Mathematical look at frequency of injections

[The Man Child]

Nice post Andy. Probably the second best Post I've read this week


I've always been a fan for ED injections. I always thought it was a placebo affect, but now I guess that it really works. HMMM

 

[Stew Meat]

Andy, what I meant for the life of enthanate being 10-12 days is that hormone return to baseline in about 12 days (depending on the dossage)... you stated the halfife was about 7 days...

Regardless of all the hearsay about "halflives" of various AS, there has to be some difinitive truth. Given this would probably be dependant on an individual's level of esterase... I'm sure there are individual differences.
I have to admit, I was under the impression that an ester existed as a chain of carboxal groups each bound to an individual molecule of hormone and that breaking down the ester (removing the carbon group) frees up the hormone for circulation... I really didn't understand your explanation, but it seemed to be a lot different than my thoughts on esterfication...

Riker, a "roid calculator" just repeats the non-empiracal garbage that floats around about de-esterfication/ halflives. I was asking Andy if he'd ever seen scientific evidence of ACTUAL time periods for complete de-esterfication of various esters...

Twisted Steel, Paneri answered your Sustonon question perfectly. It can be ok to use, but it complicates your cycle and should not be a choiice for a newbie. It contains a mix of esters including prop. It needs to be shot EOD then followed by a more stable AS such as prop or enthanate so that HPTA isn't supressed for months and clomid therapy can be timed. Continued supression of HPTA does not lead to keeping more of your gains, and is in fact counterproductive in the sense. It will delay the loss of gains, but when they come, you'll actually loose more as long as you don't keep cycling.

Conan, I feel ya on the insulin pumps, brotha!

Hugh, I agree... I don't think oil base slows the break down of AAS one iota. I see no difference between oil based and water based... oil based may have better dispursement... and be less painful. I perfer a water base because it would cut down on acne... I don't know why more esterfied hormones aren't contained in water bases...? Perhaps due to carboxilic acid's oxidation of the ester? Andy?


Andy, I forgot about the DNP thread...j




-Stew

 

[M.O.D]

i would only do one test and that is propionate..EOD for me..


btw there are some smart mofo's on this thread....me like it

 

[Andy13]

Andy, what I meant for the life of enthanate being 10-12 days is that hormone return to baseline in about 12 days (depending on the dossage)... you stated the halfife was about 7 days...

Regardless of all the hearsay about "halflives" of various AS, there has to be some difinitive truth. Given this would probably be dependant on an individual's level of esterase... I'm sure there are individual differences.
I have to admit, I was under the impression that an ester existed as a chain of carboxal groups each bound to an individual molecule of hormone and that breaking down the ester (removing the carbon group) frees up the hormone for circulation... I really didn't understand your explanation, but it seemed to be a lot different than my thoughts on esterfication...

Hugh, I agree... I don't think oil base slows the break down of AAS one iota. I see no difference between oil based and water based... oil based may have better dispursement... and be less painful. I perfer a water base because it would cut down on acne... I don't know why more esterfied hormones aren't contained in water bases...? Perhaps due to carboxilic acid's oxidation of the ester? Andy?

Andy, I forgot about the DNP thread...j

-Stew

Actually, I was stammering around ester hydrolosis as to not step on anybody's toes . But i'm positive on how the ester works.
The ester is made from a single carboxilic acid (fatty acid). Without the ester, it would not dissolve all that well in oil. According to the pharmacological theory of the ester, it increases it's hydrophobicity so that it is more soluble in the oil and leaks out of the depot over time. The shorter the ester (shorter fatty acid= less hydrophobicity) the faster it diffuses from the oil . when it exits the oil, the ester is hydrolyzed by the esterase enzyme. The result is the parent steroid plus the fatty acid in which it was made of.

Injectable steroids without esters have somwhat poor solubility in oil. And so water is used, after all, it doesn't have an ester so it's solubility in oil related to release rate doesn't apply here. Esterified steroids in water would defeat the purpose of the ester.

Andy

 

[DEI]

Great Info

 

[GaryWary]

just thought I'd bump this.

Although test esters still confuse me..

fuck knows what my test levels are at the moment. I injected 500mg of sust the other day. Just threw caution to the wind.

So is Omnadren or Testoviron quicker acting and shorter living than SUST?

 

[Andy13]

just thought I'd bump this.

Although test esters still confuse me..

fuck knows what my test levels are at the moment. I injected 500mg of sust the other day. Just threw caution to the wind.

So is Omnadren or Testoviron quicker acting and shorter living than SUST?

Well, bro, making a graph for sustanon would be a major pain in the arse.

I would split up sustanon (at least) into twice weekly injections..

Andy

 

[CYCLEON]

yes - only a math freak would enjoy that calculation. ady is right tho - all of them have better results injected 2x per week, and better still ETD - lets u get the prop.

 

[-Falcon-]

My brain hurts !!!!!!!
how about just telling me how many injections would I need per week if I were taking test enathate, cypinate and eq for the optimal results.......

 

[noplay]

bu