Not sure if anyone's actually interested in the risks of insulin resistance caused by exo-GH, but I've got another question along these lines.
Assuming the insulin resistance results from the mechanisims in my post above (fatty acid mobilization and shift towards insulin-resistant type II muscle fibers), then wouldn't someone with low or very low total bodyfat offset at least some of those risks?
Assuming the insulin resistance results from the mechanisims in my post above (fatty acid mobilization and shift towards insulin-resistant type II muscle fibers), then wouldn't someone with low or very low total bodyfat offset at least some of those risks?