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Stew Meat
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I must have missed something....
Where did it mention anything about a potential for stronger muscular contractions? Central Nervous System stimulation would have nothing to do with muscular function as it consists of only the brain and spinal cord... Periphial Nervous System is what regulates muscular contraction. And I don't have my physiological psychology book handy, but it seems to me that GABA function is a counter measure against chaotic impulses...
Nerve cells, or neurons, communicate by releasing neurotransmitters. These chemical messengers flow onto other neurons that act as receivers. The neurotransmitter attaches to a slot on the neuron, or receptor site. Once attached different neurotransmitters either trigger "go" signals that allow the message to be passed on to other cells or produce "stop" signals that prevent the message from being forwarded. GABA is the most common message-altering neurotransmitter in the brain.
The brain has to keep tight control of this message delivery system to avoid communication chaos. A single receiving neuron has thousands of receptor sites and receives many different messages and passwords at once. Each neuron adds up the incoming signals and determines whether or not to pass the information along to other cells. Enzymes help out by patrolling the brain and eliminating excess message-halting GABA to ensure a balance in communication.
Normally each neuron affects only a limited number of other cells. If a sufficient amount of GABA is lacking, however, the system goes out of whack, and tens of thousands of neurons send messages rapidly, intensely and simultaneously, resulting in a seizure. This is actually the primary cause of epilepsy... a lack of sufficient enzymes to keep GABA levels up.
Benzodiazepine is another neurotransmitter that binds to the exact same dendritic receptor as GABA. This receptor is known as the GABA receptor as GABA is the primary neurotransmitter to bind there. GHB also binds to the GABA receptor btw. Anywho, the study showed that stanozol can actually increase the GABA receptor's affinity toward benzodiazepine neuroreceptor. Benzodiazepine as I recall has a "chilling" affect on the CNS meaning that stanozol may actually reverse halo's tendency toward roid rage but I don't see anything that would cause a stimulation of muscle nerves in the PNS... It would probably only acheive this through AR-mediation (which is its primary anabolic means).
It is my understanding that this would only have psychological effects... effects that are not seen with other androgenic/anabolic steroids.
-Stew
Where did it mention anything about a potential for stronger muscular contractions? Central Nervous System stimulation would have nothing to do with muscular function as it consists of only the brain and spinal cord... Periphial Nervous System is what regulates muscular contraction. And I don't have my physiological psychology book handy, but it seems to me that GABA function is a counter measure against chaotic impulses...
Nerve cells, or neurons, communicate by releasing neurotransmitters. These chemical messengers flow onto other neurons that act as receivers. The neurotransmitter attaches to a slot on the neuron, or receptor site. Once attached different neurotransmitters either trigger "go" signals that allow the message to be passed on to other cells or produce "stop" signals that prevent the message from being forwarded. GABA is the most common message-altering neurotransmitter in the brain.
The brain has to keep tight control of this message delivery system to avoid communication chaos. A single receiving neuron has thousands of receptor sites and receives many different messages and passwords at once. Each neuron adds up the incoming signals and determines whether or not to pass the information along to other cells. Enzymes help out by patrolling the brain and eliminating excess message-halting GABA to ensure a balance in communication.
Normally each neuron affects only a limited number of other cells. If a sufficient amount of GABA is lacking, however, the system goes out of whack, and tens of thousands of neurons send messages rapidly, intensely and simultaneously, resulting in a seizure. This is actually the primary cause of epilepsy... a lack of sufficient enzymes to keep GABA levels up.
Benzodiazepine is another neurotransmitter that binds to the exact same dendritic receptor as GABA. This receptor is known as the GABA receptor as GABA is the primary neurotransmitter to bind there. GHB also binds to the GABA receptor btw. Anywho, the study showed that stanozol can actually increase the GABA receptor's affinity toward benzodiazepine neuroreceptor. Benzodiazepine as I recall has a "chilling" affect on the CNS meaning that stanozol may actually reverse halo's tendency toward roid rage but I don't see anything that would cause a stimulation of muscle nerves in the PNS... It would probably only acheive this through AR-mediation (which is its primary anabolic means).
It is my understanding that this would only have psychological effects... effects that are not seen with other androgenic/anabolic steroids.
-Stew
