Libido Problems? Read this!

[drveejay11]

You can buy pharmacutical yohimbee from http://www.rejuvenationfoundation.org/ They work fucking wonders on the sex drive compared to the Bull shit they sell in america. This shit i took 3X/day and had to drop the dose because i was losing control of my erections and libido at work. I wanted to fuck every good looking girl in there.

Proviron works GREAT too!

JB....I agree with you when you declare Yohimbe to aid with sex drive BUT it does it in an entirely different fashion. The mechanism that Yohimbe/Maca/ Tribulis all work on is by aiding in restoring Test levels. Sex Drive will be restored if in fact THIS is theonly problem!

What Dostinex does is actually work on libido (desire) and the mental facet of "sex drive". It is a dopamine agonist. By restoring some peple's HPTA back, will NOT automatically restore sex drive/desire. Some people have coconuts for balls (mega-test levels) and NO libido. However, dopamine agonist have been known for years to increase libido (they actually discovered Parkinson's patients had crazy high libido issues once they hbegan taking the meds......dopaime agonists).

Proviron DOES in fact mitigate a chemical response in the brain that will trigger the DESIRE part of sex drive. It is effective IMHO.

Dostinex combats the hyperprolactinemia (raised Prolactin Levels)
and will help lack of sexual desire sufferers in 2 possible different ways:
1) those who have painful post orgasmic erections (caused by increased Prolactin levels)
2) Decrease high prolactin levels thereby inhibiting negative chemical signals to the brain and allowing for sexual desire to be physiologically achieved!

 

[drveejay11]

If this is real how do you go about getting this stuff as I didn't really look at it?

check your pm's bro

 

[Sigmund Roid]

VJ, is there an attenuation effect after a while, i.e. is the effectiveness of the medication decreasing over time?

 

[drveejay11]

VJ, is there an attenuation effect after a while, i.e. is the effectiveness of the medication decreasing over time?

To date, I have yet to read ANYTHING stating that one would attenuate after timed usage.

I think there would be 2 schools of thought if one were to guess:

1) The body DOES seem to attenuate to most drugs after long periods of time (yes-very general statement)

Conversely:

2) Due to the fact that its mechanism of action is based upon the drug's ability to decrease Prolactin levels; I would say NO attenuation could be achieved. Its action could be compared to that of liquidex. It simply inhibits the activity of a hormone that is considered "too high"; in this case Prolactin vs Estrogen.

 

[drveejay11]

^

 

[Sigmund Roid]

Thx, VJ

Another Q:

Do you think that long term use of high dosage AAS will supress dopamine levels in the brain? There are several cases of people with permanent low libido, even after they restored their HPTA by the use of HCG, clomid or testosterone for extended periods.

 

[drveejay11]

Thx, VJ

Another Q:

Do you think that long term use of high dosage AAS will supress dopamine levels in the brain? There are several cases of people with permanent low libido, even after they restored their HPTA by the use of HCG, clomid or testosterone for extended periods.

IMO-----yes there is a chance! Long term use of any drugs which directly effect hormonal chemistry will in some way effect the body's ability to function 100% NORMALLY! We can protect/restore our HPTA as much as possible but ultimatley it's the body's response (or not) to counter-act such attempts. Here we go again with genetics----some people WILL BE effected as you mentioned, some won't.

This is NOT speculation. I have read reports on this very subject! I will try to find and paste when i get a chance!

AS are great in so many ways but don't ever forget we are dealing with hormone altering DRUGS here. When done properly, the benefits out-weigh the risks------but try asking that to someone who's genetics didn't come through for him!

 

[drveejay11]

.

 

[Sigmund Roid]

Ok VJ, you know your stuff!

Another Q:

Mostly, right after ejaculation you have a 'remission period' it is called? When you are hypersensitive and have an irritating feeling 'over there'. Is this caused by some neurotransmitter depletion, like dopamine or overstimulation of the nerve endings? I cannot figure out the neurofysiological difference that makes men different from women in that aspect. In other words, how can men overcome this remission period in a much shorter time?

 

[drveejay11]

Ok VJ, you know your stuff!

Another Q:

Mostly, right after ejaculation you have a 'remission period' it is called? When you are hypersensitive and have an irritating feeling 'over there'. Is this caused by some neurotransmitter depletion, like dopamine or overstimulation of the nerve endings? I cannot figure out the neurofysiological difference that makes men different from women in that aspect. In other words, how can men overcome this remission period in a much shorter time?

The answer could come from several things bro; sorry but it’s actually a complicated MULTI-FACETED question:

Prostate dysfunction. The most common problem is the "temporary" prostate enlargement, as a result of overproduction of dihydrotestosterone (DHT) due to the burning of testosterone by an enzyme called 5-alpha reductase. The excessive DHT trapped in the prostate and its surround muscle, as a result of a poor local blood circulation and an overburst of testosterone or 5-alpha reductase in a sexual encounter, will cause erection or post-orgasmic pain for men.

IC “Interstitial cystitis”. IC can contribute to painful ejaculations, and normal sexual response can certainly be adversely affected by pain or the anticipation of pain. Plenty of evidence suggests that men treated successfully for IC usually experience normal erections and ejaculations.

(Interstitial cystitis (IC) is a chronic inflammatory condition of the bladder wall. Its cause is unknown. "Common" cystitis, also known as a urinary tract infection, is caused by bacteria and is usually successfully treated with antibiotics. Unlike common cystitis, IC is believed not to be caused by bacteria and does not respond to conventional antibiotic therapy. It is important to note that IC is not a psychosomatic disorder nor is it caused by stress.)


Hormonal Deficiencies leading to Nervous System dysfunction Upon ejaculating, one may experience an abrupt drop of the neurotransmitters acetylcholine, dopamine and serotonin and the hormone testosterone in the brain and an excessive conversion of dopamine-norepinephrine-epinephrine (adrenalin) in the brain and adrenal medulla. As a result, the Central Nervous System contains excessive cortisol that induced initial Euphoria followed by subsequent depression and then lowered the threshold for seizure activity, resulting in the instability of his brain.

Like this: ( response to orgasm”ejaculation”):
Orgasmic Stress => Dopamine-adrenalin conversion => Stimulating Central Nervous System => Hypothalamus ==(CRF)==> Anterior Pituitary ==(ACTH)==> Adrenal Cortex => secreting Androgen, Aldosterone and Cortisol, where Cortisol, in turn, floods and inhibits the Central Nervous System, Hypothalamus and Anterior Pituitary. Thus, excessive adrenalin triggers the flooding of excessive Cortisol into the brain.

Deficiency of the relaxation hormone prostaglandin E-1This problem is associated with a deficiency of the relaxation and tissue-elastic hormone prostaglandin E-1 (PGE-1) and the excessive trapping of the stress neurhormone epinephrine (adrenalin) in the tissues in tailbone, rectum, anus, perineum and groins, in the post-orgasm or post-ejaculation state when you lost your semen to ejaculation. Generally, semen contains PGE-1 and PGE-2. Here, Seminal PGE-1 is diffused into the pelvic tissues to increase the relaxation and tissues elasticity for touching, stretching, and expansion/contraction; PGE-2 is to enhance the contraction (intensity) of orgasm in the tissues around the pelvic cavity. When your tissues produce insufficient PGE-1, you need the PE-1 diffusion to retain the tissue elasticity and relaxation. Once your ejaculate, your seminal vesicles are empty or semi-empty and your tissues and ligaments around the pelvic cavity from the low abdomen, groins, testicles, scrotum and perineum to your anus/rectum/tailbone experiences an abrupt drop of the PGE-1. You will feel rigidity and tightness in your low back and groins after ejaculating. The worst situation is that orgasm is a result from sympathetic fire accompanying with the dopamine-epinephrine conversion. CAUSING PAIN