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napsgear
genezapharmateuticals
domestic-supply
puritysourcelabs
RESEARCHSARMSUGFREAKeudomestic
napsgeargenezapharmateuticals domestic-supplypuritysourcelabsRESEARCHSARMSUGFREAKeudomestic

ATD is it useless afterall?

you are reposting the same studies.

the first with the same erroneous conclusion. ATD does not inhibit T binding, suppression of oestrogen reduces AR receptor expression in those tissues.

with respect to your claims about 17a-methyl-17b-hydroxyl-3-keto-delta 1,4,6-etioallocholtriene (taken directly from ALRI who developed this compound), there are no studies to back up these claims.

with respect to RIA anaylsis, its been proven to be quite innaccurate.
 
macrophage69alpha said:
you are reposting the same studies.

the first with the same erroneous conclusion. ATD does not inhibit T binding, suppression of oestrogen reduces AR receptor expression in those tissues.

with respect to your claims about 17a-methyl-17b-hydroxyl-3-keto-delta 1,4,6-etioallocholtriene (taken directly from ALRI who developed this compound), there are no studies to back up these claims.

with respect to RIA anaylsis, its been proven to be quite innaccurate.
Well there also no studies proving any of this wrong. We should have the UCLA study in the next few weeks. Now once that is in there really is no disputing it. University certified and published.

Take Triple J from above. There are lots and lots of users reporting this every day.

WE SELL ATD, but feel it may be unfit topical or oral for post cycle or standalone. I can post voicemail after voice mail from customers reporting these sides. - none with mATD.
 
AX_Ryan said:
Well there also no studies proving any of this wrong. We should have the UCLA study in the next few weeks. Now once that is in there really is no disputing it. University certified and published.

what kind of logic is that: there are no studies proving it wrong so its true???????


as far as your study:
what peer reviewed journal is it being published in?

how exactly did you get the human subjects review board (IRB) to approve use on humans?

Use of a compound that is actually illegal under the DSHEAS? (btw- dont care about this but any university would)




oh and BTW- one study does not make claims irrefutable.
 
What studies have you done to show ATD is usefull and backs up its claims.

1. All of the info will be in the study.

2. You have to give it to AX for dishing out 40k for a study because consumers are calling in daily with negative reports on ATD.

3. They do not care about DSHEA but ATD and mATD are DSHEA compliant and both have been filed by AX. Same with Phera-Plex.
 
AX_Ryan said:
What studies have you done to show ATD is usefull and backs up its claims.

1. All of the info will be in the study.

2. You have to give it to AX for dishing out 40k for a study because consumers are calling in daily with negative reports on ATD.

3. They do not care about DSHEA but ATD and mATD are DSHEA compliant and both have been filed by AX. Same with Phera-Plex.

A. ATD is a clinically and pharmacologically proven steroidal aromatase inhibitor, albeit one with poor oral bioavailability.

1. ok, so a non answer
2. not really, its an advertising ploy. Just like the 6-oxo study, never actually published. Like the ZMA study, never actually published, etc.etc..
3. they are not in the database
 
btw- M-atd may be effective (regardless of the study- the drawbacks of any study merely being pointed out given the nature of the compound and the restrictions of university testing as well as peer review issues) but a number of erroneous statements have been regarding the efficacy and action of ATD. ATD is a proven steroidal AI backed by over 100 published and peer reviewed studies.
 
macrophage69alpha said:
btw- M-atd may be effective (regardless of the study- the drawbacks of any study merely being pointed out given the nature of the compound and the restrictions of university testing as well as peer review issues) but a number of erroneous statements have been regarding the efficacy and action of ATD. ATD is a proven steroidal AI backed by over 100 published and peer reviewed studies.
Sure I can agree with you on that. It used to be our number one AI. Just recently we began to question the facts that other companies (big ones) were publishing studies that claimed huge libido increases when used standalone. We were seeing the exact opposite from our customers.

The questions as stated above we are getting aswers for are:

1) If testosterone is the hormone that makes us horny (increased libido), then why is it that users with testosterone levels at 400-600% above normal have such a low libido? It is very common for people using ATD products to completely lose their sex drive.

2) Since ATD is such a wonderful anti-aromatase (anti-estrogen), we need to ask why so many users are reporting gynocomastia? (Gyno is caused by high estrogen levels or hormone environments that allow estrogen activity to exceed testosterone activity). Gyno on ATD standalone or post cycle.

Thank you for being polite throughout..........
 
1. oestrogen suppression is the cause of loss of libido. Its quite common for those on cycle taking a gram of test a week to get libido loss when using letrozole (why? oestrogen suppression)

2. mainly because they are getting gynecomastia from compounds that do not aromatize, like superdrol, m1t, pheraplex, 1test, etc... as a note taking an aromatase inhibitor with a compound like this (or one like winstrol, tren, alone) will destroy your lipids and libido. And you need to keep in mind that there was at least one bad batch of RXT, eventually, recalled by designer supps.
 
macrophage69alpha said:
And you need to keep in mind that there was at least one bad batch of RXT, eventually, recalled by designer supps.
True but never by AX. And AX has sold 10X more than DS mainly in retail stores. We are just finding these things out from AX customers. Most people are running it stand alone and getting these sides (gyno, libido)

Of course anyone taking a anabolic compound like the compounds you named run the risk of getting gyno.
 
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