shortstack said:
why do people always want the easy way to answers. just do some reaserch buddy.
your statment is very un-educated.... first of all, a healthy person,can still have a thyroid disorder or disease, this beng why there are ove a MILLION un-diagnosed cases in the US. so basically if someone where to be un-educated and have say.....graves disease (a disease were you have to much thyroid) and say to themselves oh well im pretty healthy im gonna take some t3. you know what could happen...... actually alot of things, maybe and most likeley a "thyroid storm" in which it has a fatality rating of around close to 100%. but no you have all the answers so go ahead buddy, take your thyroid cause you think your healthy, but do me a favor, dont imply that to people that they can take t3 if they think there healthy.
When you make too much thyroid = hyperthyroidism. Easily countered with beta-blockers such as propanolol, which is what people who have abnormal(high) thyroid production normally take.
And if you're going to try to prove something, at least use some science. Not your opinions, which are baseless.
And Graves Disease? Never ceases to make me wonder how people always single out cases that are so specific to be completely nonsensical to the gist of the thread. Graves disease has a completely different mode of action.
A brief little explanation:
1. Thyroid disease in anormal person? Sure....if you have ZERO iodine in y
your diet. And don't get any from supplements.
Do you even know how the thyroid works?
Only 20% of the circulating T3 in your blood plasma is from endogeneous production. The rest(80%) comes, from the conversion of T4 to T3 via the de-iodinase enzyme(iodine based). This enzyme basically cleaves of an iodine molecule from T4 to make it into T3.
So, if you don't take iodine in any shape or form T4 to T3 conversion is reduced, and you end up with less T3 circulating in your blood plasma.
2. ESS Syndrome. Very common in athletes. Their natural T4 to T3 conversion does not happen efficiently. The problem (due to a whole host of factors), is that the T4 gets converted to rT3 (reverse T3). rT3 is the optical isomer of T3, and is metabolically inactive, therefore, your plasma T3 levels will be low again.
3. Graves disease is not a problem with the thyroid itself. It's a problem with the persons TSH, which is very high. Therefore, it indirectly stimulates the production of too many thyroid hormones. So again, your statement has no basis.
You're welcome to try to debate this with me, but don't ever tell me I haven't done my research...because that is ridiculous.
And lol@thyroid storm...sure, if you took 600mcg in one go.
And near 100% fatality? That number is complete and utter BS. No offense. As I stated before Beta-blockers reduce thyroid hormone production significantly. Run a search for propanolol and it's uses. It's the most common drug in treating thyro-toxicosis in patients with hyperthyroidism.
And btw, as T3 increases your metabolism, it also lowers your total cholesterol. Which again is beneficial.
Gist of my post: T3 has many, many uses in our pharmaceutical arsenal, but just because you don't understand it's mechanisms, doesn't mean you have to have to be afraid of it, and make other people afraid of it.