The 25 MOST ASKED (and misunderstood) Questions On EF. Don't Ask Again!

[Nelson Montana]

Not bad old friend.

I agree with most of your statements, but some of them are just downright false, first and foremost being that "Test is Test". QUALITATIVELY AND QUALITATIVELY, that statement is just wrong. Stop listening to ANTHONY!

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SORRY MY FRIEND BUT I BELIEVE IT IS YOU WHO IS WRONG.


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You also state that the ideal cycle length is "One month minimum, 12 weeks maximum. Anything less is a waste, anything more may cause permanent suppression." This is simply nonsense Nelson, there is absolutely ZERO scientific evidence to validate that assertion, but rather, quite the CONTRARY.

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YOU MEAN THERE IS EVIDENCE THAT LONGER AND HIGHER DOSAGES ARE NO MORE SUPPRESSIVE? COME ON BRO, I DON'T MIND A DEBATE BUT THAT'S JUST RIDICULOUS.


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As far as there being "no such thing as faster acting esters", this is again COMPLETELY FALSE. The ester does not only determine the half-life, but also the RATE OF RELEASE. Propionate is released much faster and leaves the system much faster than Enanthate or Cypionate, which is also why not all "Test is Test".

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WRONG AGAIN BRO. THE FASTER RATE OF RELEASE OF PROP IS INCONSEQUENTIAL. DO A SHOT OF CYP AND TAKE A BLOOD TEST A FEW HOURS LATER. YOUR T LEVELS WILL HAVE QUADRUPLED. DOES IT REALLY MATTER THAT PROP IS ACTIVE A FEW HOURS SOONER? IN THE LONG RUN, NO.


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Oh, and saying that Winstrol is stronger than Anadrol...LOL. It is just misleading! MILIGRAM FOR MILIGRAM, meaning at each drugs EFFECTIVE DOSAGE, Anadrol makes Winstrol look like BABYFOOD!

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YOU'RE CONFUSED.


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Who cares which drug requires a LOWER DOSAGE, each drug has a completely different chemical profile, this indicates NOTHING about the strength of the compound at the EFFECTIVE DOSAGE.

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MG PER MG COMPARISON MEANS EVERYTHING! IF I PUT TOGETHER A PILL THAT IS 500 MGS DOES THAT MAKE IT THE MOST POWERFUL COMPOUND? NO, IT'S JUST HIGHER DOSED.

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You crack me up... lol

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YOU'RE PRETTY FUNNY YOURSELF.

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It also seems that you do not undertand how to properly Bridge, and for who and what purpose it serves. Bridging is an essential part of cycling for any intermediate bodybuilder.

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THAT IS FLAT OUT TERRIBLE ADVICE.

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For a NOVICE, these guidelines should suffice.

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NICE TRY AT CONDESCENSION BUT YOU'RE MAKING YOURSELF LOOK SILLY. YOU SHOULD FIRST TRY AND GET A FEW THINGS PUBLISHED SO THAT SOME EDITORS CAN STRAIGHTEN YOU OUT AS TO WHAT IS ACCURATE AND WHAT IS JUST ACTING LIKE AN EXPERT. AFTER WHICH, YOU CAN THEN TRY AGAIN AND MAKE SOME POSTS WITH A BETTER UNDERSTANDING BEHIND WHAT YOU SAY. SOMEDAY YOU MAY TO ABLE TO MAKE SOME NICE CONTRIBUTIONS

>>

 

[Ross]

>>

Nelson, you should know my personality by now, argumentation is MY WAY of showing love. I have 2 books due to be published by ELITEFITNESS within a few months, so get ready my friend..

I think you know you're training pretty well, but if I am not mistaken, you're steroid EXPERIENCE is somewhat limited. Still, nothing but respect from me brother, like I said, I just love to argue, especially about things I know alot about.

 

[the_alcatraz]

>>

Bridging is bs. No such thing as bridging.

However, anadrol is definately stronger that winstrol. No comparision. Winstrol gives medium strength gains, whereas anadrol's gains are through the roof. Mg to mg, yes dbol is stronger than anadrol. But anadrol cannot even be compared to winstrol. One gives extreme superhuman strength and cause u to bloat and the other gives intermediate strength but REAL "bloat-free" lean muscle gains.

 

[Ross]

Bridging is bs. No such thing as bridging.

However, anadrol is definately stronger that winstrol. No comparision. Winstrol gives medium strength gains, whereas anadrol's gains are through the roof. Mg to mg, yes dbol is stronger than anadrol. But anadrol cannot even be compared to winstrol. One gives extreme superhuman strength and cause u to bloat and the other gives intermediate strength but REAL "bloat-free" lean muscle gains.

The Bridge
By Ross



Post-cycle regimens containing Aromatase Inhibitors and SERM's are simply not enough for the SERIOUS bodybuilder to maintain his muscular gains post-cycle. Once a bodybuilders reaches a certain point of muscular development, the continued use of a mild anabolic becomes justified..

The steroid user has TWO options:

1.) A Bridge
2.) A Cruise

In this chapter, we discuss the purpose of the BRIDGE.

The Bridge allows you to remain in an anabolic state while simultaneously having a MINIMAL intereference with HPTA function. Once you are FULLY RECOVERED and your PCT is complete, you can begin bridging while awaiting your FULL CYCLE. This will allow you to make GREATER THAN NATURAL GAINS, while still maintaining normal testosterone levels.

Bridging can ONLY be accomplished using a very specific and limited number of compounds. The selected compound must first be MINIMALLY supressive to the HPTA, and secondly, must still be healthy and effective in small dosages.

The following steroid combinations can be used effectively for Bridging.

Anavar/Proviron= 20mgs/25mgs
Anavar/Masteron= 20mgs/300mgs
Primobolan/Masteron= 200mgs/200mgs
Turinabol/Proviron= 30mgs/25mgs
Turinabol/Masteron= 30mgs/300mgs
Winstrol/Masteron= 25mgs/200mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/200mgs

**ADD AndroGenerator to COMPLETELY minimize HPTA inhibition!


NOT ALL ANDROGENS CAUSE SHUTDOWN*

"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.

SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)

Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.

The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
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Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links

Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.

Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.

We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS



[R]

 

[alphatest]

#6: Q: Should I use an anti aromatase during a cycle and a SERM afterward?

A: A little anti aromatase both during and after. Too much will hinder gains and health. SERMS are inconsistent, unreliable and have the potential for too many negative side effects including backlash of symptoms.

#8 Q: How long should I stay on?

A: One month minimum, 12 weeks maximum. Anything less is a waste, anything more may cause permanent suppression.

Do you have any data to support the above statements. Clearly studies on SERMs, especially clomid show that they raise test with minimal sides. I'm also interested in seeing data on the bodies recovery time based on length of cycle? Do you have blood work or is that statement based mostly on anecdotal evidence?

 

[Nelson Montana]

Nelson, you should know my personality by now, argumentation is MY WAY of showing love. I have 2 books due to be published by ELITEFITNESS within a few months, so get ready my friend..

I think you know you're training pretty well, but if I am not mistaken, you're steroid EXPERIENCE is somewhat limited. Still, nothing but respect from me brother, like I said, I just love to argue, especially about things I know alot about.

Well, pal, you can use lots of smiley faces and call me bro all day long but you tend to be a little disparaging in a passive aggressive way and frankly you're out of your league. You may have done a lot of gear but knowing what's best in all applications is a different story.

 

[al420]

#23 Q: I get gyno if I use Deca. What should I do?

A: Get some Cabaser.

I fixed it for you Nelson. You can thank me later.

Ross......

 

[get456]

Well, pal, you can use lots of smiley faces and call me bro all day long but you tend to be a little disparaging in a passive aggressive way and frankly you're out of your league. You may have done a lot of gear but knowing what's best in all applications is a different story.

I dunno NM, you seem to think Nolvadex creates horrible sides and should be avoided... just that fact alone makes me question most all of what you write.

In fact Ross actually made some good points IMO

 

[Mavafanculo]

Guys let's stick to debating and attacking ideas and opinions presented and not turn this into a drama thread.

The members reading this thread care about which ideas are best and can help them grow, not who's dick is bigger.

The personal stuff is for PM's.

 

[al420]

who's dick is bigger.

As a competitive Weightlifter I am sure my dick is the biggest. You should see me in my singlet.