SteelWeaver
New member
A very interesting study on vinegar and glycogen repletion. So if you're training hard but kinda broke - hit the apple cider vinegar
http://www.ncbi.nlm.nih.gov:80/entrez/query.fcgi?CMD=Display&DB=PubMed
J Nutr 2001 Jul;131(7):1973-7
Acetic acid feeding enhances glycogen repletion in liver and skeletal muscle of
rats.
Fushimi T, Tayama K, Fukaya M, Kitakoshi K, Nakai N, Tsukamoto Y, Sato Y.
Central Research Institute, Mitsukan Group Company Limited, Handa 475-8585, Japan.
[email protected]
To investigate the efficacy of the ingestion of vinegar in aiding recovery from fatigue, we examined
the effect of dietary acetic acid, the main component of vinegar, on glycogen repletion in rats. Rats
were allowed access to a commercial diet twice daily for 6 d. After 15 h of food deprivation, they
were either killed immediately or given 2 g of a diet containing 0 (control), 0.1, 0.2 or 0.4 g acetic
acid/100 g diet for 2 h. The 0.2 g acetic acid group had significantly greater liver and gastrocnemius
muscle glycogen concentration than the control group (P < 0.05). The concentrations of citrate in
this group in both the liver and skeletal muscles were >1.3-fold greater than in the control group (P
> 0.1). In liver, the concentration of xylulose-5-phosphate in the control group was significantly
higher than in the 0.2 and 0.4 g acetic acid groups (P < 0.01). In gastrocnemius muscle, the
concentration of glucose-6-phosphate in the control group was significantly lower and the ratio of
fructose-1,6-bisphosphate/fructose-6-phosphate was significantly higher than in the 0.2 g acetic
acid group (P < 0.05). This ratio in the soleus muscle of the acetic acid fed groups was <0.8-fold
that of the control group (P > 0.1). In liver, acetic acid may activate gluconeogenesis and inactivate
glycolysis through inactivation of fructose-2,6-bisphosphate synthesis due to suppression of
xylulose-5-phosphate accumulation. In skeletal muscle, acetic acid may inhibit glycolysis by
suppression of phosphofructokinase-1 activity. We conclude that a diet containing acetic acid may
enhance glycogen repletion in liver and skeletal muscle.
http://www.ncbi.nlm.nih.gov:80/entrez/query.fcgi?CMD=Display&DB=PubMed
J Nutr 2001 Jul;131(7):1973-7
Acetic acid feeding enhances glycogen repletion in liver and skeletal muscle of
rats.
Fushimi T, Tayama K, Fukaya M, Kitakoshi K, Nakai N, Tsukamoto Y, Sato Y.
Central Research Institute, Mitsukan Group Company Limited, Handa 475-8585, Japan.
[email protected]
To investigate the efficacy of the ingestion of vinegar in aiding recovery from fatigue, we examined
the effect of dietary acetic acid, the main component of vinegar, on glycogen repletion in rats. Rats
were allowed access to a commercial diet twice daily for 6 d. After 15 h of food deprivation, they
were either killed immediately or given 2 g of a diet containing 0 (control), 0.1, 0.2 or 0.4 g acetic
acid/100 g diet for 2 h. The 0.2 g acetic acid group had significantly greater liver and gastrocnemius
muscle glycogen concentration than the control group (P < 0.05). The concentrations of citrate in
this group in both the liver and skeletal muscles were >1.3-fold greater than in the control group (P
> 0.1). In liver, the concentration of xylulose-5-phosphate in the control group was significantly
higher than in the 0.2 and 0.4 g acetic acid groups (P < 0.01). In gastrocnemius muscle, the
concentration of glucose-6-phosphate in the control group was significantly lower and the ratio of
fructose-1,6-bisphosphate/fructose-6-phosphate was significantly higher than in the 0.2 g acetic
acid group (P < 0.05). This ratio in the soleus muscle of the acetic acid fed groups was <0.8-fold
that of the control group (P > 0.1). In liver, acetic acid may activate gluconeogenesis and inactivate
glycolysis through inactivation of fructose-2,6-bisphosphate synthesis due to suppression of
xylulose-5-phosphate accumulation. In skeletal muscle, acetic acid may inhibit glycolysis by
suppression of phosphofructokinase-1 activity. We conclude that a diet containing acetic acid may
enhance glycogen repletion in liver and skeletal muscle.