Please Scroll Down to See Forums Below
napsgear
genezapharmateuticals
domestic-supply
puritysourcelabs
UGL OZ
UGFREAK
napsgeargenezapharmateuticals domestic-supplypuritysourcelabsUGL OZUGFREAK

Bridging cycles with Primobolin

Not a big fan of bridging. It's just another way of saying that you are still on, but
at a very low level. Unless you compete and bodybuilding puts food on your table, I don't recommend bridging, regardless of the compound.
 
Anthony Starks said:
Nope, primo will still keep you shut down and you won't recover. 10mg Dbol in the AM is about the only way to bridge without keeping your HTPA suppressed, but even that's debateable.

not true!

primo at 100mg wont affect hpta..
in fact, some say that even 200mg a week wont affect hpta although thats pushing it.

100mg is totally safe.
 
Boston Strangler said:
so I should run my cycle, do pct and I can run primo between, or can I skip pct

pct and primo bridge should be ran together..
then, continue with primo only for a few weeks.
 
Do you have any studies supporting this? Any anabolic constantly in your system is going to suppress your HTPA, why would primo be any different?

satchboogie said:
not true!

primo at 100mg wont affect hpta..
in fact, some say that even 200mg a week wont affect hpta although thats pushing it.

100mg is totally safe.
 
Anthony Starks said:
Thats not a study, its some guy's opinion on a website. Even he admits that Primo will effect the body's natural testosterone production. If it is anabolic enough to give you gains, its going to be anabolic enough to suppress your recovery.

fair enough..
why dont you do some research and come back to us with conclusive evidence.. yes.. we all await :coffee:
 
Reseach that anabolics shut down your HTPA? Its all over the place. Why you think Primo is some wonder drug that operates completely differently is beyond me. Trust me satch, I wish you were right, but it just doesn't work that way.
 
Anthony Starks said:
Thanks Apex for the support. I'm just shocked that more users don't know enough to chime in on this!

I'm not saying satch doesn't know his shit. He's definitely one of the more
knowledgeable and helpful bros on the board.
I'm just not convinced by the link that he put up.
 
I'm not debating that Satch is helpful, but on this he certainly does not know his shit. So Apex, is your opinion is that Primo will not supress your HTPA too? Where are the mods/vets when you need them?!?
 
Bump for replies? Why is it that if you mention Ancillory Guys or R-Ala you have every mod and vet posting within 5 seconds but disagree with a "Platnum Member" and all of a sudden no one is around? Hmmmmmmm
 
Thats funny, still no replies. No Apex, no Satch, no mods/vets. Are you conceding that you are giving some terrible advice to the newbies on this board Satch? Why is no one rushing in to correct him?
 
I just get tired of everyone being afraid to argue with a "Platnum Member", even when they know he is wrong. There is a lot of dangerous BS information floating around on this board because no one will question certain "good bros". Notice how this thread just basically shut down, no one will back me up because they won't disagree with satch, and no one will back satch up because they know he is wrong. I have nothing against the guy, but this info could really mess with someone's PCT.
 
Maybe he has tried it and it works for him and others. :)

Me, personally I look at my off times as making sure all my levels are back to normal and see what actual gains are mine.

I just got my blood work checked out today to make sure I am ready for my up coming cycle, Starting Monday(Woo hoo).

I don't mind the off times, since I really don't feel like I'm losing anything that won't come in time. :chomp:
 
Anthony Starks said:
I just get tired of everyone being afraid to argue with a "Platnum Member", even when they know he is wrong. There is a lot of dangerous BS information floating around on this board because no one will question certain "good bros". Notice how this thread just basically shut down, no one will back me up because they won't disagree with satch, and no one will back satch up because they know he is wrong. I have nothing against the guy, but this info could really mess with someone's PCT.

Hey wise guy, it appears to me that he has given you the ball on this one, and isn't argueing with you any way. I think you need to chill meng, you're trying to stir up shit that doesn't need to be.

Mike
 
I'm not trying to stir up shit at all, or be a "wise guy". I'm just trying to get out the right information, and if he agrees with me he should say so, it would probably help unconfuse a lot of people.
 
Fair enough, just take it easy with calling people out...too many trolls do that just to start shit with longtime members here that's uncalled for. Discussing the information they post is another thing entirely, as long as you do it with out the bullshit..

It is good to discuss thing's though, so, as you were..
 
Back to the question at hand... I've bridged two ways, low dose of Deca/EQ, which may have still slightly suppressed HPTA and with Anavar this past year. THat stuff really surprised me. I was expecting to just "maintain" on it, but I made surprising mass and strength gains over a few months on it, not dramatic, like with Dbol or something, but very impressive. Plus no water bloat and it's not supposed to supress HPTA.
 
ATLmuscles said:
Plus no water bloat and it's not supposed to supress HPTA.

Jesus christ, see what I mean?!? Here is another "good bro" with some very bad info. And I guarentee no one will speak up and tell him that he is wrong, they will just not post anything and ignore it. Unfortunatly, that is how these ideas stay alive.
 
Anthony Starks said:
Jesus christ, see what I mean?!? Here is another "good bro" with some very bad info. And I guarentee no one will speak up and tell him that he is wrong, they will just not post anything and ignore it. Unfortunatly, that is how these ideas stay alive.
He is wrong, anavar will supress, doesnt matter how much, it still will.
 
I agree with Anthony in the point that if you have a steroid in your system causing anabolic effects, should by theory keep your hpta affected. Now, since satch has a lot of info about primo, if there's a dose that is "safe" for your hpta, maybe is not enough anabolic to keep the gains and at the end will do nothing. These are my conclusions, I'm starting to read about primo in these days. But regarding these points I think you can use a low dose for bridge, keepin your hpta down or you can use a lower dose keeping your hpta safe but without anabolic effects. Am I right? Anyway I'd like to see studies and proof of both partys...
 
Anthony Starks said:
Thats funny, still no replies. No Apex, no Satch, no mods/vets. Are you conceding that you are giving some terrible advice to the newbies on this board Satch? Why is no one rushing in to correct him?

All right, I'm back. I left work and it's kinda hard to log on at home without my girl asking me why I know so much about steroids if I don't do them.

I've read what are basically opinions for both sides of this arguement, however, I've never seen an actual study. Satch has way more experience with primo than I do, but I personally feel that he's wrong on this one. In my educated opinion, all steroids effect the body's abitlity to create it's own natural testosterone......at some level.
Now the question here is primo safe to bridge with. I'll go back to my original statement about bridging. Using primo is still being "on", unless you're stepping on stage, DON'T DO IT.

If anyone out there has any studies that they could direct me to, i'll send
some green your way.

Oh yeah, Starks, you gotta lighten up bro. We're all on the same team here.
and thanks for the green, I'll be hittin you back with some.
 
i agree with ya bro........ALL roids affect hpta function.......even the suggestion you made about dbol too.Bridging has its uses but anyone who thinks its not affecting natural test production is wrong.

And not that i wanna get into a beef with these guys ,but its not hard to see why satch promotes primo and that 101 site,or why some of the mods are all over the "sponsor" threads.lol.think about it.

and are you the same dude from M/T.........your name`s mighty familiar?
 
macdaddy-1 said:
i agree with ya bro........ALL roids affect hpta function.......even the suggestion you made about dbol too.Bridging has its uses but anyone who thinks its not affecting natural test production is wrong.

And not that i wanna get into a beef with these guys ,but its not hard to see why satch promotes primo and that 101 site,or why some of the mods are all over the "sponsor" threads.lol.think about it.

and are you the same dude from M/T.........your name`s mighty familiar?

I agree with everything you are saying, thanks for having the balls to come out and say it. I'm not the same guy from M/T, but I'm not surprised there is another Tony Starks floating around. Its the name of the alter ego for the comic book character "Ironman".
 
if i had a few hours to spare, i'd find more than a few studies showing that 100mg primo per week wont affect hpta...

but work and pleasure takes most of my day so ill pass.

so if you dont wanna take my word, thats fine.
disagreee all you want my friends..

1+1 is still 2 and you can argue that all day long.
 
Mikus said:
Hey wise guy, it appears to me that he has given you the ball on this one

I guess not, huh? And I guess all the mod's computers are down or something, because this thread is invisable to them.
 
The thing about supression is that it's not an all or nothing situation
Deca can supress 100% within a very short period of time, as can tren
Test is suppressive, but not quite as bad
Var is only mildly supressive, as is primo etc.....etc

....What this means is that there will be a certain dosage at which any AS will supress the HPTA by less than 100% -

....You could argue that an HPTA supression of less than, say, 25% won't cause too many problems for an individual -

The question is what AS / doses could be used to help retain muscle, but keep HPTA supression at or below 25%?

100mg Primo EW will be probably be supressive, but only mildly, as would say 15mg Var ED
I think you'll find a few drinks will cause more supression than these small doses of 'mild' AS -
I'm not defending bridgeing ( I don't bridge)- I just think we need to think in quantitive terms and not just say something IS or ISN'T supressive - exactly HOW HARD it shuts you down is the key - if you're only 25% shut down by 100mg primo EW then your clomid & nolva etc can still do alot of its work to get natty test going again....

(obviously all these figures are made up)

ps. Tony - I get what you're saying, but chill out - stress is the enemy! :supercool
 
Anthony Starks said:
Reseach that anabolics shut down your HTPA? Its all over the place. Why you think Primo is some wonder drug that operates completely differently is beyond me. Trust me satch, I wish you were right, but it just doesn't work that way.

last thing i mention before i let this thread evaporate...

you joined elite not long ago.. what a few weeks back?
ok... well some of us have been around for a while and have seen many many many threads about this.

trust me this isnt a new issue.
its unfortunate that a few very solid bros whose names i wont mention had to leave this board for personal reasons. bros that would have posted these studies you're after in a few seconds.

your knowledge of gear is quite limited.. if even that!

you may know how to train... but we dont even know that as you never posted your pics.

so why dont you find a rock to hind under for the time being..

truly yours,

boogieman.
 
satchboogie said:
last thing i mention before i let this thread evaporate...

you joined elite not long ago.. what a few weeks back?
ok... well some of us have been around for a while and have seen many many many threads about this.

trust me this isnt a new issue.
its unfortunate that a few very solid bros whose names i wont mention had to leave this board for personal reasons. bros that would have posted these studies you're after in a few seconds.

your knowledge of gear is quite limited.. if even that!

you may know how to train... but we dont even know that as you never posted your pics.

so why dont you find a rock to hind under for the time being..

truly yours,

boogieman.

Its a shame that ,you of all people satch, need to resort to bitchy remarks bro.This guy aint no guru....and neither am i.....but we come to these boards to debate what we have learned,improve,and learn more.And we look to people such as yourself to enlighten us..........saying "i havent the time" to post the studies you want is a cop out!Either you wanna help out or you dont.If you dont then why cruise round these places acting like youre "all that" mate.I understand the reasons why you frequent these places bro,TRUST ME,but if all you wanna do is bash on people...why bother?Youre supposed to be a knowledgeable bro......so act like one and prove it.
 
satchboogie said:
last thing i mention before i let this thread evaporate...

you joined elite not long ago.. what a few weeks back?
QUOTE]

Good reply MacDaddy. By the way, I used to post on Elite a lot a couple of years ago with a different user name. How are you such a guru if I never saw you on here even a couple years ago? Go sell your primo somewhere else Satch and stop acting like you know the first thing about this topic.
 
i just got a boner reading this thread

listen, its a well known fact that using aas does NOT affect your hpta. 5 grams of test a week will not shut you now

:)

are people this fucking stupid? when you introduce a synthetic form of a hormone into your body, your body will begin to stop making it on its own. what's the point of bridging with anything less than what your hpta would produce anyway? actually, what's the point of bridging at all? if youre gonna be supressed you may as well go for the gold and just stay on eh?
 
satchboogie said:
if i had a few hours to spare, i'd find more than a few studies showing that 100mg primo per week wont affect hpta...

but work and pleasure takes most of my day so ill pass.

so if you dont wanna take my word, thats fine.
disagreee all you want my friends..

1+1 is still 2 and you can argue that all day long.
I'd like to see you find a study satch. I'd really like that. I'm sure you can prove us all wrong and take an hour of your precious time to find a study.
 
yomama said:
I'd like to see you find a study satch. I'd really like that. I'm sure you can prove us all wrong and take an hour of your precious time to find a study.

fair enough..
ill get on it soon.
 
satchboogie said:
fair enough..
ill get on it soon.

While we are all holding our breath waiting for you to "get on it", here is Bill Robert's opinion. If you won't take my opinion, maybe you will take his and admit you don't know what the hell you are talking about. Just in case you are not a big reader, don't miss this quote: "Any time you have something that is an agonist (activator) of the AR it is going to be somewhat suppressive."

"Primo Depot and inhibition

Hey Bill—

I wanted to know at what dosage and duration does Primo Depot typically cause inhibition. I know for first time users you recommend about 400mg/week and start with 800mg week 1, but the only anecdotal use I saw was at 200mg/week for maxed out natural resulting in 17 pound gain.

Well, that’s a nice result!

Perhaps at this dosage suppression is minimal so endogenous Test. works concurrently or is this the exception to the rule?

Any time you have something that is an agonist (activator) of the AR it is going to be somewhat suppressive. But you are right, I think 200 mg/week Primo is low enough to be only partially suppressive, so some natural testosterone certainly would be in there. In fact I think 400 mg is only partially suppressive, though moreso of course."
 
Anthony Starks said:
Jesus christ, see what I mean?!? Here is another "good bro" with some very bad info. And I guarentee no one will speak up and tell him that he is wrong, they will just not post anything and ignore it. Unfortunatly, that is how these ideas stay alive.

Dude, you seem really bitchy! Having "Platinum" by someone's name does not mean people are afraid to question them; all it means is I paid a few bucks to get some additional benefits of the web site.

Now, where do you do your research?? In regards to var:

From "Chemical Muscle Enhancement: "avg dose, men: 20-50 mg daily...Decreases HPTA function: Unlikely even in high doses."

From "Anabolics 2002:"
"Studies using low doses of this compund note minmal interferences with natural testostersone production. Likewise when it is used alone in small amounts there is typically no need for ancillary drugs like CLomid/Nolvadex or HCG."

I don't go spouting off like some know-it-all, but I do lots of research on this stuff, beyond heresey on a discussion board. SO cool your jets!
 
I agree that "low doses" of anavar will have "minimal" interferrences with natural test, but the fact that it will interfere with natural test AT ALL means that you are not doing anything but prolonging your cycle when you try to bridge with it. At the end of a cycle your test levels are extremely low, the last thing you need is another AS that is going to interfere with their rebound.
 
ATLmuscles said:
Back to the question at hand... I've bridged two ways, low dose of Deca/EQ

Kind of off topic, but where did you get the idea that this was a good bridge? I feel bad for your body when you finally end this one gigantic cyle you have been on.
 
ATLmuscles said:
Dude, you seem really bitchy! Having "Platinum" by someone's name does not mean people are afraid to question them; all it means is I paid a few bucks to get some additional benefits of the web site.

Now, where do you do your research?? In regards to var:

From "Chemical Muscle Enhancement: "avg dose, men: 20-50 mg daily...Decreases HPTA function: Unlikely even in high doses."

From "Anabolics 2002:"
"Studies using low doses of this compund note minmal interferences with natural testostersone production. Likewise when it is used alone in small amounts there is typically no need for ancillary drugs like CLomid/Nolvadex or HCG."

I don't go spouting off like some know-it-all, but I do lots of research on this stuff, beyond heresey on a discussion board. SO cool your jets!
Chemical Muscle enhancement???? Post a link for a complete study. That's a bunch of crap that it doesnt suppress at 50mg. Bullshit.
 
adam wj said:
I'd still like to know why people bridge if they ever have intentions of comming off.

Someone enlighten me

Really

:)
It's an addiction. :evil: Cycles longer than 4months suck, for some reason my tendons start to bother me, my fuckin knee mostly. I got it scanned a year ago and it showed minor tears, since then it healed but now it came back. So I have no choice but to stop cycle and have it recover.
 
yomama said:
It's an addiction. :evil:

At least your honest about it.

Jenetic
 
well i think this is the same idea of proviron.....supposidly proviron will keep u suppressed if already suppressed...but when taken when test levels are normal it doesnt suppress....maybe its the same with primo..idk...maybe thats why primo is so easy to recover from??....im not scientist and i dont use these methods but im throwing things out there
 
heres a study on proviron..still lookin on the primo:
Oral synthetic 17-a alkylated androgens such as methyltestosterone (96), fluoxymesterone (97), methandienone (98) and danazol (99, 100) suppress spermatogenesis but azoospermia is rarely achieved and the inherent hepatotoxicity of the 17-a alkyl substitutent (101) renders them unsuitable for long-term use. Athletes self-administering supratherapeutic doses of androgens also exhibit suppression of spermatogenesis (98, 102). Synthetic androgens lacking the 17-a alkyl substituent have been little studied although injectable nandrolone esters produce azoospermia in 88% of European men (103, 104) whereas oral mesterolone is ineffective (105). On the other hand, nandrolone hexyloxyphenylpropionate alone was unable to maintain spermatogenic suppression induced by a GnRH antagonist (106) in a prototype hybrid regime (where induction and maintenance treatment differ) whereas testosterone appears more promising (107). A 7-methyl derivative of nandrolone (MENT), which is partly aromatisable but resistant to 5α reductive amplification of androgenic potency, has been studied as a non-oral androgen for hormonal male contraceptive regimens (108). While it is prostate-sparing (109), dose titration to achieve essential androgen replacement at each relevant tissue is more complex than for testosterone and may be difficult to achieve (110). More potent, synthetic androgens lacking 17-a alkyl groups (111, 112) remain to be fully evaluated.

97. Jones TM, Fang VS, Landau RL, Rosenfield RL 1977 The effects of fluoxymesterone administration on testicular function. J Clin Endocrinol Metab 44:121-129

98. Holma PK 1977 Effects of an anabolic steroid (metandienone) on spermatogenesis. Contraception 15:151-162

99. Skoglund RD, Paulsen CA 1973 Danazol-testosterone combination: a potentially effective means for reversible male contraception. a preliminary report. Contraception 7:357-365

100. Sherins RJ, Gandy HM, Thorslund TW, Paulsen CA 1971 Pituitary and testicular function studies. I. Experience with a new gonadal inhibitor, 17a-pregn-4-en-20-yno-(2,3-d) isoxazol-17-ol (Danazol). J Clin Endocrinol Metab 32:522-531

101. Ishak KG, Zimmerman HJ 1987 Hepatotoxic effects of the anabolic-androgenic steroids. Semin Liver Dis 7:230-236

102. Knuth UA, Maniera H, Nieschlag E 1989 Anabolic steroids and semen parameters in bodybuilders. Fertil Steril 52:1041-1047

103. Knuth UA, Behre H, Belkien L, Bents H, Nieschlag E 1985 Clinical trial of 19-nortestosterone hexoxyphenylpropionate (Anadur) for male fertility regulation. Fertil Steril 44:814-821

104. Schurmeyer T, Knuth UA, Belkein L, Nieschlag E 1984 Reversible azoospermia induced by the anabolic steroid 19-nortestosterone. Lancet 1:417-420

105. Schellen TNCM, Beek JMJHA 1972 The influence of high doses of mesterolone on the spermiogram. Fertil Steril 23:712-714

106. Behre HM, Kliesch S, Lemcke B, von Eckardstein S, Nieschlag E 2001 Suppression of spermatogenesis to azoospermia by combined administration of GnRH antagonist and 19-nortestosterone cannot be maintained by this non-aromatizable androgen alone. Hum Reprod 16:2570-7

107. Swerdloff RS, Bagatell CJ, Wang C, Anawalt BD, Berman N, Steiner B, Bremner WJ 1998 Suppression of spermatogenesis in man induced by Nal-Glu gonadotropin releasing hormone antagonist and testosterone enanthate (TE) in maintained by TE alone. J Clin Endocrinol Metab 83:3527-33

108. Sundaram K, Kumar N 2000 7alpha-methyl-19-nortestosterone (MENT): the optimal androgen for male contraception and replacement therapy. Int J Androl 23 Suppl 2:13-5

109. Cummings DE, Kumar N, Bardin CW, Sundaram K, Bremner WJ 1998 Prostate-sparing effects in primates of the potent androgen 7alpha-methyl-19-nortestosterone: a potential alternative to testosterone for androgen replacement and male contraception. Journal of Clinical Endocrinology & Metabolism 83:4212-9

110. Anderson RA, Wallace AM, Sattar N, Kumar N, Sundaram K 2003 Evidence for tissue selectivity of the synthetic androgen 7 alpha-methyl-19-nortestosterone in hypogonadal men. J Clin Endocrinol Metab 88:2784-93

111. Avery MA, Tanabe M, Crowe DF, Detre G, Peters RH, Chong WKM 1990 Synthesis and testing of 17ab-hydroxy-7a methyl-D-homoestra-4,16-dien-3-one: a highly potent orally active androgen. Steroids 55:59-64

112. Grootenhuis AJ, de Gooyer ME, Louw J, Bursi R, Leysen D 2004 Synthesis and pharmacological profiling of new orally active steroidal androgens. In: Nieschlag E, Behre HM (eds) Testosterone: Action, Deficiency and Substitution, 3rd ed. Springer-Verlag, Berlin, pp 665-84
 
Last edited:
bicepts101 said:
heres a study on proviron..still lookin on the primo:
Oral synthetic 17-a alkylated androgens such as methyltestosterone (96), fluoxymesterone (97), methandienone (98) and danazol (99, 100) suppress spermatogenesis but azoospermia is rarely achieved and the inherent hepatotoxicity of the 17-a alkyl substitutent (101) renders them unsuitable for long-term use. Athletes self-administering supratherapeutic doses of androgens also exhibit suppression of spermatogenesis (98, 102). Synthetic androgens lacking the 17-a alkyl substituent have been little studied although injectable nandrolone esters produce azoospermia in 88% of European men (103, 104) whereas oral mesterolone is ineffective (105). On the other hand, nandrolone hexyloxyphenylpropionate alone was unable to maintain spermatogenic suppression induced by a GnRH antagonist (106) in a prototype hybrid regime (where induction and maintenance treatment differ) whereas testosterone appears more promising (107). A 7-methyl derivative of nandrolone (MENT), which is partly aromatisable but resistant to 5α reductive amplification of androgenic potency, has been studied as a non-oral androgen for hormonal male contraceptive regimens (108). While it is prostate-sparing (109), dose titration to achieve essential androgen replacement at each relevant tissue is more complex than for testosterone and may be difficult to achieve (110). More potent, synthetic androgens lacking 17-a alkyl groups (111, 112) remain to be fully evaluated.
I dont get what the point of this study is.
 
ok heres something on primo...it has no reference so im sure you wont accept it:

For example, in one study more than half of the patients receiving only 30-45 mg noted a suppression of gonadotropin levels of 15% to 65% a. This is a dose far less than most bodybuilders would use, and no doubt increasing it would only lead to worse suppression. One would therefore still need a testosterone stimulating drug like HCG or Clomid®/Nolvadex® when concluding a low-dose Primobolan® cycle, unless a deliberately small dose were being used.

basically this say what ive always heard...it will be easier to recover from cause it doesnt shut you down too hard
 
heres some info on primo if anyones interested:

This section refers to the oral Primobolan® preparation, which contains the drug methenolone acetate. It is very similar in action to the injectable Primobolan® Depot (methenolone enanthate), but obviously here the drug is designed for oral administration. At one time Schering was in fact also manufacturing an injectable methenolone acetate (Primobolan® acetate, out of manufacture since 1993), which proved to be very useful for pre-contest cutting purposes. This steroid is now gravely missed, as it was once a favorite among European competitors. Although we still have the acetate in oral form, it is a close, but not equal substitute (injection is a much more efficient form of delivery for this steroid).

Methenolone regardless of the ester is a very mild anabolic steroid. The androgenic activity of this compound is considerably low, as are its anabolic properties. One should not expect to achieve great gains in muscle mass with this drug. Instead, Primobolan® is utilized when the athlete has a specific need for a mild anabolic agent, most notably in cutting phases of training. It is also a drug of choice when side effects are a concern. A welcome factor is that Primobolan® is not c17 alpha alkylated as most oral steroid are. Due to the absence of such an alteration, this compound is one of the few commercially produced oral steroids that is not notably stressful to the liver. While liver enzymes values have been affected by this drug in some rare instances, actual damage due to use of this substance is not a documented problem. Unfortunately the 1 alkylation and 17-beta esterification of Primobolan® do not protect the compound very well during first pass however, so much of your initial dose will not make circulation. This is obviously why we need such high daily dose with the oral version of Primobolan®.

Primobolan® will also not aromatize, so estrogen related side effects are of no concern. This is very useful when leading up to a bodybuilding contest, as subcutaneous water retention (due to estrogen) can seriously lessen the look of hardness and definition to the muscles. Non-aromatizing steroids are therefore indispensable to the competitor, helping to bring about a tight, solid build the weeks leading up to a show. And of course without excess estrogen there is little chance of the athlete developing gynecomastia. Likewise there should never be a need for anti-estrogen use with this steroid. Primobolan® is also said to have a low impact on endogenous testosterone production. Although this may well be true in small clinical doses, it will not hold true for the bodybuilder. For example, in one study more than half of the patients receiving only 30-45 mg noted a suppression of gonadotropin levels of 15% to 65% a. This is a dose far less than most bodybuilders would use, and no doubt increasing it would only lead to worse suppression. One would therefore still need a testosterone stimulating drug like HCG or Clomid®/Nolvadex® when concluding a low-dose Primobolan® cycle, unless a deliberately small dose were being used.

It is also important to note that although the androgenic component of Primobolan® is low, side effects are still possible. One may therefore notice oily skin, acne and facial/body hair growth during treatment. Men with a predisposition for hair loss may also find it exacerbates this condition, and wish to avoid this item (nandrolone injectables are a much better choice). While always possible, side effects rarely reach a point where they interfere with the progress of cycle. Primobolan® is clearly one of the milder and safer oral steroids in production. Female athletes, older or more sensitive individuals and steroid beginners will no doubt find this a comfortable steroid to experiment with.

The dosage for men is somewhere in the range of 75-150mg daily. This can obviously be tedious (and costly) if one can only obtain the 5mg tablets from Mexico and S. America. A mild anabolic such as Primobolan® is often used in conjunction with other steroids for optimal effect, so some users find a slightly lower dose effective when stacking. During a dieting or cutting phase, thought to be its primary application, a non-aromatizing androgen like Halotestin® or trenbolone can be added for example. Such combinations would enhance the physique without water retention, and help bring out a harder and more defined look of muscularity. Non-aromatizing androgen/anabolic stacks like this are in fact very popular among competing bodybuilders. This compound is also occasionally used with more potent androgens during bulking phases of training. The addition of testosterone, Dianabol or Anadrol 50® would prove effective for instance, although the gains are likely to be accompanied by some level of smoothness due to the added estrogenic component.

Among women, Primobolan® is one of the most popular steroids in use. At a dosage of 50-75mg daily, virilization symptoms are extremely uncommon. One would of course not expect a tremendous amount of muscle mass with this drug, and instead should expect a slow and steady (quality) increase. Some women choose to further add-in other anabolics such as Winstrol® or oxandrolone, in an effort to increase the muscle building effectiveness of a cycle. While both of these compounds are quite tolerable to women, one must be sure not to use too high an accumulated dosage. Troublesome androgenic side effects are always a possibility with steroid use, even with very mild substances. Taken at too high a dosage, these weak anabolics can become a formidable danger to femininity. It would therefore be the best advice not to use the normal dosage range of both, but instead start with a much lower dosage of each steroid to compensate for the other. On the black market Primobolan® orals are popular, but still much less commonly found than the injectable. This is due to the higher cost effectiveness of the injectable, which uses the same active compound but with 100% bioavailability due to the form of administration.
 
Sorry to bring back a post over two weeks old, but I was wondering if everyone still thinks Primo, or any other AS for that matter, will not suppress your HTPA.
 
Top Bottom