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Active Rest and Recovery

Arioch

New member
A little bit of something I have been working on. More will follow.

Active Recovery

What is active recovery? Active recovery is a method of not only clearing blood lactate, but using the bodies natural recovery ability to enhance athletic performance. Even though most of this has been directed at endurance athletes, it can be adapted to strength training athletes as well. By stimulating the clearance of lactate, the glycotic system, which is one of the primary methods of allowing the muscles to continue to generate force, can be more readily replenished.

The process of metabolism of fat, carbohydrate, or protein, which can occur as a result of intense muscular activity, generally results in the production and accumulation of lactate and hydrogen ions (H+) ( a symport mechanism where two molecules are moved simultaneously in the same direction) due to the dissociation of inorganic acids. Please note that lactate and lactic acid are not the same compound. Lactic acid is an acid with the chemical formula C3H6O8, and lactate is any salt of lactic acid. When lactic acid releases H+, the remaining compound joins with Na+ or K+ to form a salt. Anaerobic glycolysis produces lactic acid, but it quickly dissociates and the salt - lactate - is formed1. This process not only increases intramuscular levels of lactate but circulating levels as well.2,3 Elevated levels of lactate both inhibits further glycogen breakdown during resistance exercise4,5 as well as inhibiting the activity of glycotic enzymes.6 In addition, the acid decreases the muscle fibers’ calcium-binding capacity and thus may further impede muscle contraction.

There has been a great deal of research showing the positive effects of active recovery on lactate removal, and it has been shown that lactate removal occurs far more rapidly during continuous aerobic recovery.7,8,9,10,11 Most of these studies have been on endurance type activities, but have shown that the greatest reductions in blood lactate occur at an average of 63% aerobic power (O2 max). The rate of blood lactate removal is related to the intensity of the recovery exercise with optimal recovery occurring between 25% and 63% of O2 max.12,13

There has not been much research into the effects of active recovery on high-intensity exercise, although a few studies have been performed.14,15,16 They have all shown that short duration active recovery provides superior performance in comparison to passive rest. This has been demonstrated to improve both efficiency as well as power output. Only a single study has been performed on the effects of active rest during resistance training.17 During a squat workout, where ten rep sets were performed, low intensity cycling was done at 25 and 50% of onset of blood lactate accumulation (OBLA). Active recovery was performed on a bicycle ergometer. Recovery was shown to be greatest at 25% OBLA, which was indicated by the greatest increase in performance on a maximal repetition squat test. This clearly shows the benefits of active recovery for resistance trainers.

Active recovery is using submaximal work to stimulate the removal of lactate, as was previously mentioned. There are a great many varieties of active recovery. The simplest would be performing submaximal (as based on perceived effort levels) sets of a similar type of activity during a period of time when one has recovered enough to execute the movements safely, but not so far recovered that there will be little to no benefit. An example would be executing a few sets of squats on the day following an intense squat workout. While the percentages may vary, a simple plan would be 50% of the weight, 50% of the volume. This may not provide enough stimulation, and the percentages could be varied based on the recovery ability as well as the skill of the individual. While this sort of workout will do little to clear lactate, which should already be back to normal, it can help with the micro trauma caused following resistance training by promoting an increase in blood flow to the area, and this inflammation can simulate some of the effects of the inflammation stage of injury, which promotes the release of chemical mediators from the cells into the extra cellular fluid, and can cause the release of inflammatory mediators such as kinins and prostoglandins.18 The use of bands, such as jumpstretch bands, can also be used to achieve a similar effect, as many of the exercise performed in the gym can be duplicated in the home, which will not only provide active recovery, but convenience as well. For these purposes, the bands can be considered light recovery work, similar to a low intensity set.

Active recovery can be used during a training session, as was previously discussed. In a method similar to the above light working sets, low intensity sets can be performed in between higher intensity sets. At Westside, many lifters will perform sets of pushdowns during rest periods when executing heavy tricep movement. This is active recovery. The bands can be used as well. Pushups could be performed in-between bench sets, etc. The variations are practically endless.

Passive recovery, although not really discussed, should at least be mentioned. It is just what it says: passive, doing nothing. The best example of passive recovery is going to sleep, and this is something that should never be neglected like certain idiots are doing as they finish typing this at 0200.

Other methods of improving recovery. Conditioning takes many names, and one of the most popular is GPP, or general physical preparedness. This has been discussed exhaustively by both Louie Simmons and Dave Tate, and everyone should know where to look for their articles. It is also discussed extensively in the Russian literature that the idea was taken from, along with the need to raise training volume. Like any other skill, the more you practice, the better you become, assuming you are performing properly, hence the need to recover. There is one school of thought, which claims, ad nauseum, that as you get stronger, you must increase your recovery time, and decrease your training volume. Only two things will be said about this: They are wrong, and you never see them winning lifting championships.

Recommended reading:

Devlin’s Biochemistry with Clinical Correlations. Thomas M. Devlin, ed. Wiley-Liss Pub. New York.

Science and Practice of Strength Training. Vladimir M. Zatsiorsky. Human Kinetics. Ill.

The Training of the Weightlifter. RA Roman. Sportivny Press. Livonia, MI.

A System of Multi-Year Training in Weightlifting. AS Medvedyev. Sportivny Press.

Managing the Training of Weightlifters. Laputin and Oleshko. Sportivny Press.

Fundamentals of Special Strength Training in Sport. Verkoshansky. Sportivny Press.

Supertraining. Siff and Verkoshansky. Supertraining Publications. CO.
 
For some reason, my references do not seem to be copying. I will post them later. Probably tomorrow.
 
References:


1Physiology of Sport and Exercise, Willmore and Costill, Human Kinetics, 1994.

2Hermansen, L., and I. Stensvold. Production and removal of lactate during exercise in man. Acta Physiol. Scand. 86:191–201. 1972. [PubMed Citation]

3Karlsson, J., B. Hulten, and B. Sjodin. Substrate activation and product inhibition of LDH activity in human skeletal muscle. Acta Physiol. Scand. 92:21–26. 1974. [PubMed Citation]

4Hogan, M.C., L.B. Gladden, S.S. Kurdak, and D.C. Poole. Increased [lactate] in working dog muscle reduces tension development independent of pH. Med. Sci. Sports Exerc. 27:317–327. 1995.

5Nakamaru, Y., and A. Schwartz. The influence of hydrogen ion concentration on calcium binding and release by skeletal muscle sarcoplasmic reticulum. J. Gen. Physiol.. 59:22–32. 1972.

6Danforth, W.H., and E. Helmreich. Regulation of glycolysis in muscle. The conversion of phosphorlyase b to phosphorylase a in frog sartorius muscle. J. Biol. Chem. 239:3133–3138. 1964.

7 Belcastro, A.N., and A. Bonen. Lactic acid removal rates during controlled and uncontrolled recovery exercise. J. Appl. Physiol. 39:932–936. 1975. [PubMed Citation]
8Davies, C.T.M., A.V. Knibbs, and J. Musgrove. The rate of lactic acid removal in relation to different baselines of recovery exercise. Int. Z. Angew. Physiol. 28:155–161. 1970.

9Gisolfi, C., S. Robinson, and E.S. Turrell. Effects of aerobic work performed during recovery from exhausting work. J. Appl. Physiol. 21:1767–1772. 1966. [PubMed Citation]

10Hermansen, L., and I. Stensvold. Production and removal of lactate during exercise in man. Acta Physiol. Scand. 86:191–201. 1972. [PubMed Citation]

11Newman, E.V., D.B. Dill, H.T. Edwards, and F.H. Webster. The rate of lactic acid removal in exercise. Am. J. Physiol. 118:457–462. 1937.

12Stamford, B.A., R.J. Moffatt, A. Weltman, C. Maldonado, and M. Curtis. Blood lactate disappearance after supramaximal one-legged exercise. J. Appl. Physiol. 45:244–248. 1978. [PubMed Citation]

13Weltman, A., B.A. Stamford, and C. Fulco. Recovery from maximal effort exercise: Lactate disappearance and subsequent performance. J. Appl. Physiol. 47:677–682. 1979. [PubMed Citation]

14Bogdanis, G.C., M.E. Nevill, H.K.A. Lakomy, C.M. Graham, and G. Louis. Effects of active recovery on power output during repeated maximal sprint cycling. Eur. J. Appl. Physiol. 74:461–469. 1996. [PubMed Citation]
15Signorile, J.F., C. Ingalls, and L.M. Tremblay. The effects of active and passive recovery on short-term, high-intensity power output. Can. J. Appl. Physiol. 18:31–42. 1993. [PubMed Citation]

16Weltman, A., B.A. Stamford, R.J. Moffatt, and V.L. Katch. Exercise recovery, lactate removal, and subsequent high-intensity exercise performance. Res. Q. 48:786–796. 1977. [PubMed Citation]

17Effects of Active and Passive Recovery Conditions on Blood Lactate, Rating of Perceived Exertion, and Performance During Resistance Exercise. KEITH P. CORDER, JEFFREY A. POTTEIGER, KAREN L. NAU, STEPHEN F. FIGONI, and SCOTT L. HERSHBERGER.Department of Health, Sport, and Exercise Sciences, University of Kansas, Lawrence, Kansas 66045.

18Oakes, BW. The classification of injuries and mechanisms of injury, repair and healing. In: Textbook of Science and Medicine in Sport. Bloomfield J, Fricker PA, Fitch KD, eds. 1991: 200-217. Human Kinetics Books, Ill.
 
Spatts, could you give me some specific questions?

For the most part I tend to use them as simply low intensity exercise to assist recovery. I also use them for compensatory acceleration, but that is quite another issue.
 
I will try and give some specifics tomorrow.

I do not understand why any bodybuilder does anything, so you are way ahead of me on that one.
 
"I do not understand why any bodybuilder does anything"

Outstanding quote.......

BTW, yes active recovery (20 - 30% VO2max) will enhance lactate removal, BUT if you're goal is to replenish muscle glycogen between bouts of high-intensity exercise, sit down and rest.

When blood lactate is high and pH low, lactate efflux from muscle is slowed, this is one possible reason, in addition to high levels of intracellular glucose that glycogen resynthesis is so rapid after high intensity exercise vs prolonged glycogen depleting exercise. Thus, if the goal is to replenish glycogen for a later bout of high intensity work, then complete rest between sets is the way to go. Remember also that too high (+30% VO2max) active recovery exercise will slow La removal.

W6
 
Don't know which I hate more...the bands or the sled :)

B True
 
Actually Spatts, the bands can produce more eccentric overload when used in conjunction with speed training, so they are cycled in and out with chains, which do not provide as much eccentric overload.
 
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