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Has anyone tried Nelson Montana's Post Cycle Alternatives to Clomid?

I'm on week 2 of Clomid after a Winny (50mg/day) cycle.
Followed standard protocol.
The thing is I don't feel any different.
My nuts where fine during cycle, so does that mean I wasn't shut down?
How do you know if you need the therapy without getting blood tests? It seems like everyone is shooting in the dark. A kind of "it won't hurt: so lets take it anyway".
Well my libido HAS sufferered, don't know if thats due to the cycle or the clomid.
Do I finish the Clomid?
What are my alternatives?
 
EL GAUCHO said:
I'm on week 2 of Clomid after a Winny (50mg/day) cycle.
Followed standard protocol.
The thing is I don't feel any different.
My nuts where fine during cycle, so does that mean I wasn't shut down?
How do you know if you need the therapy without getting blood tests? It seems like everyone is shooting in the dark. A kind of "it won't hurt: so lets take it anyway".
Well my libido HAS sufferered, don't know if thats due to the cycle or the clomid.
Do I finish the Clomid?
What are my alternatives?

You´ve done a winstrol 50mg/ED cycle for some weeks, so you´ve inhibited your endogenous test production, period.
You have low test levels after the cycle, period.
Take Proviron during Clomid therapy to ease the transition.
 
Rio 2001 said:


You´ve done a winstrol 50mg/ED cycle for some weeks, so you´ve inhibited your endogenous test production, period.
You have low test levels after the cycle, period.
Take Proviron during Clomid therapy to ease the transition.
...........................................
This kinda doesn't make sense. Since Winny and Proviron are both derivitives of DHT it's sort of a continuation of the cycle. Winny won't cause gyno so why take something to prevent it? And why lower e with Clomid more than it needs to be lowered?

Clomid: A chastity belt in a pill.
 
Nelson, Clomid won´t lower E. I don´t know why do you insist on that...
It will prevent e receptors in the hypothalamus and pituitary to be activated by any e that is left in this low test/est ratio post cycle situation, leading hypothalamus to produce GnRh and Pituitaruy to produce LH.
There is a lot of evidence of Clomid raising LH, but there is still low evidence of that action significatively helping post cycle recovery. For some say yes, and others say no, and we dont have a prospective, randomized, controlled study to evaluate that . YET.

BTW, I´d like to add that not only Clomid does not lower E, it may as well increase it. Since increased LH will raise test production and Clomid has no anti A activity, E will actually RISE, which can respond for a lot of different symptoms in the Clomid therapy. Rising E during clomid won´t inhibit HPTA since HP receptors are occupied by Clomid ( antagonis-agonist action).This action of clomid in HPO is simmilar of it´s antagonist - agonist action in the breast,and there is this curious but illustrative case reporting Gynecomastia upon WITHDRAWAL of Clomid, and authors concluded that when breast E receptors got freed and E was high from Clomid, Gyno developed. So, I guess you can say even that " CLOMID CAUSE GYNO" and add some fire to this brain storm...lol...
 
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Rio 2001 said:
Nelson, Clomid won´t lower E. I don´t know why do you insist on that...
It will prevent e receptors in the hypothalamus and pituitary to be activated by any e that is left in this low test/est ratio post cycle situation, leading hypothalamus to produce GnRh and Pituitaruy to produce LH.
There is a lot of evidence of Clomid raising LH, but there is still low evidence of that action significatively helping post cycle recovery. For some say yes, and others say no, and we dont have a prospective, randomized, controlled study to evaluate that . YET.

BTW, I´d like to add that not only Clomid does not lower E, it may as well increase it. Since increased LH will raise test production and Clomid has no anti A activity, E will actually RISE, which can respond for a lot of different symptoms in the Clomid therapy. Rising E during clomid won´t inhibit HPTA since HP receptors are occupied by Clomid ( antagonis-agonist action).This action of clomid in HPO is simmilar of it´s antagonist - agonist action in the breast,and there is this curious but illustrative case reporting Gynecomastia upon WITHDRAWAL of Clomid, and authors concluded that when breast E receptors got freed and E was high from Clomid, Gyno developed. So, I guess you can say even that " CLOMID CAUSE GYNO" and add some fire to this brain storm...lol...


You're absolutely right Rio. But someties I over simplify because most people think of it as an "anti-e"

BTW: I've read studies that suggested Clomid lowered LH and FSH. The stuff sucks ass so hard....aah, don't get me started.
 
KA-BAR said:
I don't know about all the scientific mubo jumbo but I have tried not using it at end of cycle and using it and there is no comparison IMO. Clomid definitely works great for me. Brought my balls back alot faster and I had no sides while using it except my loads could put someone’s eye out. lol. I also like to run HCG, proviron and nolvadex at the end of cycle.

if you combined clomid with hcg, proviron, and nolvadex how can you assume that it was the clomid that brought you back? how do you know it wasnt the nolvadex (which many here say works great) coupled with the hcg?
i believe this is a common statement made by many here, which lead them to believe that clomid did the trick for them when in actuality it could very well have been the hcg and nolvadex and oh ya, proviron
 
just take the clomid, whats the big fuss, might as well take it, it wont harm u and take some avena sativa too
 
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