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nov cycles thread. Post them up boys.

Still doing some PT for my elbow.
8 weeks out from upperbody, started lifting lightly (upperbody) this week and
so far so good!!!

ive been on cycle for about 14 weeks
im on
tezt(650mg-ew)
eqz(400mg-ew)
deka(400-600mg ew)

im about to finish up and go natty for a while!!!!!
GUES WHOS DESIGNING MY PCT!!! YOU!!!!

then do a SUPER cycle after new years!!!
when im doin pct
im gonna do a stricter carb cycling diet w/ clen 2wks on 2 wks off
then ill be fucking shredded going into my jan cycle
i plan on being 8% by my jan cycle
 
holy ghost said:
Still doing some PT for my elbow.
8 weeks out from upperbody, started lifting lightly (upperbody) this week and
so far so good!!!

ive been on cycle for about 14 weeks
im on
tezt(650mg-ew)
eqz(400mg-ew)
deka(400-600mg ew)

im about to finish up and go natty for a while!!!!!
then do a SUPER cycle after new years!!!
I think I am going to start stalking up for my next cycle too. Takes me a few months to get all I need for my cycles. :evil:
 
Almost half way done w/ my

Tbol 40 mg ED1-4
Test E 250mg EW 1-10

I was doing like 10-20 lbs on everything when i started (coming off shattering my clavicle)

Now my bench is

4x4 295

Close grip bench

185 4X8

pulldowns

270 to failure 4 times

straightleg deadlift 315 4X6

Deadlift 335 4X6

I'm scared to do squats b/c of all the weight on my shoulder :(

leg press 11 plates on each side, 4X8

I've lost a good amount of bf too, but I'm so fucking fat that I'm going to wait 5 more wks to post pics lol. I decided the first 5 weeks I'd focus purely on strength size, then use these 5 wks to cut more. I'm going to really cut down on carbs and start doing way more intense cardio and a lot more frequently. I figured if I cut during pct there's a good 9 wks of weightloss in total :p
 
my cycle is in my sig., i going to start in about 10 days (witing for my gear LOL)

also, i am part of "The Omega Project" so i am pumped. this should be a good run.
 
Cal- thats great but you didnt tell us how much weight your body went up!!!

I mean, how does your body look...more toned, less chunky, tighter, ?????

First cycles are the BEST cycles.

Im honored to be in your signature!!!

Acela - I cant wait bro im pumped too, pick up some fucking flax or fish oil though.
It will save your ass. Also for all you F'ers that dont know, Im doing acelas diet for the contest.

Lets all make needto the OFFICAL PCT - post cycle therapy - guy for this thread.
 
Natty and haten it ,start bulker in 4 weeks trying a dif bulker this time
a bread & butter with tren e .
Brad.
 
holy ghost said:
Cal- thats great but you didnt tell us how much weight your body went up!!!

I mean, how does your body look...more toned, less chunky, tighter, ?????

First cycles are the BEST cycles.

Im honored to be in your signature!!!

Acela - I cant wait bro im pumped too, pick up some fucking flax or fish oil though.
It will save your ass. Also for all you F'ers that dont know, Im doing acelas diet for the contest.

Lets all make needto the OFFICAL PCT - post cycle therapy - - post cycle therapy - guy for this thread.

no doubt with the diet Bro. carb cycling for the first time fellas.

i will grab both flax and fish oil
 
Just grab Flax, its a lot easier digestable

I get fish burbs from fish oil

Flax is PACKED with omega EFA's

I got a site to get 50lb bales of whey-protein too!!
 
holy ghost said:
Just grab Flax, its a lot easier digestable

I get fish burbs from fish oil

Flax is PACKED with omega EFA's

I got a site to get 50lb bales of whey-protein too!!

Do you mind letten me know if its US so i can take a looky .
Brad
 
Im on week 5 of 250 test c and 300 equipose....Seeing great results already and my strengh is awesome!! Today i did 295 on bench for 6 reps. Before the cycle it was more like 275 for 3 reps. Gained about 12 pounds and some water weight but not bad. And if you read my post yesterday, well i guess it is in the muscle as i have a sore ass now. lol
 
holy ghost said:
Cal- thats great but you didnt tell us how much weight your body went up!!!

I mean, how does your body look...more toned, less chunky, tighter, ?????

First cycles are the BEST cycles.

Im honored to be in your signature!!!

Acela - I cant wait bro im pumped too, pick up some fucking flax or fish oil though.
It will save your ass. Also for all you F'ers that dont know, Im doing acelas diet for the contest.

Lets all make needto the OFFICAL PCT - post cycle therapy - - post cycle therapy - guy for this thread.

That's cause I'm ashamed of how disgusting i let my body get during my 6 months out due to injury :( lol.

Plus it's hard for me to tell how much change has really occured til I lose more fat. I mean the scale just jumps up and down because I'm getting bigger, but also losing weight. I just stopped weighing myself altogether lol. I wouldn't consider myself anywhere close to lean yet. But there's definitely a noticeable difference. haha. One of the cool priest/Christian brothers @ my college was like "hey brotha your chest is getting huge, I wanna get mine big like that" haha he was born in east l.a. and is hella cool. They named a building after the guy so many people like him lol. Also one of my old roommates asked me if i was juicing because my body's is getting closer to what it looked like when i was in shape. Maybe I'm being too hard on myself...I'm going to keep being hard on myself though, it's good motivation lol.
 
needtogetaas said:
Lets see your cycles this month guys. How they look, How they are going for you, and all that good sheeeet. :)

I'm doing this... starting Jan. 1st.

1-12 Test Enth @ 500mg EW
1-15 Liquidex @ 0.25mg ED
3-12 500iu HGC EW
8-12 Anavar @ 50mg ED
13-15 50 mgs Clomid ED
16-17 Dermacrine Sustain 5-6 pumps ED

I'm feeling like a bag of smashed a-holes these days. I'm down 10lbs. (to 195lbs.) and I feel weak like a little girly-man.

I'm going to try and step up the eating again soon. Lock and load.
 
cal- you need to find that motivation in yourself. Sounds like your diet BLOWS.

badguy- thats a lot of DEX, you ought to run the var minimum 6 weeks imo
 
Im 6 weeks into this.

1-5 Dianabol 40mg ed.
1-7 Tren A 50mg ed.
1-20 Test 750mg ew.
1-18 EQ 600mg ew.
7-15 Anavar 50mg ed.

Yeah I ran tren and Dianabol at the same time and loved it. Not sure what my liver thought of it though.
My bodyfat is now at 9.3% on digital calipers. I would venture to say I was 12-14 percent at the beginning of the run. Ghost you saw the before pic. Going to take and post up some pics at 10 weeks in but looking vascular and hard strength is through the roof.
 
i thought you dropped the tren you fag

bad ass cycle though!!!!! '

go get some winny and run it from 16-20 to cut up!
 
Test Enth 250mg mon,fri weeks 1-10
D-bol 40mg ed weeks 1-6
Liver assist
liquid L-dex 1mg per day
I'm on week 4 and the test has just started to really kick in. Muscles are getting bigger and fuller. Incredible muscle pumps. I have already put on 15 lbs since I started. This week the test seems to be having a great effect on lowering my bodyfat. My stomach is starting to appear much more muscular, and bodyfat everwhere is decreasing even though i'm eating more. It could be the L-dex, I just started it 4 days ago to fight gyno and aromatization. This is my first cycle and it's going good. I'm getting some tren that i'm going to run for 4 weeks mid cycle. Can L-dex be used as pct? I know it increases testosterone and takes estrogen down to almost 0. Would it help get the old boys back to producing T, or do I have to buy clomid? I could research this, I know, I was just wondering if anyone has run L-dex for pct successfully..
 
Cycle i am currently running, and i am in two weeks. . .


Test cypionate 500mg weekly
Equipose 400mg weekly
Anadrol 50mg ED
Still planning my PCT

and plan on cutting down near end of cycle
 
702daswoll1 said:
Im 6 weeks into this.

1-5 Dianabol 40mg ed.
1-7 trenbolone A 50mg ed.
1-20 Test 750mg ew.
1-18 Equipoise - boldenone undecylenate - 600mg ew.
7-15 Anavar 50mg ed.

Yeah I ran trenbolone and Dianabol at the same time and loved it. Not sure what my liver thought of it though.
My bodyfat is now at 9.3% on digital calipers. I would venture to say I was 12-14 percent at the beginning of the run. Ghost you saw the before pic. Going to take and post up some pics at 10 weeks in but looking vascular and hard strength is through the roof.
This cycle is a needto special. :)
 
Starting my first cycle later this month.

250mg Test Enan Mon. / Thurs EW for 12w
multi vit, chrysin and Indole-3-Carbinol ED
PCT 50mg Clomid ED 1st week, 20mg Nolva ED 4 weeks
 
Yes, Needto originally outlined this run for me and it has been a great ride so far. My stomach really has leaned out the last couple of weeks especially. Im using an old school eca stack with real ephedrine HCL in it so that is helping out alot.
 
702daswoll1 said:
Im 6 weeks into this.

1-5 Dianabol 40mg ed.
1-7 trenbolone A 50mg ed.
1-20 Test 750mg ew.
1-18 Equipoise - boldenone undecylenate - 600mg ew.
7-15 Anavar 50mg ed.

Yeah I ran trenbolone and Dianabol at the same time and loved it. Not sure what my liver thought of it though.
My bodyfat is now at 9.3% on digital calipers. I would venture to say I was 12-14 percent at the beginning of the run. Ghost you saw the before pic. Going to take and post up some pics at 10 weeks in but looking vascular and hard strength is through the roof.
Dam Mate I like the sound of that Dbol & Tren to crank up a cycle. Never thought of that. Did ya get any bloat with the tren in there? Think I might blast up my cycle & put a bit dbol in. Cause got the rest sorted except for var. Sounds delicious! Nice cycle Bro
 
holy ghost said:
Im going to run Dianabol - methandrostenolone - with superdrol im excited about this superdrol shit.

Anavar - oxandrolone - is the best bro all around
Ha Ha Holy you love the var. Thats what most of my female BB friends use!
 
Well.. I startet on a test,deca,dbol 3 weeks ago..
400 mg test e ew 1-12 weeks
200 mg deca ew 1-10 weeks
15 mg dbol ed 1-4 weeks
+ pct..
And hell, my back is getting pretty big :P
 
702daswoll1 said:
Yes, Needto originally outlined this run for me and it has been a great ride so far. My stomach really has leaned out the last couple of weeks especially. Im using an old school eca - ephedrine - caffeine - aspirin stack with real ephedrine HCL in it so that is helping out alot.

702, what's your diet like bro?
you doing the HolyGhost carb cycle like myself?

that's some impressive BF % to drop
 
cal_21 said:
Can't wait to try it when cycle # 2 comes along. I'm so tempted to throw it in @ the end of this one :evil: ...but I won't LOL.


good to hear bro save it for your 2nd cycle, you will love it
 
holy ghost said:
cal- you need to find that motivation in yourself. Sounds like your diet BLOWS.

badguy- thats a lot of DEX, you ought to run the Anavar - oxandrolone - minimum 6 weeks imo

I'm paraniod when it comes to gyno. Especially since I got a small lump on my last cycle.

I'll consider running the var for 6 weeks.
 
holy ghost said:
Yeah Acela - i got that nigga on a diet

badguy- just have the dex, Femera - letrozole - , and shit on hand

I have EVERYTHING right here. I'm trying HARD to wait until Jan.
 
rudy76 said:
Starting my first cycle later this month.

250mg Test Enan Mon. / Thurs EW for 12w
multi vit, chrysin and Indole-3-Carbinol ED
PCT - post cycle therapy - 50mg Clomid ED 1st week, 20mg Nolvaldex - tamoxifen citrate - ED 4 weeks
I don't like your pct bro. The clomid part is fine you should have just that dose 4 weeks. Any way you should add some dermacrin sustain to that nolva.
 
i want state, unequivocally, that i do not plan to do, nor do i have anywhere in my house, car, office etc, the means to do

weeks 1-16 500/equipoise
weeks 1-16 500/test e.
weeks 1-4 30/d-bo.l (cannot go over--30, bad sides for me)

instead i will spend much money at gnc to get big the joe weider way
 
All you guys doing long cycles 12 weeks and up. You need to run your hcg during cycle on long cycles like this imo. I think you should run it during cycle all the time, but even more so on longer cycles. A good dose would be 500ui's ew.
 
needtogetaas said:
All you guys doing long cycles 12 weeks and up. You need to run your HCG - human chorionic gonadotropin - - human chorionic gonadotropin - during cycle on long cycles like this imo. I think you should run it during cycle all the time, but even more so on longer cycles. A good dose would be 500ui's ew.

the only thing i am concerned w/HC.G each week is desensitizing the leydig cells so they don't work w/o use of HC.G (meaning perm)- --i cannot find a clear answer either way--
 
Sustanon 400mg a week wk 1-9

Winstrol - stanozolol 100mg ed wk 3-9
liquidex .25 eod
AIFM throughout

PCT - post cycle therapy - HCG - human chorionic gonadotropin - 1000 ius ed for 5 days
post cycle and unleashed after

2nd cycle

6-4 255 @ 12%bf

goal 260 @ 10%bf
 
eddymerckx said:
the only thing i am concerned w/HC.G each week is desensitizing the leydig cells so they don't work w/o use of HC.G (meaning perm)- --i cannot find a clear answer either way--
I have read many studies that show prolonged high doses,or even short high doses of hcg can cause desensitizing of the Leydig cells. Even so these studies
show that with time sensitivity dose come back more often then not. I have also read some studies on long time/short time low dose hcg use. Witch is what we are talking about here. Most of the ones I have seen, show that this dos not cause any desensitizing of the Leydig cells. Here I just pulled this one out of the hat but I am sure there is a lot more. I don't feel like looking any more though lol.
http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=Retrieve&db=PubMed&list_uids=3260774&doptcmdl=Abstract
 
needtogetaas said:
I have read many studies that show prolonged high doses,or even short high doses of HCG - human chorionic gonadotropin - - human chorionic gonadotropin - can cause desensitizing of the Leydig cells. Even so these studies
show that with time sensitivity dose come back more often then not. I have also read some studies on long time/short time low dose HCG - human chorionic gonadotropin - use. Witch is what we are talking about here. Most of the ones I have seen, show that this dos not cause any desensitizing of the Leydig cells. Here I just pulled this one out of the hat but I am sure there is a lot more. I don't feel like looking any more though lol.
http://www.ncbi.nlm.nih.gov/sites/entrez?cmd=Retrieve&db=PubMed&list_uids=3260774&doptcmdl=Abstract

thanks neddeto--that is what i was looking for--got to protect all those future little eddymerckxes
 
eddymerckx said:
i want state, unequivocally, that i do not plan to do, nor do i have anywhere in my house, car, office etc, the means to do

weeks 1-16 500/equipoise
weeks 1-16 500/testosterone enanthate.
weeks 1-4 30/d-bo.l (cannot go over--30, bad sides for me)

instead i will spend much money at gnc to get big the joe weider way
Ha Ha Buy a Bowflex then!
 
eddymerckx said:
the only thing i am concerned w/HC.G each week is desensitizing the leydig cells so they don't work w/o use of HC.G (meaning perm)- --i cannot find a clear answer either way--

This is an interesting article covering PCT & Ledig cells etc. Originally written I think by Eric Potratz. I have posted it on other sites. If you think it OK copy to a new thread for others to read.

Everything That's Wrong With Your post cycle therapy (interesting article)

--------------------------------------------------------------------------

Discussion of pharmaceutical agents below is presented for information only. Nothing here is meant to take the place of advice from a licensed health care practitioner. Consult a physician before taking any medication.


In the world of steroid users, it has become mandatory to follow post cycle therapy (post cycle therapy) upon cessation of steroid use. Many great post cycle therapy protocols have been outlined over the years, and many individuals have had great success with following such protocols. Nevertheless, what works can always work better. This is especially the case for those that have had a lack of success following popular advice. In this article I will address the major problems with popular post cycle therapy protocols and clarify exactly how we should use the items at our disposal for optimum recovery from anabolic steroids. Three main topics will be covered in this article –

• hCG on cycle -- I will show you the best way to use HCG, which will protect your "testicular real-estate", and prime your HPTA for the fastest and most complete recovery possible.

• SERMs. -- Drugs such as Clomid and Nolvadex are some of the most toxic drugs in a steroid-users cabinet. I will present the evidence of this toxicity and provide alternatives.

• Peptides for post cycle therapy -- Peptides such as Growth Hormone and IGF-1 have much more of a role in PCT than most people realize. Besides preserving muscle gains, these hormones can actually help restore testicular function after a cycle.

HCG unraveled

Human Chorionic Gonadotropin (hCG) is a peptide hormone that is used in place of LH to stimulate hormone production from the gonads. LH is the primary signal sent from the pituitary to the testes, which stimulates the leydig cells within the testes to produce testosterone. When steroids are administered, LH levels rapidly decline. The absence of an LH signal from the pituitary causes the rapid onset of testicular degeneration. The testicular degeneration begins with a reduction of leydig cell volume, and is then followed by rapid reductions in intra-testicular testosterone (ITT), peroxisomes, and Insulin-like factor (INSL3) – All important bio-markers and factors for proper testicular function and testosterone production.2-6,19 However, this degeneration can be prevented by a small maintenance dose of hCG ran throughout the cycle. Unfortunately, most steroid users have been engrained to believe that hCG should be used after a cycle. Though, we will learn that a faster and more complete recovery is possible if hCG is ran during a cycle.
Firstly, we must understand the clinical history of hCG to understand the most efficient way to use it. Many popular "steroid profiles" advocate an hCG dose of 2500-5000iu once or twice a week. These were the kind of dosages used in the historical hCG studies for hypogonadal men who had reduced testicular sensitivity due to prolonged LH deficiency.85,86 That is, testes desensitize when not presented with a sufficient LH signal. In men with normal LH levels and testicular sensitivity, the maximum increase of testosterone is seen from a dose of only ~250iu, with minimal increases obtained from 500iu or even 5000iu. (It appears the testes maximum secretion of testosterone is about 140% above base line.12-18) So, if you have allowed your testes to desensitize over the length of a typical steroid cycle, (8-16 weeks) then you would require a higher dose to elicit a response in an attempt to restore normal testicular size and function – but there is cost to this, and a high probability that you won’t regain full testicular function.

To get an idea of how quickly testicular degeneration occurs from your average multi-anabolic steroids cycle, consider this: LH levels are rapidly decreased by the 2nd day of steroid administration.2,9,10 By shutting down the LH signal and allowing the testis to be non-functional over a 12-16 week period, leydig cell volume decreases 90%, ITT decreases 94%, INSL3 decreases 95%, while the capacity to secrete testosterone decreases as much as 98%.2-6 It should be mentioned that visually analyzing testes size is a poor method of judging your actual testicular function, since testicular size is not directly related to the ability to secrete testosterone.4 This is because the leydig cells, which are the primary sites of testosterone secretion, only make up about 10% of the total testicular volume. Therefore, testicular size may appear normal on a cycle, but the testes ability to secrete testosterone upon LH or hCG stimulation can actually be significantly diminished.3-5

The decreased testosterone secretion capacity was well demonstrated in a study on power athletes who used steroids for 16 weeks, and were then administered 4500iu hCG post cycle. It was found that the steroid users were about 20 times less responsive to hCG, when compared to normal men who did not use steroids.8 In other words, their testosterone secretion capacity was dramatically reduced because they did not receive an LH signal for 16 weeks. The testes essentially became desensitized and crippled. Case studies with steroid using patients show that aggressive long-term treatment with hCG at dosages as high as 10,000iu E3D for 12 weeks were unable to return full testicular size. Other studies with men using low dose steroid implants for 6 weeks showed unsuccessful return of Insulin-like factor-3 (INSL3) concentration in the testes upon 5000iu/wk of HCG treatment for 12 weeks.

These studies show that postponing hCG usage until the end of a cycle, increases your need for a higher dose of hCG, and decreases your odds of a full recovery. As a consequence to using a higher dose of hCG, estrogen will be increased disproportionately, which then causes further HPTA suppression while increasing the risk of gyno.11 For example, high doses of hCG are known to raise estradiol 165%, while only raising testosterone 140%.11 Higher doses of hCG are also known to reduce LH receptor concentration and degrade the enzymes responsible for testosterone synthesis within the testes12,13,19 (the last thing someone wants during recovery). While these negative effects of hCG can be partly mitigated by the use of a drug such as tamoxifen, it will create further problems associated with using a toxic SERM. (covered in the next section)

In light of the above evidence, it becomes obvious that we must take preventative measures to avoid this testicular degeneration. Besides, with hCG being so readily available, and such a painless shot, it makes you wonder why anyone wouldn’t use it on cycle. Based on studies with normal men using steroids, ~100iu HCG administered everyday was enough to preserve full testicular function and ITT levels, without causing desensitization typically associated with higher doses of hCG.2 It is important that low-dose hCG is started before testicular degeneration occurs, which appears to rapidly manifest within the first 2-3 weeks of steroid use.

Recap – For optimal preservation of testicular function during cycle, use 100iu hCG ED starting 3 days after your first anabolic steroids dose. Drop the hCG a week before the anabolic steroids clear the system. For example, you would drop hCG a week after your last Testosterone Enanthate shot. Or, if you are ending the cycle with orals, you would drop the hCG a week before your last oral dose. This will allow for a sudden and even drop in hormone levels, while initiating LH and FSH production from the pituitary, making for a seamless recovery.
A more convenient alternative to the above recommendation would be a weekly shot of 500iu hCG, throughout the entire cycle. Beyond this dose, one could calculate a rough estimate for their required hCG dosage by multiplying 40iu x days of LH absence. (40iu x 60 days = 2400iu HCG dose)
As an alternative to the on cycle hCG protocol, you could follow a plan based on modulation of the gonadotropin pulse generator. (seen here)
Note: If following any of these protocols, hCG should NOT be used after the cycle.

Clomid & Nolva; A closer look

The use of Clomid and Nolvadex, as Selective Estrogen Receptor Modulators (SERMs), has gradually become well established in the steroid using community. The popular push of these drugs has almost made them mandatory. They have essentially become hormonal vitamins – vitamins that can do no wrong and provide seemingly endless benefits of testosterone support, bloat reduction, gynecomastia prevention and cholesterol health. It seems that we are all well educated about the benefits of Clomid and Nolvadex, so in this segment, I will present the risks and consequences from the short and long term use of Clomid and Nolvadex.
Upon examination of the research available for Clomid (clomiphene) and Nolvadex (tamoxifen) we find that the research is quite extensive, and contradicting.21 We see many early studies with tamoxifen done on breast cancer patients, which show an acceptable "safety profile", with an apparent lack of adverse effects.22 On the other hand, many of the early in vivo animal studies showed severely toxic effects, with the development of cancer in the liver, uterus, or testes upon tamoxifen administration.30-34,41 However, this evidence was largely disregarded by ex vivo (test tube) research on human cell-lines which appeared to show a lack of toxic effects.

For example, tamoxifen was generally accepted as being non-toxic to human liver upon the conclusion that tamoxifen did not cause noticeable DNA adducts (damage) during short-term ex vivo studies with human liver cells. This was in contrast to the in vivo animal studies showing dramatic carcinogenic effects on the liver.30-34,41 As scientists learned that the toxic effects from tamoxifen are from the metabolism and buildup of the a-hydroxytamoxifen, 4-hydroxytamoxifen and N-desmethyltamoxifen metabolites. It became apparent that ex vivo research was largely flawed due to low-rate metabolism.21 The carcinogenic effects of tamoxifen proved to be even more unusual and elusive, when it was hypothesized that tamoxifen had both genomic and non-genomic toxicity, which affecting different animals, in different organs.21 This created an obvious clinical challenge for measuring genotoxicity in a test tube. Eventually, it was established that tamoxifen was a bona-fide carcinogen in all species, at least in one way or another. Recent human studies have shown tamoxifen treated women to have 3x the risk of developing fatty liver disease, which appeared as soon as 3 months into therapy at only 20mg/day.24-26 In some cases, the disease lasted up to 3 years, despite cessation from tamoxifen therapy. Five and ten year follow-ups with patients on long term tamoxifen therapy showed cases of deadly hepatocellular carcinoma.27-29 In a 2000 case study involving tamoxifen induced liver disease, D.F Moffat et al made a profound statement –

"In addition, hepatocellular carcinoma in tamoxifen treated patients may be under-reported since there may be reluctance to biopsy liver tumours which are assumed to be secondary carcinoma of the breast."

In other words, it appears that the liver carcinoma from a large number of breast cancer patients on tamoxifen therapy has been misdiagnosed as a metastasis infection from the breast cancer itself. Upon closer examination it was found that the cancerous lesions in the livers of the long-term tamoxifen therapy case studies were identical to those seen in the early animal studies showing tamoxifen to be a potent hepatotoxin.28-34 Although the effects took much longer to manifest, it became obvious that tamoxifen was toxic to the human liver.

Another well known risk of tamoxifen therapy is the increased risk of developing endometrial cancer (uterine cancer). This is due to tamoxifen actually acting as an estrogen agonist in the uterus, presumable from the 4-hydroxytamoxifen metabolite. This estrogenic metabolite triggers abnormal growth of the uterus and the formation of cancer causing DNA adducts.33 As male bodybuilders we assume this presents no risk. On the contrary, the implications are quite scary when we realize the male equivalent to the uterus is the prostate -- differentiating from the same embryonic cell line and sharing the same oncogene, Bcl-2, and high concentration of the estrogen receptor. It is likely that tamoxifen has the same estrogenic action, and DNA damaging effects within the prostate. It is no wonder that tamoxifen failed as a treatment for prostate carcinoma.

Aside from restoring testosterone levels post cycle, tamoxifen is often used to combat gyno during cycle when "flare ups" occur. While tamoxifen may provide immediate inhibition of growth, and serve as valuable tool, it also has the ability to up-regulate the progesterone receptor.54-56 This is a true contradiction, which dramatically increases your chances of bringing upon gyno in future cycles when utilizing Nandrolone (Deca) or Trenbolone, both of which act upon the progesterone receptor. It is interesting to speculate: is tamoxifen use directly related to the increased gyno occurrences seen with modern day steroid users?

When we bring our attention to Clomid, we find less research is available on long term human toxicity, probably because of the relatively short term (3-4 week) clinical application for ovarian stimulation,59 although long term follow ups with patients who received Clomid for ovulation induction have shown an increased risk of developing uterine cancer.74 This is to be expected, since many of the same carcinogenic tendencies found with tamoxifen are the same effects seen with clomiphene.44,45,57,58 Upon analysis of anecdotal reports from Clomid and nolva users, we see the typical short term side effects of low libido, erectile dysfunction, and emotional instability – despite many men showing normalized testosterone and estrogen levels during the use of these SERM’s. Research on male breast cancer patients also shows frequent reports of low libido, thrombosis (arterial blockage), and hot flashes with tamoxifen use.47 Another common side effect associated with both SERMs, but more common with Clomid, is the loss of visual accuracy and development of visual "tracers", due to the ocular toxicity.

As the medical community became more aware of the side-effects associated with clomiphene and tamoxifen treatment, newer and safer SERMs, such as toremifene and raloxifene hit the developmental fast track. Toremifene appears to be less liver toxic, but it is an analog of tamoxifen, so it also carries many of the related genotoxic effects. Raloxifene appears to be even safer by being the least liver toxic, and not having any potential issue with the uterus or prostate. Unfortunately, raloxifene has been associated with a higher incidence of thromboembolism52 (arterial blockage), and also has very low oral absorption, making it an expensive alternative at a typical 120mg/day dose. Still, raloxifene could presumably be equally effective as Clomid or Nolvadex at restoring HPTA function, while imparting less side effects. Newer SERMs are already being evaluated such as bazedoxifene, arzoxifene, and lasofoxifene, in hopes of reducing risk even further.

Another SERM that may be useful for post cycle therapy is resveratrol.87,88 Resveratrol is a natural polyphenol extracted from grape skin, that has recently been under heavy research for its cancer fighting effects in the breast, prostate and liver. Contrary to Nolva or Clomid, resveratrol appears to actually have beneficial effects on the liver,70 as well as having multiple benefits on cardiovascular health by limiting LDL oxidation and improving endothelial function.71-73 Improved blood vessel function may be a mechanism by which resveratrol improves erectile function in many men. Research also suggests that resveratrol may actually extend life, by reducing oxidative stress on organs such as the heart,77 and preventing the metabolic syndrome by fighting insulin resistence. It’s becoming well known that insulin resistance is a leading cause of low testosterone.82 More specifically, improving insulin sensitivity will increase your leydig cell sensitivity, and therefore increase the testes response to LH.

It should be pointed out that resveratrol may not be the best choice to combating emergency gyno, due to its lower binding affinity to the human ER of about 90x less than tamoxifen, and about 30x less than clomiphene.75,76 However, considering that resveratrol is a pure estrogen antagonist at the pituitary,89 while Clomid has mixed agonist/antagonistic effects,90-94 resveratrol could be a suitable substitute for post cycle therapy. Aside from acting as a SERM, resveratrol can also help control estrogen by actually limiting aromatase enzyme production.82 Based on the research, it appears that at least 100mg/day would needed to increase LH, FSH and testosterone production.84 This is comparable dose of resveratrol found in an advanced topical based product, Dermacrine Sustain (found here - http://www.primordialperformance.com).

Admittedly, no steroid users are dropping dead from a 4 week protocol of Nolva or Clomid, and many will say "the consequences far outweigh the benefits" -- but why deal with the potential consequences when alternatives are available?

Peptides for testicular recovery

It’s a common practice these days for experienced bodybuilders to implement some dosage of IGF-1 either during or after a cycle to "pick up" a lagging body part, or to preserve gains in muscle. Growth Hormone (GH) is also a versatile drug for cutting or bulking, with increasing popularity as it becomes more affordable. The value of IGF-1 and GH becomes so much more significant when we realize there integral role in testicular function. In fact, it seems that these hormones are more effective at building testes, than muscles.

Research has shown GH to be vitally important in testicular function, but it is generally accepted that the beneficial effects are directly mediated by hGH’s conversion to IGF-1.98 As many of you know, IGF-1 is created in the liver by GH, upon interacting with insulin. So, we will be focusing on the usage and benefits of IGF-1, rather than GH, as it seems more cost effective and directly related to our purpose of optimizing recovery.

In short, IGF-1 increases steroidogenic acute regulatory protein (sTAR), and cholesterol side chain cleaving enzyme (CYP 11A)99. These are both rate-limiting steps and are critical factors for converting cholesterol into hormones, such as testosterone. IGF-1 also has the ability to increase the concentration of steroidogenic enzymes in the testes, such as 3b HSD.100 IGF-1 can also increase the testes sensitivity to LH and hCG by increasing the number of LH receptors.

These positive effects on testicular function make IGF-1 an ideal drug for post cycle therapy. A dose of IGF-1 Lr3 at 80mcg/day, split two times per day, would likely be the most cost effective dose.

In conclusion, we have learned that utilizing hCG during a steroid cycle will significantly prevent testicular degeneration. This helps create a seamless transition from "on cycle" to "off cycle". Then, by avoiding the deleterious SERMs such Clomid and Nolvadex and opting for safer alternatives, you can seemingly avoid any sort of post cycle crash, while maintaining a strong libido and uncompromised emotional health.
 
That is a great post nzrodney. I would like to see you add more like this to the board. Whats up with all the bombs bro? Send me a pm. I know there is always two sides to every story. Again great post.
 
Just started some dbawl on thursday at 30mg a day. Gonna run that for 2 more weeks. Tomorrow i start 600mg su.st and 400mg eqz then when i drop the dbawl up the eqz to 600mg. Not sure if im gonna run the sust for 14 weeks or drop that around 10-12 and keep running the eq by itself with prov.iron longer.
 
b_light said:
Just started some dbawl on thursday at 30mg a day. Gonna run that for 2 more weeks. Tomorrow i start 600mg su.st and 400mg eqz then when i drop the dbawl up the eqz to 600mg. Not sure if im gonna run the Sustanon for 14 weeks or drop that around 10-12 and keep running the Equipoise - boldenone undecylenate - by itself with Proviron - mesterolone - .iron longer.
You got any more comps coming up bro?
 
Might do a strongman comp in dec. not sure though as my calf still has a nagging pain and keeps me from going 100%. If i don't do that one next show will probably be this time next year BBing comp probably do some strongman shit over the summer as well again.
 
Turanabol 60mg/wk for 5 weeks
testosterone enanthate 500mg/wk for 14 weeks
Equipoise - boldenone undecylenate - 300mg/wk for 14 weeks
Proviron 50mg/wk for last 6 weeks

PCT - post cycle therapy - - post cycle therapy - :
HCG - human chorionic gonadotropin - - human chorionic gonadotropin - 500iu/wk for 6 weeks
nolvadex 40/40/20/20mg 4 weeks.
 
holy ghost said:
why Cal?

also n2 brings up a good pt with the HCG - human chorionic gonadotropin - during long protocls.

Just scared about losing the hair...plus it'll shed for another 2 wks after I stop too right? Damn. Should've just done EQ & Deca lol. Fuck deca dick is only temporary right? Can't lose my hair @ 21 lol
 
shit bro how much hair are you losing?!?!?!?

Should have stuck with deca! you aint getin any to worry about deca dick negro!!!

Looks youll be running VAR/EQ/TBOL/primo cycles!!! Bummers!
 
b_light said:
Might do a strongman comp in dec. not sure though as my calf still has a nagging pain and keeps me from going 100%. If i don't do that one next show will probably be this time next year BBing comp probably do some strongman shit over the summer as well again.
That leg is still hurting bro. :worried:
 
I am 10 weeks in to this cycle

week 1-4 dbol 30mg/day
week 1-16 Sust 500/week
week 1-16 Eq 600/week
week 10-16 winstrol 100mg/day
HCG at 250iu's e3d...
adex .5/day
thinking about some proviren for the last six weeks

I have gained about 15lbs..I started at 148 and now am 162-163...I getting more and more vascular...Strenth went throught the roof since day one and my body fat is down from 10-11% to 8% when I checked at week 6...I am hoping this winstrol and the cardio 4x a week in the am on a empty stomach really does the trick to get the bf % lower and these abs looking sick
 
I have ran that exact cycle. Jesus bro 148??

4x a week cardio may be a bit excessive judging by your body type, How tall are you more importantly.. You are an ecto right???
 
Nah fuck that, the Juice will heal up the leg im just gonna go easy right now with legs should be good in another week or two.
 
starting my cycle as soon as it all gets here... good look a little like this


sust..750mg 1-12
eq 600mg 1-16
tren Ent. 400mg 1-12
40mg dbol preworkout
40mcg igf1.. 4 weeks on 4 weeks off.. gonna do that for about 16 weeks

i have winny too but i havent decided if im gonna run it at the end, or just save it for next time
 
No dude the legs not that bad. It's been healed for a while it just hurts like if i hit it weird and even then not that bad, the docs said its gonna be like that for a long time maybe never back to 100%.
 
holy ghost said:
shit bro how much hair are you losing?!?!?!?

Should have stuck with Deca-Durabolin - nandrolone decanoate - ! you aint getin any to worry about Deca-Durabolin - nandrolone decanoate - dick negro!!!

Looks youll be running Anavar - oxandrolone - /Equipoise - boldenone undecylenate - /Turanabol/Primobolan - methenolone - cycles!!! Bummers!

Enough hair to worry me.... :worried: If i lose anymore it'll be super obvious...if it isn't now :(

Fuck

I'm going to stock up on deca/EQ til i can afford primo. lol. Tbol's more of my friend than I had anticipated from now on lol.
 
hehe, im not that stupid. I've been waiting for everything to be a green before i hop on. First shot of sust and eq tomorrow morning cant fucking wait. 500lb deadlifts gonna be mine this cycle and its the perfect timing to because late winter ill be off and just ready to go back on to cut for summer.
 
WHATS your cycle bro??? what supps you taking ED???

whats diet bro??

you look so fucking skinny for a PL it trips me out bro, you have bb asthetics.
 
I know im starting to get some chub tho, lol. 600mg sus.t 400mg E.Q a week and 30mg dbawl ED for 2 more weeks. Once i drop the DBawl uppind e.q to 600mgs then i think im gonna drop the sust at week 10 or 12 and run eq for 14-16wks and provir.on throughout i got 100 of them. Taking protien, multis, flax and glucosamine with other shit in it.
 
holy ghost said:
WHATS your cycle bro??? what supps you taking ED???

whats diet bro??

you look so fucking skinny for a PL it trips me out bro, you have bb asthetics.
He dos look skinny for pl But he is a beats bro. Just a fucking beast. I Know because I am a pl and I am a big big boy, But there is some small powerful people out there. I will be at the New england record breakers next year. I hope to see some people there.
 
Yeah i don't really do PL i just did strongman for fun because they had the shit at my gym and i had to play with it and it just took off from there and its alot easier, form goes out the window for the most part with strongman and you just need to focus on one thing not the long haul and you can eat shit all the time.
 
nzrodney said:
This is an interesting article covering PCT - post cycle therapy - & Ledig cells etc. Originally written I think by Eric Potratz. I have posted it on other sites. If you think it OK copy to a new thread for others to read.

Everything That's Wrong With Your post cycle therapy (interesting article)

--------------------------------------------------------------------------

Discussion of pharmaceutical agents below is presented for information only. Nothing here is meant to take the place of advice from a licensed health care practitioner. Consult a physician before taking any medication.


In the world of steroid users, it has become mandatory to follow post cycle therapy (post cycle therapy) upon cessation of steroid use. Many great post cycle therapy protocols have been outlined over the years, and many individuals have had great success with following such protocols. Nevertheless, what works can always work better. This is especially the case for those that have had a lack of success following popular advice. In this article I will address the major problems with popular post cycle therapy protocols and clarify exactly how we should use the items at our disposal for optimum recovery from anabolic steroids. Three main topics will be covered in this article –

• HCG - human chorionic gonadotropin - on cycle -- I will show you the best way to use HCG, which will protect your "testicular real-estate", and prime your hpta - hypothalamic-pituitary-testicular axis - for the fastest and most complete recovery possible.

• SERMs. -- Drugs such as Clomid and Nolvadex are some of the most toxic drugs in a steroid-users cabinet. I will present the evidence of this toxicity and provide alternatives.

• Peptides for post cycle therapy -- Peptides such as Growth Hormone and IGF-1 have much more of a role in PCT than most people realize. Besides preserving muscle gains, these hormones can actually help restore testicular function after a cycle.

HCG unraveled

Human Chorionic Gonadotropin (hCG) is a peptide hormone that is used in place of lh - leutenizing hormone - to stimulate hormone production from the gonads. LH is the primary signal sent from the pituitary to the testes, which stimulates the leydig cells within the testes to produce testosterone. When steroids are administered, LH levels rapidly decline. The absence of an LH signal from the pituitary causes the rapid onset of testicular degeneration. The testicular degeneration begins with a reduction of leydig cell volume, and is then followed by rapid reductions in intra-testicular testosterone (ITT), peroxisomes, and Insulin-like factor (INSL3) – All important bio-markers and factors for proper testicular function and testosterone production.2-6,19 However, this degeneration can be prevented by a small maintenance dose of hCG ran throughout the cycle. Unfortunately, most steroid users have been engrained to believe that hCG should be used after a cycle. Though, we will learn that a faster and more complete recovery is possible if hCG is ran during a cycle.
Firstly, we must understand the clinical history of hCG to understand the most efficient way to use it. Many popular "steroid profiles" advocate an hCG dose of 2500-5000iu once or twice a week. These were the kind of dosages used in the historical hCG studies for hypogonadal men who had reduced testicular sensitivity due to prolonged LH deficiency.85,86 That is, testes desensitize when not presented with a sufficient LH signal. In men with normal LH levels and testicular sensitivity, the maximum increase of testosterone is seen from a dose of only ~250iu, with minimal increases obtained from 500iu or even 5000iu. (It appears the testes maximum secretion of testosterone is about 140% above base line.12-18) So, if you have allowed your testes to desensitize over the length of a typical steroid cycle, (8-16 weeks) then you would require a higher dose to elicit a response in an attempt to restore normal testicular size and function – but there is cost to this, and a high probability that you won’t regain full testicular function.

To get an idea of how quickly testicular degeneration occurs from your average multi-anabolic steroids cycle, consider this: LH levels are rapidly decreased by the 2nd day of steroid administration.2,9,10 By shutting down the LH signal and allowing the testis to be non-functional over a 12-16 week period, leydig cell volume decreases 90%, ITT decreases 94%, INSL3 decreases 95%, while the capacity to secrete testosterone decreases as much as 98%.2-6 It should be mentioned that visually analyzing testes size is a poor method of judging your actual testicular function, since testicular size is not directly related to the ability to secrete testosterone.4 This is because the leydig cells, which are the primary sites of testosterone secretion, only make up about 10% of the total testicular volume. Therefore, testicular size may appear normal on a cycle, but the testes ability to secrete testosterone upon LH or hCG stimulation can actually be significantly diminished.3-5

The decreased testosterone secretion capacity was well demonstrated in a study on power athletes who used steroids for 16 weeks, and were then administered 4500iu hCG post cycle. It was found that the steroid users were about 20 times less responsive to hCG, when compared to normal men who did not use steroids.8 In other words, their testosterone secretion capacity was dramatically reduced because they did not receive an LH signal for 16 weeks. The testes essentially became desensitized and crippled. Case studies with steroid using patients show that aggressive long-term treatment with hCG at dosages as high as 10,000iu E3D for 12 weeks were unable to return full testicular size. Other studies with men using low dose steroid implants for 6 weeks showed unsuccessful return of Insulin-like factor-3 (INSL3) concentration in the testes upon 5000iu/wk of HCG treatment for 12 weeks.

These studies show that postponing hCG usage until the end of a cycle, increases your need for a higher dose of hCG, and decreases your odds of a full recovery. As a consequence to using a higher dose of hCG, estrogen will be increased disproportionately, which then causes further HPTA suppression while increasing the risk of gynecomastia.11 For example, high doses of hCG are known to raise estradiol 165%, while only raising testosterone 140%.11 Higher doses of hCG are also known to reduce LH receptor concentration and degrade the enzymes responsible for testosterone synthesis within the testes12,13,19 (the last thing someone wants during recovery). While these negative effects of hCG can be partly mitigated by the use of a drug such as tamoxifen, it will create further problems associated with using a toxic selective estrogen receptor modulator. (covered in the next section)

In light of the above evidence, it becomes obvious that we must take preventative measures to avoid this testicular degeneration. Besides, with hCG being so readily available, and such a painless shot, it makes you wonder why anyone wouldn’t use it on cycle. Based on studies with normal men using steroids, ~100iu HCG administered everyday was enough to preserve full testicular function and ITT levels, without causing desensitization typically associated with higher doses of hCG.2 It is important that low-dose hCG is started before testicular degeneration occurs, which appears to rapidly manifest within the first 2-3 weeks of steroid use.

Recap – For optimal preservation of testicular function during cycle, use 100iu hCG ED starting 3 days after your first anabolic steroids dose. Drop the hCG a week before the anabolic steroids clear the system. For example, you would drop hCG a week after your last Testosterone Enanthate shot. Or, if you are ending the cycle with orals, you would drop the hCG a week before your last oral dose. This will allow for a sudden and even drop in hormone levels, while initiating LH and FSH - follicle stimulating hormone - production from the pituitary, making for a seamless recovery.
A more convenient alternative to the above recommendation would be a weekly shot of 500iu hCG, throughout the entire cycle. Beyond this dose, one could calculate a rough estimate for their required hCG dosage by multiplying 40iu x days of LH absence. (40iu x 60 days = 2400iu HCG dose)
As an alternative to the on cycle hCG protocol, you could follow a plan based on modulation of the gonadotropin pulse generator. (seen here)
Note: If following any of these protocols, hCG should NOT be used after the cycle.

Clomid & Nolvaldex - tamoxifen citrate - ; A closer look

The use of Clomid and Nolvadex, as Selective Estrogen Receptor Modulators (SERMs), has gradually become well established in the steroid using community. The popular push of these drugs has almost made them mandatory. They have essentially become hormonal vitamins – vitamins that can do no wrong and provide seemingly endless benefits of testosterone support, bloat reduction, gynecomastia prevention and cholesterol health. It seems that we are all well educated about the benefits of Clomid and Nolvadex, so in this segment, I will present the risks and consequences from the short and long term use of Clomid and Nolvadex.
Upon examination of the research available for Clomid (clomiphene) and Nolvadex (tamoxifen) we find that the research is quite extensive, and contradicting.21 We see many early studies with tamoxifen done on breast cancer patients, which show an acceptable "safety profile", with an apparent lack of adverse effects.22 On the other hand, many of the early in vivo animal studies showed severely toxic effects, with the development of cancer in the liver, uterus, or testes upon tamoxifen administration.30-34,41 However, this evidence was largely disregarded by ex vivo (test tube) research on human cell-lines which appeared to show a lack of toxic effects.

For example, tamoxifen was generally accepted as being non-toxic to human liver upon the conclusion that tamoxifen did not cause noticeable DNA adducts (damage) during short-term ex vivo studies with human liver cells. This was in contrast to the in vivo animal studies showing dramatic carcinogenic effects on the liver.30-34,41 As scientists learned that the toxic effects from tamoxifen are from the metabolism and buildup of the a-hydroxytamoxifen, 4-hydroxytamoxifen and N-desmethyltamoxifen metabolites. It became apparent that ex vivo research was largely flawed due to low-rate metabolism.21 The carcinogenic effects of tamoxifen proved to be even more unusual and elusive, when it was hypothesized that tamoxifen had both genomic and non-genomic toxicity, which affecting different animals, in different organs.21 This created an obvious clinical challenge for measuring genotoxicity in a test tube. Eventually, it was established that tamoxifen was a bona-fide carcinogen in all species, at least in one way or another. Recent human studies have shown tamoxifen treated women to have 3x the risk of developing fatty liver disease, which appeared as soon as 3 months into therapy at only 20mg/day.24-26 In some cases, the disease lasted up to 3 years, despite cessation from tamoxifen therapy. Five and ten year follow-ups with patients on long term tamoxifen therapy showed cases of deadly hepatocellular carcinoma.27-29 In a 2000 case study involving tamoxifen induced liver disease, D.F Moffat et al made a profound statement –

"In addition, hepatocellular carcinoma in tamoxifen treated patients may be under-reported since there may be reluctance to biopsy liver tumours which are assumed to be secondary carcinoma of the breast."

In other words, it appears that the liver carcinoma from a large number of breast cancer patients on tamoxifen therapy has been misdiagnosed as a metastasis infection from the breast cancer itself. Upon closer examination it was found that the cancerous lesions in the livers of the long-term tamoxifen therapy case studies were identical to those seen in the early animal studies showing tamoxifen to be a potent hepatotoxin.28-34 Although the effects took much longer to manifest, it became obvious that tamoxifen was toxic to the human liver.

Another well known risk of tamoxifen therapy is the increased risk of developing endometrial cancer (uterine cancer). This is due to tamoxifen actually acting as an estrogen agonist in the uterus, presumable from the 4-hydroxytamoxifen metabolite. This estrogenic metabolite triggers abnormal growth of the uterus and the formation of cancer causing DNA adducts.33 As male bodybuilders we assume this presents no risk. On the contrary, the implications are quite scary when we realize the male equivalent to the uterus is the prostate -- differentiating from the same embryonic cell line and sharing the same oncogene, Bcl-2, and high concentration of the estrogen receptor. It is likely that tamoxifen has the same estrogenic action, and DNA damaging effects within the prostate. It is no wonder that tamoxifen failed as a treatment for prostate carcinoma.

Aside from restoring testosterone levels post cycle, tamoxifen is often used to combat gynecomastia during cycle when "flare ups" occur. While tamoxifen may provide immediate inhibition of growth, and serve as valuable tool, it also has the ability to up-regulate the progesterone receptor.54-56 This is a true contradiction, which dramatically increases your chances of bringing upon gynecomastia in future cycles when utilizing Nandrolone (Deca-Durabolin - nandrolone decanoate - ) or Trenbolone, both of which act upon the progesterone receptor. It is interesting to speculate: is tamoxifen use directly related to the increased gynecomastia occurrences seen with modern day steroid users?

When we bring our attention to Clomid, we find less research is available on long term human toxicity, probably because of the relatively short term (3-4 week) clinical application for ovarian stimulation,59 although long term follow ups with patients who received Clomid for ovulation induction have shown an increased risk of developing uterine cancer.74 This is to be expected, since many of the same carcinogenic tendencies found with tamoxifen are the same effects seen with clomiphene.44,45,57,58 Upon analysis of anecdotal reports from Clomid and Nolvaldex - tamoxifen citrate - users, we see the typical short term side effects of low libido, erectile dysfunction, and emotional instability – despite many men showing normalized testosterone and estrogen levels during the use of these SERM’s. Research on male breast cancer patients also shows frequent reports of low libido, thrombosis (arterial blockage), and hot flashes with tamoxifen use.47 Another common side effect associated with both SERMs, but more common with Clomid, is the loss of visual accuracy and development of visual "tracers", due to the ocular toxicity.

As the medical community became more aware of the side-effects associated with clomiphene and tamoxifen treatment, newer and safer SERMs, such as toremifene and raloxifene hit the developmental fast track. Toremifene appears to be less liver toxic, but it is an analog of tamoxifen, so it also carries many of the related genotoxic effects. Raloxifene appears to be even safer by being the least liver toxic, and not having any potential issue with the uterus or prostate. Unfortunately, raloxifene has been associated with a higher incidence of thromboembolism52 (arterial blockage), and also has very low oral absorption, making it an expensive alternative at a typical 120mg/day dose. Still, raloxifene could presumably be equally effective as Clomid or Nolvadex at restoring HPTA function, while imparting less side effects. Newer SERMs are already being evaluated such as bazedoxifene, arzoxifene, and lasofoxifene, in hopes of reducing risk even further.

Another SERM that may be useful for post cycle therapy is resveratrol.87,88 Resveratrol is a natural polyphenol extracted from grape skin, that has recently been under heavy research for its cancer fighting effects in the breast, prostate and liver. Contrary to Nolva or Clomid, resveratrol appears to actually have beneficial effects on the liver,70 as well as having multiple benefits on cardiovascular health by limiting LDL oxidation and improving endothelial function.71-73 Improved blood vessel function may be a mechanism by which resveratrol improves erectile function in many men. Research also suggests that resveratrol may actually extend life, by reducing oxidative stress on organs such as the heart,77 and preventing the metabolic syndrome by fighting insulin resistence. It’s becoming well known that insulin resistance is a leading cause of low testosterone.82 More specifically, improving insulin sensitivity will increase your leydig cell sensitivity, and therefore increase the testes response to LH.

It should be pointed out that resveratrol may not be the best choice to combating emergency gynecomastia, due to its lower binding affinity to the human ER of about 90x less than tamoxifen, and about 30x less than clomiphene.75,76 However, considering that resveratrol is a pure estrogen antagonist at the pituitary,89 while Clomid has mixed agonist/antagonistic effects,90-94 resveratrol could be a suitable substitute for post cycle therapy. Aside from acting as a SERM, resveratrol can also help control estrogen by actually limiting aromatase enzyme production.82 Based on the research, it appears that at least 100mg/day would needed to increase LH, FSH and testosterone production.84 This is comparable dose of resveratrol found in an advanced topical based product, Dermacrine Sustain (found here - http://www.primordialperformance.com).

Admittedly, no steroid users are dropping dead from a 4 week protocol of Nolva or Clomid, and many will say "the consequences far outweigh the benefits" -- but why deal with the potential consequences when alternatives are available?

Peptides for testicular recovery

It’s a common practice these days for experienced bodybuilders to implement some dosage of IGF-1 either during or after a cycle to "pick up" a lagging body part, or to preserve gains in muscle. Growth Hormone (GH) is also a versatile drug for cutting or bulking, with increasing popularity as it becomes more affordable. The value of IGF-1 and GH becomes so much more significant when we realize there integral role in testicular function. In fact, it seems that these hormones are more effective at building testes, than muscles.

Research has shown GH to be vitally important in testicular function, but it is generally accepted that the beneficial effects are directly mediated by hGH’s conversion to IGF-1.98 As many of you know, IGF-1 is created in the liver by GH, upon interacting with insulin. So, we will be focusing on the usage and benefits of IGF-1, rather than GH, as it seems more cost effective and directly related to our purpose of optimizing recovery.

In short, IGF-1 increases steroidogenic acute regulatory protein (sTAR), and cholesterol side chain cleaving enzyme (CYP 11A)99. These are both rate-limiting steps and are critical factors for converting cholesterol into hormones, such as testosterone. IGF-1 also has the ability to increase the concentration of steroidogenic enzymes in the testes, such as 3b HSD.100 IGF-1 can also increase the testes sensitivity to LH and hCG by increasing the number of LH receptors.

These positive effects on testicular function make IGF-1 an ideal drug for post cycle therapy. A dose of IGF-1 Lr3 at 80mcg/day, split two times per day, would likely be the most cost effective dose.

In conclusion, we have learned that utilizing hCG during a steroid cycle will significantly prevent testicular degeneration. This helps create a seamless transition from "on cycle" to "off cycle". Then, by avoiding the deleterious SERMs such Clomid and Nolvadex and opting for safer alternatives, you can seemingly avoid any sort of post cycle crash, while maintaining a strong libido and uncompromised emotional health.
Good post!
 
b_light said:
Yeah i don't really do PL i just did strongman for fun because they had the shit at my gym and i had to play with it and it just took off from there and its alot easier, form goes out the window for the most part with strongman and you just need to focus on one thing not the long haul and you can eat shit all the time.
lol eat is the key word for me. I am a chef :worried:
 
Currently on:
Weeks 1-12 600mg/w Test-testosterone cypionate split into Sat and Wed
Weeks 1-8 50mg/ED trenbolone-A
A little bit of Fermara ED throughout (this stuff tastes like shit)

I started on the third week of October. Bench and dead are both up about 20lb so far. Weight has gone up about 5lb and I am leaner. I haven't been able to eat as much as I would like because of my schedule (40hrs work and taking 16hrs in classes). Up until a few days ago I was only getting in about 2500-3200 calories but now have rescheduled some things in my daily calender and added more to each meal and am now getting around 3700 (thanks whole milk). Totally looking forward to Thanksgiving and Christmas. I will dominate that kitchen. Sides are minimal (other than increased leanness and strength :P). I have had trenbolone cough once so far, no bacne or hairloss, and had a bit of insomnia but marijuana and/or Tylenol pm have helped me counter that problem :).
 
I"m taking a small break, I"m 3 weeks into a small cycle designed to clean my receptors a litttle bit. 3 weeks ago I dropped everything from my cycle (dropped my deca, anavar, and dbol), and kept 750 mg of test ew with 4 ius of hgh 6 days a week. I only lost 2 lbs since then. I'm going to start a bulker that is going to be: 1 gram of sustanon ew, 400 mg deca ew, and 50 mg anadrol the first 4 weeks, with 4 ius of hgh ed.
 
I'm starting on my 6th week today of a
1-4 dianabol 25mg ED
1-10 test E 500mg EW

I have gone from 212 to 230 so far. I know a lot of it is water weight though. I've been drinking water like a horse. I've noticed a big change in my muscularity, but still have more fat then I want. I don't have the privilage right know of really seeing shredded muscles, so I'm going to start taking in less calories and start up a 4x3 carb cycle. It's cool being big and bulky, but it's much cooler being shredded.
 
holy ghost said:
I have ran that exact cycle. Jesus bro 148??

4x a week cardio may be a bit excessive judging by your body type, How tall are you more importantly.. You are an ecto right???

I am 5'7...I am just having a problem getting my the fat off my stomach..it seems all 8% is residing in my lower abs...Piss's me off b/c even when I was alot alot heavier and juicing, I was shredded....Even when I was 175lbs you could see my abs, here is a pic...

Bodybuildingblur.jpg


I had to take aloooooong time off due to some "trouble" and starting my own business...
 
damn bro you lok realy familiar lol

If youre trying to put on size you need to drop cardio to 1-2 times a week bro

put on some size, AND FAT INEVITABLY

THEN month or two later then cut bro you will keep your size its what pros do
 
I am trying to put on lean mass..I am 162 with 6 weeks left and just added the winnie..if I could put on 5-8 more lbs and drop the body fat by like 2-4% i would be happy....I do cardio on a empty stomach but then go eat a huuuuuge breakfast....my idea is to force my body to use the fat as energy when i havent eaten and then maintain high calories throughout the day to grow...
 
I am starting that this week...remember I was asking you about that..lol...I am going 4x3...what exercises while get the fat off the lower abs..my abs now look like that picture..the first 4 show but not the bottem....
 
1tight_tl said:
I am starting that this week...remember I was asking you about that..lol...I am going 4x3...what exercises while get the fat off the lower abs..my abs now look like that picture..the first 4 show but not the bottem....
There is no such thing as spot fat reduction bro. The fat comes of where ever it wants to. Think of your body like a lake, And your abs are the middle of the lake. When a lake starts to dry up the middle is the last to go. The outer parts go first just like fat on the body. Legs,arms,chest,and other parts loose fat first just like the outer parts of a lake dry up first. The middle is always the last to go and it's always the hardest to get to dry up. There is no way to get the middle of a lack to dry up before the outer parts and your body is the same way. There is only calories in calories out when it comes to your body, and there is only water in and water out when it comes to a lake. More calories have to be going out then coming in to get the body to dry up. More water has to be going out then coming in to get a lake to dry up.

So just keep doing what has been working for you until the lake dries up bro.
 
holy ghost said:
Just grab Flax, its a lot easier digestable

I get fish burbs from fish oil

Flax is PACKED with omega EFA's

I got a site to get 50lb bales of whey-protein too!!


Pm me the site bro. thanks
 
702daswoll1 said:
Im 6 weeks into this.

1-5 Dianabol 40mg ed.
1-7 trenbolone A 50mg ed.
1-20 Test 750mg ew.
1-18 Equipoise - boldenone undecylenate - 600mg ew.
7-15 Anavar 50mg ed.

Yeah I ran trenbolone and Dianabol at the same time and loved it. Not sure what my liver thought of it though.
My bodyfat is now at 9.3% on digital calipers. I would venture to say I was 12-14 percent at the beginning of the run. Ghost you saw the before pic. Going to take and post up some pics at 10 weeks in but looking vascular and hard strength is through the roof.


Thats a monster cycle. Looking forward to teh pics. I need some new jack off material anways. lol
 
cal_21 said:
Can't wait to try it when cycle # 2 comes along. I'm so tempted to throw it in @ the end of this one :evil: ...but I won't LOL.


Sorry to hear about your clavicle. that sucks ass and there isnt much to do for it but narcs and a clavicle strap. how the fuck did you sleep at night?
 
needtogetaas said:
All you guys doing long cycles 12 weeks and up. You need to run your HCG - human chorionic gonadotropin - during cycle on long cycles like this imo. I think you should run it during cycle all the time, but even more so on longer cycles. A good dose would be 500ui's ew.


How do you feel about all oral on cycles? I wanna do a short 6-8 weeker of something in feb maybe march. whatcha got in mind?
 
holy ghost said:
OH I was about to say

fuck Nigga Im not helping you anymore!! lol

Get on some Deca-Durabolin - nandrolone decanoate - bro.


I thought you were from the school of thought the A.AS help healing/regeneration of muscles and tendons?
 
errn247 said:
Sorry to hear about your clavicle. that sucks ass and there isnt much to do for it but narcs and a clavicle strap. how the fuck did you sleep at night?

I had a pin put in it. That helped a lot I think. I was laid up for a long time. For the first 2-3 wks think it is, I only left the recliner to go to the bathroom/shower. I had to sleep in it so there'd be less pressure on it.
I was out of commission for months and months.

They gave me a lot of norcos and a lottttttt of percocet. It was a pretty depressing time, just over medicating myself thinking about how I was missing the playoffs and just life in general. Got uber fucking fat. It sucked bro.

My 2nd surgery was much easier to recover from. Losing all my strength was a big factor in me doing my cycle now. Otherwise I would have probably waited a yr or so.
 
Nothing wrong with oral only, dont let the hype fool you.

Dbol/proviron
Winny/var
var/tbol


GET BLOODWORK - main thing

there was a guy on here back a while ago, he was in the army, and all he could get ahold of was dbols, so he took 25mg ED for a YEAR PLUS got blood work done and was COMPLETLEY FINE.
 
needtogetaas said:
There is no such thing as spot fat reduction bro. The fat comes of where ever it wants to. Think of your body like a lake, And your abs are the middle of the lake. When a lake starts to dry up the middle is the last to go. The outer parts go first just like fat on the body. Legs,arms,chest,and other parts loose fat first just like the outer parts of a lake dry up first. The middle is always the last to go and it's always the hardest to get to dry up. There is no way to get the middle of a lack to dry up before the outer parts and your body is the same way. There is only calories in calories out when it comes to your body, and there is only water in and water out when it comes to a lake. More calories have to be going out then coming in to get the body to dry up. More water has to be going out then coming in to get a lake to dry up.

So just keep doing what has been working for you until the lake dries up bro.

that was a good example needto , well said

peace
 
needtogetaas said:
There is no such thing as spot fat reduction bro. The fat comes of where ever it wants to. Think of your body like a lake, And your abs are the middle of the lake. When a lake starts to dry up the middle is the last to go. The outer parts go first just like fat on the body. Legs,arms,chest,and other parts loose fat first just like the outer parts of a lake dry up first. The middle is always the last to go and it's always the hardest to get to dry up. There is no way to get the middle of a lack to dry up before the outer parts and your body is the same way. There is only calories in calories out when it comes to your body, and there is only water in and water out when it comes to a lake. More calories have to be going out then coming in to get the body to dry up. More water has to be going out then coming in to get a lake to dry up.

So just keep doing what has been working for you until the lake dries up bro.

So I completely get what your saying and understand that i cant spot reduce...by the your analogy was the shit bro.. :) ...I am trying to reduce bf% overall. I figured that by doing cardio on a empty stomach in the morning that the body would be forced to use fat as fuel b/c i had not fed it any calories yet? correct?
 
1tight_tl said:
So I completely get what your saying and understand that i cant spot reduce...by the your analogy was the shit bro.. :) ...I am trying to reduce bf% overall. I figured that by doing cardio on a empty stomach in the morning that the body would be forced to use fat as fuel b/c i had not fed it any calories yet? correct?
I feel this can work. Others think not. Just do it and if it works for you then fuck what people say.
 
Hey needto, any other suggestions on getting the abs just extremely shredded...any good excersises for the lower abs below the bellybutton where guys get that fat pouch?
 
yes

abdominal hyperextesions

russian decline twists for obliques

stiff hanging leg lifts

you wont see muscle till you burn the fat bro!
 
holy ghost said:
yes

abdominal hyperextesions

russian decline twists for obliques

stiff hanging leg lifts

you wont see muscle till you burn the fat bro!

I can see the top 4 now and I am woking on burning the fat...this way when its gone, they are ready :) ....straight leg on the hanging leg lifts or bent? and what are the hyperextensions? I will try to go find a pic
 
straight leg

set the ab station up to the MOST incline you can , Hang by your tippy toes, then do situps/twists

also, try to kneel on the ground at the cable station and grab the rope and do rope crunches ( end in muslim kneel)
 
holy ghost said:
straight leg

set the ab station up to the MOST incline you can , Hang by your tippy toes, then do situps/twists

also, try to kneel on the ground at the cable station and grab the rope and do rope crunches ( end in muslim kneel)

Sweet !! thanks ghost...
 
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