASS....we must never forget that...
and maybe TREN....

 

[hammercurls]

I Want To Hear More........

 

[krishna]

If you use any AAS, your endogenous test production will be shut down and you will have very low test levels. Considering that test serves other purposes in the body contributing to overall general health, it is wise to always include at least a minimal dose of test.

As far as the liver is concerned, I didn't say that it would screw it up, I said that it could. Stacking orals greatly increases the risk of damaging your liver. Liver damage can happen very quickly and suddenly with oral steroids, so just because your liver values are fine after 8 weeks, doesn't mean you're not going to get it. In fact, your at a greater risk the longer you go.

 

[Indy]

Where has that puny little 6'2" lit fuse been, anyway?

Been wondering the same thing, not like him to keep quiet, that's for sure..

 

[Ross]

If you use any AAS, your endogenous test production will be shut down and you will have very low test levels. Considering that test serves other purposes in the body contributing to overall general health, it is wise to always include at least a minimal dose of test.

As far as the liver is concerned, I didn't say that it would screw it up, I said that it could. Stacking orals greatly increases the risk of damaging your liver. Liver damage can happen very quickly and suddenly with oral steroids, so just because your liver values are fine after 8 weeks, doesn't mean you're not going to get it. In fact, your at a greater risk the longer you go.

This is not true. Anavar , primo, tbol, will not shut you down--they will LOWER T-CONCENTRATIONS--not PITUAITARY/Testicular shutdown--as is the case with TESTOSTERONE, Trenbolone, and Anadrol.

Furthermore, TEST does not serve a purpose to overall general HEALTH--in Supraphysiological doses. THE testes produce 10mgs of test a day if that. Who shoots 10mgs as a REPLACEMENT dose? No one--anything over 10mg is SUPRAPHYSIOLOGICAL, therefore SUPERFLUOUS to add to a MILD cycle, as it wll only mess things up.

 

[krishna]

Var, primo, and tbol WILL shut you down, and YES test still serves it's hormonal health functioning in greater doses, although it is more exaggerated.

 

[mendo]

Yes Var, Primo, and Tbol will all shut you down. Krishna is right. By shut down...we mean they cause your testes to respond to excess androgens by stopping production of testosterone completely... Although they are milder than many AAS...these will shut you down at moderate dosages. It is true that you can recover your test production quicker from a cycle of one of these only, compared to a stack with lots of androgenic injectables.

 

[Ross]

Var, primo, and tbol WILL shut you down, and YES test still serves it's hormonal health functioning in greater doses, although it is more exaggerated.

Nope

Anavar won't shut you down, but it can suppress you.


Clin Endocrinol (Oxf). 1993 Apr;38(4):393-8.
The effects of oxandrolone on the growth hormone and gonadal axes in boys with constitutional delay of growth and puberty.
Malhotra A, Poon E, Tse WY, Pringle PJ, Hindmarsh PC, Brook CG.

Endocrine Unit, Middlesex Hospital, London, UK.

OBJECTIVE: We studied the effects of oxandrolone on serum concentrations of LH, FSH, testosterone, GH, SHBG, DHEAS, IGF-I and insulin in boys with constitutional delay of growth and puberty. DESIGN: Ten boys with constitutional delay of growth and puberty, mean age 13.8 years (range 12.4-15.5) were studied. Twenty-four-hour serum concentration profiles of GH, LH and FSH were constructed by drawing blood samples at 20-minute intervals. Three study occasions over a period of 6 months were chosen to assess hormone concentrations before, during and 6 weeks after a 3-month course of oxandrolone (2.5 mg once daily) therapy. RESULTS: Growth velocity increased during oxandrolone treatment and stayed higher after therapy (pre 3.9 +/- 0.5; on 6.3 +/- 0.8; post 6.4 +/- 0.9 cm/year (mean +/- SEM) two way ANOVA, F = 5.3, P = 0.02). Oxandrolone had androgenic effects, suppressing mean serum LH concentrations from 1.7 +/- 0.3 to 1.1 +/- 0.2 U/I and serum testosterone concentrations from 1.9 +/- 0.6 to 0.8 +/- 0.1 nmol/l. SHBG concentrations were also reduced from 130.9 +/- 14.6 to 30.7 +/- 7.3 nmol/l. Serum GH concentration fell slightly from 5.9 +/- 0.6 to 4.8 +/- 0.5 mU/l. After cessation of treatment, there was a significant 'rebound' in mean 24-hour serum LH (2.6 U/l +/- 0.4) and testosterone concentrations (3.2 +/- 0.9 nmol/l) but no change in serum GH concentrations. SHBG values also rose but not to the same extent as those observed before therapy (82.0 +/- 8.4 nmol/l). There were no statistically significant differences in serum concentrations of FSH, DHEAS, IGF-I and insulin over the study period. In a stepwise multiple regression analysis of factors that might influence the growth rate observed, the 24-hour mean serum testosterone concentration and the treatment (on or off) with oxandrolone were the main influences. The relationship was described by the equation Height velocity = 0.69 (24-hour mean serum testosterone concentration)+1.70 (treatment regimen)+3.37 (adjusted R2 = 0.35, F = 8.39, P = 0.001). CONCLUSIONS: Oxandrolone has an androgenic action as shown by changes in serum LH, testosterone and SHBG concentrations and by the lack of effect on FSH. No effect of oxandrolone on the GH axis was documented. We suggest that the growth promoting effects of oxandrolone are related in part to the mild androgenic effects of the steroid and the growth acceleration following oxandrolone withdrawal may reflect increasing total serum testosterone concentrations and decreasing levels of SHBG and progress in puberty.

PMID: 8319371 [PubMed - indexed for MEDLINE

 

[nishnish]

All I know is that I'm getting really sick and tired of the necrosis that I experience from injecting UG gear that has high BA content. I have pits in the sweeps of both thighs that are about half a golfball deep when my leg is flexed. I'm not really interested in continuing on that path to "see what happens".
I am seriously considering nothing but oral cycles in the future. It will take a little more monitoring of my liver, but I think I will suffer less test flu, sore injection sites, and other bullshit.

yikes bro!!!!!

stop using that particular UG "brand"!!!!

seriously, there IS UG stuff out there that does not cause infections or necrosis...

necrosis- jesus bro stop using that shit!