winny is way more toxic and stresses out the liver a lot more, is known to throw your cholesterol levels out of whack, and I remember someone posted some medical studies here against winny if you want to search that. So if it was between those two C-17 steroids, I would chose anavar.. and anyway the real "cutter" is diet, cardio, and workout plan.
Here's a case report where a Bodybuilder who used normal dosages (nothing insane) and got wild liver damage, and i would think winny would be the likely culprit out of those three aas:
It's a case report, so is it possible the bodybuilder didn't tell everything he was taking? Just thought it was worth pondering if anyone knows about this report and can comment.
SS
Here's a case report where a Bodybuilder who used normal dosages (nothing insane) and got wild liver damage, and i would think winny would be the likely culprit out of those three aas:
Androgenic/Anabolic steroid-induced toxic hepatitis.
Stimac et al. - J Clin Gastroenterol 2002
When used in therapeutic doses, these drugs produce clinical jaundice in just a small number of recipients. We present a 26-year-old male bodybuilder who self-administered high doses of androgenic/anabolic steroids that induced liver damage. One month before admission to the hospital, he used testosterone enanthate (500 mg intramuscularly, twice weekly), stanozolol (40 mg/d), and methylandrostenediol (30 mg/d by mouth, for 5 weeks). On admission, his bilirubin level was 470 micromol/L (direct, 360 micromol/L), his aspartate aminotransferase (AST) level was 5,870 IU/L, his alanine aminotransferase (ALT) level was 10,580 IU/L, his alkaline phosphatase (ALP) level was 152 IU/L, his gamma-glutamyl-transpeptidase level was 140 IU/L, his albumin level was 27.6 g/L, and his prothrombin time was 29%. During the patient's prolonged hospitalization, multiple tests and liver biopsy were performed, showing only toxic hepatic lesions. The patient was provided with supportive medical treatment. Clinical signs and laboratory findings improved substantially 12 weeks after the patient discontinued androgenic/anabolic steroids. The reasons for presenting this case were the much higher values of AST and ALT levels than reported in other studies, although the values of bilirubin and ALP were similar to those found in the literature. To our knowledge, it is the first case of toxic hepatitis induced by androgenic/anabolic steroids with predominantly hepatocellular necrosis instead of intrahepatic cholestasis.
It's a case report, so is it possible the bodybuilder didn't tell everything he was taking? Just thought it was worth pondering if anyone knows about this report and can comment.
SS