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The Bridge
By Ross
Post-cycle regimens containing Aromatase Inhibitors and SERM's are simply not enough for the SERIOUS bodybuilder to maintain his muscular gains post-cycle. Once a bodybuilders reaches a certain point of muscular development, the continued use of a mild anabolic becomes justified..
The steroid user has TWO options:
1.) A Bridge
2.) A Cruise
In this chapter, we discuss the purpose of the BRIDGE.
The Bridge allows you to remain in an anabolic state while simultaneously having a MINIMAL intereference with HPTA function. Once you are FULLY RECOVERED and your PCT is complete, you can begin bridging while awaiting your FULL CYCLE. This will allow you to make GREATER THAN NATURAL GAINS, while still maintaining normal testosterone levels.
Bridging can ONLY be accomplished using a very specific and limited number of compounds. The selected compound must first be MINIMALLY supressive to the HPTA, and secondly, must still be healthy and effective in small dosages.
The following steroid combinations can be used effectively for Bridging.
Anavar/Proviron= 20mgs/25mgs
Anavar/Masteron= 20mgs/300mgs
Primobolan/Masteron= 200mgs/200mgs
Turinabol/Proviron= 30mgs/25mgs
Turinabol/Masteron= 30mgs/300mgs
Winstrol/Masteron= 25mgs/200mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/200mgs
**ADD AndroGenerator to COMPLETELY minimize HPTA inhibition!
NOT ALL ANDROGENS CAUSE SHUTDOWN*
"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.
SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Tbol, Var, Wistrol, EQ, Dianabol, masteron, proviron, halo, primo)
Very Androgenic/Progestenic/Estrogenic steroids(Tren, Deca, Drol, Test) cause a COMPLETE shutdown of endogenous hormone production.
The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier PCT.
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Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links
Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.
Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.
We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS
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