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genezapharmateuticals
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Research Chemical SciencesUGFREAKeudomestic
napsgeargenezapharmateuticals domestic-supplypuritysourcelabsResearch Chemical SciencesUGFREAKeudomestic

Eating Meat is what made us Humans

jh1 said:
Fish is meat, douche bag ass fuck.
So you watched, "Human Development for Dummies?" :) There is a strong theory about fish and human brain development independent of other meat.
 
javaguru said:
So you watched, "Human Development for Dummies?" :) There is a strong theory about fish and human brain development independent of other meat.

I've heard that as well.

This wasn't specific to FISH.. it was about 'meat' in general and the levels of protein only found in the diets of non vegans.
 
jh1 said:
I've heard that as well.

This wasn't specific to FISH.. it was about 'meat' in general and the levels of protein only found in the diets of non vegans.
The fish theory deals with DHA/EPA...essential fatty acids and their prevalence in the human brain. Infant formula companies have been trying to stabilize them in infant formula, it's their "holy grail" to mimic breast milk. Their research considers it that important...it's the DHA/EPA studies that flax proponents cite when proclaiming the benefits of their product, which requires conversion in the body.
 
javaguru said:
So you watched, "Human Development for Dummies?" :) There is a strong theory about fish and human brain development independent of other meat.

There wasn't much fish in the jungles of East Africa where MOST of the cranial capacity increased and verified through fossil documentation, attributed to, eating meat. This is like anthropology 101 stuff. Hold up brain, discuss teeth of fossils, hold up petrified poop showing increased meat consumption, pass out test. Believe!!
Fish, well that's made wolves adapt into whales, to go eat fish. They got fat but lived longer. Don't know which was a smarter evolutionary tract. But, definitely, we equate intelligence in a conventional sense with abstract reasoning and overcoming extinction and the ability to adapt. Protein fueled that evolutionary change of cranial size. Most of the fish eaters still have 400cc brains. Which STRONG theory are you referring to anyways? Most all of the experts agree on where and how and why cranial size increased.
What fossil records exist or genetic retracement tests or studies, beyond thoery provide evidence to back up the fish argument?
 
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Richard_D_Feynman said:
There wasn't much fish in the jungles of East Africa where MOST of the cranial capacity increased and verified through fossil documentation, attributed to, eating meat. This is like anthropology 101 stuff. Hold up brain, discuss teeth of fossils, hold up petrified poop showing increased meat consumption, pass out test. Believe!!
Fish, well that's made wolves adapt into whales, to go eat fish. They got fat but lived longer. Don't know which was a smarter evolutionary tract. But, definitely, we equate intelligence in a conventional sense with abstract reasoning and overcoming extinction and the ability to adapt. Protein fueled that evolutionary change of cranial size. Most of the fish eaters still have 400cc brains. Which STRONG theory are you referring to anyways? Most all of the experts agree on where and how and why cranial size increased.
What fossil records exist or genetic retracement tests or studies, beyond thoery provide evidence to back up the fish argument?
Here ya go....

http://www.eurekalert.org/pub_releases/2006-02/nsae-tsf021706.php

Contact: Stephen Cunnane
[email protected]
819-821-1170
Natural Sciences and Engineering Research Council

There's something fishy about human brain evolution
Forget the textbook story about tool use and language sparking the dramatic evolutionary growth of the human brain. Instead, imagine ancient hominid children chasing frogs. Not for fun, but for food.
According to Dr. Stephen Cunnane it was a rich and secure shore-based diet that fuelled and provided the essential nutrients to make our brains what they are today. Controversially, according to Dr. Cunnane our initial brain boost didn't happen by adaptation, but by exaptation, or chance.

"Anthropologists and evolutionary biologists usually point to things like the rise of language and tool making to explain the massive expansion of early hominid brains. But this is a Catch-22. Something had to start the process of brain expansion and I think it was early humans eating clams, frogs, bird eggs and fish from shoreline environments. This is what created the necessary physiological conditions for explosive brain growth," says Dr. Cunnane, a metabolic physiologist at the University of Sherbrooke in Sherbrooke, Quebec.

The evolutionary growth in hominid brain size remains a mystery and a major point of contention among anthropologists. Our brains weigh roughly twice as much as our similarly sized earliest human relative, Homo habilis two million years ago. The big question is which came first – the bigger brain or the social, linguistic and tool-making skills we associate with it?

But, Dr. Cunnane argues that most anthropologists are ignorant or dismissive of the key missing link to help answer this question: the metabolic constraints that are critical for healthy human brain development today, and for its evolution.

Human brains aren't just comparatively big, they're hungry. The average newborn's brain consumes an amazing 75-per cent of an infant's daily energy needs. According to Dr. Cunnane, to fuel this neural demand, human babies are born with a built-in energy reservoir – that cute baby fat. Human infants are the only primate babies born with excess fat. It accounts for about 14 per cent of their birth weight, similar to that of their brains.

It's this baby fat, says Dr. Cunnane, that provided the physiological winning conditions for hominids' evolutionary brain expansion. And how were hominid babies able to pack on the extra pounds? According to Cunnane their moms were dining on shoreline delicacies like clams and catfish.

"The shores gave us food security and higher nutrient density. My hypothesis is that to permit the brain to start to increase in size, the fittest early humans were those with the fattest infants," says Dr. Cunnane, author of the book Survival of the Fattest, published in 2005.

Unlike the prehistoric savannahs or forests, argues Dr. Cunnane, ancient shoreline environments provided a year-round, accessible and rich food supply. Such an environment was found in the wetlands and river and lake shorelines that dominated east Africa's prehistoric Rift Valley in which early humans evolved.

Dr. Cunnane points to the table scrap fossil evidence collected by his symposium co-organizer Dr. Kathy Stewart from the Canadian Museum of Nature, in Ottawa. Her study of fossil material excavated from numerous Homo habilis sites in eastern Africa revealed a bevy of chewed fish bones, particularly catfish.

More than just filling the larder, shorelines provided essential brain boosting nutrients and minerals that launched Homo sapiens brains past their primate peers, says Dr. Cunnane, the Canada Research Chair in Brain Metabolism and Aging.

Brain development and function requires ample supplies of a particular polyunsaturated fatty acid: docosahexaenoic acid (DHA). DHA is critical to proper neuron function. Human baby fat provides both an energy source for the rapidly growing infant grey matter, and also, says Dr. Cunnane, a greater concentration of DHA per pound than at any other time in life.

Aquatic foods are also rich in iodine, a key brain nutrient. Iodine is present in much lower amounts from terrestrial food sources such as mammals and plants.

It was this combination of abundant shoreline food and the "brain selective nutrients" that sparked the growth of the human brain, he says.

"Initially there wasn't selection for a larger brain," argues Dr. Cunnane. "The genetic possibility was there, but it remained silent until it was catalyzed by this shore-based diet."

Dr. Cunnane acknowledges that for the past 20 years he's been swimming upstream when it comes to convincing anthropologists of his position, especially that initial hominid brain expansion happened by chance rather than adaptation.

But, he says, the evidence of the importance of key shoreline nutrients to brain development is still with us – painfully so. Iodine deficiency is the world's leading nutrient deficiency. It affects more than a 1.5 billion people, mostly in inland areas, and causes sub-optimal brain function. Iodine is legally required to be added to salt in more than 100 countries.

Says Dr. Cunnane: "We've created an artificial shore-based food supply in our salt."


###
Contact:
Dr. Stephen Cunnane
(819) 821-1170, ext. 2670 (office)
[email protected]

Arnet Sheppard
NSERC Public Affairs
(613) 859-1269

Dr. Cunnane's AAAS Presentation
Expatiation, Metabolic constraints and Human Brain Evolution,
Saturday, February 18, 2006
8:00 a.m. - 9:30 a.m. Central Time
 
For the record, he's a respected scientist.....
Dr Tom Cunnane

http://www.pharm.ox.ac.uk/academics/cunnane

University Lecturer
Fellow and Tutor in Physiological Sciences, Hertford College
Dr Cunnane graduated in Pharmacology at Bath University, and obtained a PhD in the Department of Pharmacology, Glasgow University. He continued his research in the Department of Physiology, Leicester University where he worked for two years with Professor Asa Blakeley funded by the MRC. He then set up an electrophysiological laboratory at the Karolinska Institute, Stockholm working with Professor Lennart Stjärne as an MRC and Royal Society Overseas Fellow. Dr Cunnane returned to the UK to set up his own independent research group as an MRC Senior Fellow in the Department of Pharmacology, Glasgow University, before being appointed to his College Fellowship and University Lectureship in Oxford in 1984. Dr Cunnane has developed techniques to study the relationship between action potential propagation and neurotransmitter release at the level of the individual varicosity in sympathetic nerve terminals. More recently, Dr Cunnane's group have been studying calcium dynamics in mature nerve terminals using confocal microscopy, and have discovered an unexpected and novel action of nicotine. Briefly, nicotine induces spontaneous asynchronous calcium transients in individual varicosities.

Dr Cunnane has been an Editor of the British Journal of Pharmacology and Chairman of the International Union of Physiological Sciences Commission on the Physiology of Neurotransmitters and Modulation.

Key Research Areas
Electrophysiological characterisation of neurotransmitter release mechanisms in single living varicosities in postganglionic sympathetic neurones

Presynaptic receptor activation and the role of potassium and multiple forms of calcium channels in varicose nerve terminals

Investigation of the nature of the calcium channels controlling ACh release from preganglionic sympathetic nerves

Molecular machinery controlling neurotransmitter release
 
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