Please Scroll Down to See Forums Below 
R
Ross
Guest
The Anabolic Bible
Chapter 1
*The Three ESSENTIAL Phases of A Proper Steroid Therapy*
There are three essential stages of a Proper "Steroid Therapy":
1.) The Steroid Cycle: Anabolic steroids are utilized over the course of many weeks, sometimes many months, as the bodybuilder aquires as much muscle mass as possible, or while dieting to preserve muscle and aid in fatloss.
2.) Active Recovery(Pre-PCT): This is the period of time DIRECTLY AFTER YOUR CYCLE. DO NOT GO STRAIGHT INTO post cycle therapy! This is why you experience a POST-CYCLE CRASH! Utilizing an ACTIVE RECOVERY PERIOD, will allow the body to BEGIN producing testosterone once again, while still remaining in an ANABOLIC STATE!
3.) Post Cycle Therapy: Now that your HPTA has began recovering, and you have successfully transitioned out of your steroid cycle, it is now time to FULLY RESTORE THE HPTA. Now is the time for your FULL agressive post cycle therapy regimen, including HCG, Aromasin, and Nolvadex if desired.
4.) The Bridge:(*Optional) Now that you are FULLY RECOVERED and your PCT is complete, you can begin bridging while awaiting your FULL CYCLE. This will allow you to make GREATER THAN NATURAL GAINS, while still maintaining normal testosterone levels.
For a complete list of compounds that do NOT CAUSE HPTA SHUTDOWN, please see my current article "Using Anabolic Steroids WIthout HPTA SHUTDOWN".
Chapter 2
**The STANDARD Cycle**
**The STANDARD Cycle**
A PERFECT CYCLE CONSISTS OF *BOTH*, SLOW AND FAST-ACTING STEROIDS!
In most cases, a LONG-esterfied injectable steroid such as Testosterone Enanthate would function as the BASE of the cycle, providing slow but consistent gains throughout the entire cycle's duration.
A SHORT-acting steroid is used in either the BEGINNING of a cycle or at the END of a cycle. Typically, Dianabol and Anadrol are used at the beginning of a cycle to provide STRENGTH and MASS gains BEFORE the base(and/or secondary injectable) "KICK IN". A FAST-ACTING stroid such as Anavar or Winstrol is used at the END of a cycle and RIGHT UP UNTIL post cycle therapy, while the LONG-acting steroid SLOWLY exits the system.
An optimal cycle contains several components:
1.) A Base: This is the usually the most powerful compound in the stack, and it is typically ran throughout the entire duration of the cycle. Most individuals will choose Testosterone for a base, but for those uncomfortable using testosterone; Trenbolone, Equipoise, Masteron, and even Primobolan can be used instead. In your case, the base will be testosterone Enanthate.
2.) The JUMPSTARTER: This compound must be FAST-ACTING, so as to generate muscle and strength gains BEFORE YOUR BASE AND YOUR ASSISTANT KICK IN. Jumpstarting usually applies to BULKING cycles, but they can be used effectively in CUTTING cycles as well. Dianabol and Anadrol are the most popular steroids for JUMPSTARTING a cycle, because they both induce incredible strength and mass in a very short period of time; but it important to note that there are MANY other drugs that can be used for this purpose that are usually overlooked. Halotestin at 30-40mgs will provide ENORMOUS strength at the beginning of your cycle, making it GREAT to jumpstart BULKING cycles AND CUTTING cycles. Injectable Winstrol can ALSO be used to jumpstart BULKING CYCLES and CUTTING CYCLES. The Oral is too weak to act as an effective JUMPSTARTER. I would say the same for Anavar and Turinabol. Trenbolone Acetate and Testosterone Propionate(or suspension) are also very popular choices used to jumpstart BULKING cycles. You will be using Dianabol as your JUMPSTARTER.
3.) The FINISHER: A FAST-acting steroid MUST BE UTILIZED at the end of your cycle's duration! You MUST remain ANABOLIC right up until post cycle therapy! If you FAIL to use a fast-acting steroid such as Winstrol or Oxandrolone during your FINAL weeks while your BASE and your ASSISTANT leave your system, you will LOSE GAINS BEFORE YOU EVEN BEGIN post cycle therapy. This is one of the BIGGEST mistakes people make. Test E will not leave your system for at LEAST 3 weeks after your FINAL SHOT. Therefore, you MUST remain anabolic during these 3 weeks when your adrogen levels PLUMMET! You will be using Winstrol Inject as your Finisher.
Failure to uitlize ANY of the 3 ESSENTIAL COMPONENTS will result in a cycle that is LESS THAN OPTIMAL.*
For an intermediate or advanced user, a SECONDARY ANABOLIC called the "assistant" should be added for OPTIMAL results.
*) The Assistant: This compound is ran alongside the BASE for the majority of the cycle, providing a synergistic effect throughout it's duration. Most people will choose either Deca or Equipoise, usually using testosterone as the base. Trenbolone, Masteron, and Primobolan also make great Assistants. You will be using Deca as your Assiatant.
Weeks 1-6: Dianabol, 30mgs ED
Weeks 1-10: Test E, 500mgs
Weeks 1-10: Deca, 400mg
Weeks 10-14: Winstrol Inject, 75mgs ED
The Standard Cycle is designed for OPTIMUM anabolism, utilizing precise strategies in order to gain and sustain the most muscle possible!
Chapter 3
*Using Anabolic Steroids Without HPTA SHUTDOWN!*
RESEARCH SHOWS THAT NOT ALL STEROIDS CAUSE SHUTDOWN!
- No more "Post Cycle Crash"!
- Use CERTAIN anabolic steroids DURING PCT! *(Pre-PCT)
- Run a complete cycle WITHOUT HPTA SHUTDOWN!
Some steroids only REDUCE TESTOSTERONE PRODUCTION(to varying degrees), whereas other steroids will SHUTDOWN the HPTA resulting in a complete cessation of androgen production.
*NOT ALL ANDROGENS CAUSE SHUTDOWN*
"Shutdown", is defined by a COMPLETE inhibition of the Pituitary/Testes, resulting in a TOTAL cessation of endogenous androgen production.
SOME androgens will only SUPPRESS endogenous androgen production, resulting in a DECREASED testosterone level, but not a complete shutdown. (Turinabol, Anavar, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan)
Very Androgenic/Progestenic/Estrogenic steroids(Trenbolone, Nandrolone, Anadrol, Testosterone) cause a COMPLETE shutdown of endogenous hormone production.
Steroids that cause an OVERSATURATION(too many receptors activated) of these various hormone receptors, WILL CAUSE SHUTDOWN. Steroids that DO NOT CAUSE an OVERSATURATION of ANY of these various hormone receptors, will NOT cause SHUTDOWN!
The distinction between SUPRESSION and SHUTDOWN is utterly important, as steroids that cause LESS supression of endogenous hormones will allow for greater retention of gains upon ending the cycle, and a quicker, easier recovery!
The Following steroids will NOT SHUTDOWN THE HPTA:
Turinabol, Anavar, Proviron, Halotestin, Wistrol, Equipoise, Dianabol, Masteron, Primobolan, Clostebol, and 4-ADiol.
Pre-PCT: PRE-PCT allows the HPTA to begin LH/FSH output, while still receiving additional anabolic support. This is the peroid of time where we utilize a NON-inhibitory steroid while the endogenous testosterone level begins to recover. This occurs PRIOR TO FULL PCT, so that by the time we begin full PCT the HPTA has already began recovering.
Active RECOVERY: The HPTA BEGINS to restore endogenous testosterone production once it detects the body's androgen level beginning to decline(end of cycle).
Therefore, HPTA CAN BEGIN TO RECOVER WHILE STILL IN AN ANABOLIC STATE!
The following drugs can be used during Active Recovery:
Anavar/Proviron= 40mgs/25mgs
Anavar/Masteron= 40mgs/300mgs
Primobolan/Masteron= 300mgs/300mgs
Turinabol/Proviron= 40mgs/25mgs
Turinabol/Masteron= 40mgs/300mgs
Winstrol/Masteron= 50mgs/300mgs
Dianabol/Proviron= 15mgs/25mgs
Dianabol/Masteron= 15mgs/300mgs
Examples...
In a SHORT CYCLE:
Weeks 1-4: Testosterone Propionate, 100mgs ED
Weeks 1-4: Dianabol, 50mgs ED
Weeks 1-4: NPP, 400mgs
Weeks 4-8: **PRE-PCT(ACTIVE RECOVERY)**
Weeks 8-?: **POST CYCLE THERAPY**
A Standard Cycle:
Weeks 1-6: Dianabol, 30mgs ED
Weeks 1-10: Testosterone Enanthate, 500mgs
Weeks 8-12: Winstrol, 100mgs ED
Weeks 12-16: **PRE-PCT(ACTIVE RECOVERY) **
Weeks 16-26: **POST CYCLE THERAPY**
DO NOT end your cycle ABRUPTLY! Don't just END your cycle cold-turkey! If you are SHUTDOWN, full restoration can take weeks and even MONTHS. Therefore, one should REMAIN ON minimally-inhibitive STEROIDS(HPTA) in an attempt to MAINTAIN the gains they made while ON CYCLE, while STILL BEGINNING TO RECOVER TESTOSTERONE PRODUCTION. On top of that, one still continues to progess from the mild additional anabolic support.
NOT only does it mean that you can run a COMPLETE CYCLE with NO SHUTDOWN whatsoever(as long as the right compounds, dosages, and durations are used), it also means that if you ARE SHUTDOWN from your cycle, you do NOT HAVE TO COME RIGHT OFF CYCLE! Actually, it is BETTER TO STAY ON CYCLE WHILE YOUR ENDOGENOUS TESTOSTERONE LEVEL BEGINS TO INCREASE!
You may also run a cycle that COMPLETELY AVOIDS SHUTDOWN:
Weeks 1-6: Dianabol, 40mgs ED
Weeks 1-10: Anavar, 50mgs ED
Weeks 1-10: Masteron, 100mgs EOD
Or
Weeks 1-6: Dianabol, 40mgs ED
Weeks 1-10: Primobolan, 500mgs
Weeks 6-14: Turinabol, 60mgs ED
And many many more! There are tons of NON-inhibitory cycles that you can devise using my my list above for your guideline. Your days of HPTA suffering are over!
By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
The Hypothalamus has Androgen, Estrogen, and Progesterone receptors.
Each and EVERY anabolic steroid affects these receptors DIFFERENTLY. Some steroids affect ALL receptors, while some only affect ONE type of receptor, while others have very little effect on ANY of these receptors.
UNDERSTANDING WHICH steroids affect which receptors, and to WHAT DEGREE, will FULLY enable the steroid user to COMPLETELY and systematically AVOID HPTA SHUTDOWN! By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
Steroids that cause an OVERSATURATION(too many receptors activated) of these various hormone receptors, WILL CAUSE SHUTDOWN.
Steroids that DO NOT CAUSE an OVERSATURATION of ANY of these various hormone receptors, will NOT cause SHUTDOWN!
The Following drugs either DIRECTLY or INDIRECTLY activate ESTROGEN receptors, to varying degrees:
Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
The Following drugs either DIRECTLY or INDIRECTLY activate PROGESTERONE receptors, to varying degrees:
Nandrolone
Trenbolone
Oxymetholone
The Following drugs activate Androgen receptors, to varying degrees:
Testosterone
Methandrostenolone
Mathandriol
Oxymetholone
Nandrolone
Boldenone
Trenbolone
Halotestin
Oxandrolone
Stanzolol
Chlorodehydromethltestosterone
Methyltestosterone
Methenolone...
(ALL AAS*)
As we can see, the steroids that cause HPTA SHUTDOWN either OVERSATURATE ONE SPECIFIC receptor, or they activate too many TOTAL receptors(Androgen/Estrogen/Progesterone)
For instance, Trenbolone causes HPTA SHUTDOWN because it OVERSATURATES BOTH, the ANDROGEN and the PROGESTERONE receptors. Testosterone causes SHUTDOWN because it converts to ESTROGEN and DHT, therefore, it oversaturates the Androgen/Estrogen receptors.
As we can ALSO SEE, the steroids that DO NOT cause SHUTDOWN of the HPTA, do NOT oversaturate ANY of the different hormone receptors, and thus, do NOT cause SHUTDOWN.
Methenolone(Primobolan) does not possess ANY Estrogenic or Progestational ACTIVITY WHATSOEVER. It does, by virtue of being an anabolic steroid, posses a SMALL Androgenic component. Because it lacks ANY ESTROGENIC/PROGESTATIONAL component, and it lacks a strong Androgenic component, it WILL NOT CAUSE SHUTDOWN! Oxandrolone(Anavar) posseses NO Estrogenic/Progestational component either. AND, it also lacks a strong androgenic component. Thus, Anavar will NOT cause shutdown.
By understanding WHICH steroids cause SHUTDOWN and which steroids do NOT, we can formulate a perfect EXTENDED CYCLE.
*It must also be noted, that ANY steroid in LARGE enough DOSAGES for long enough DURATIONS, can cause SHUTDOWN of the HPTA.
NOT ALL ANDROGENS CAUSE SHUTDOWN*
-------------------------------------------------------------------------
Horm Metab Res. 1984 Sep;16(9):492-7.Related Articles, Links
Effect of non aromatizable androgens on LHRH and TRH responses in primary testicular failure.
Spitz IM, Margalioth EJ, Yeger Y, Livshin Y, Zylber-Haran E, Shilo S.
We have assessed the gonadotropin, TSH and PRL responses to the non aromatizable androgens, mesterolone and fluoxymestrone, in 27 patients with primary testicular failure. All patients were given a bolus of LHRH (100 micrograms) and TRH (200 micrograms) at zero time. Nine subjects received a further bolus of TRH at 30 mins. The latter were then given mesterolone 150 mg daily for 6 weeks. The remaining subjects received fluoxymesterone 5 mg daily for 4 weeks and 10 mg daily for 2 weeks. On the last day of the androgen administration, the subjects were re-challenged with LHRH and TRH according to the identical protocol. When compared to controls, the patients had normal circulating levels of testosterone, estradiol, PRL and thyroid hormones. However, basal LH, FSH and TSH levels, as well as gonadotropin responses to LHRH and TSH and PRL responses to TRH, were increased. Mesterolone administration produced no changes in steroids, thyroid hormones, gonadotropins nor PRL. There was, however, a reduction in the integrated and incremental TSH secretion after TRH. Fluoxymesterone administration was accompanied by a reduction in thyroid binding globulin (with associated decreases in T3 and increases in T3 resin uptake). The free T4 index was unaltered, which implies that thyroid function was unchanged. In addition, during fluoxymesterone administration, there was a reduction in testosterone, gonadotropins and LH response to LHRH. Basal TSH did not vary, but there was a reduction in the peak and integrated TSH response to TRH. PRL levels were unaltered during fluoxymesterone treatment.(ABSTRACT TRUNCATED AT 250 WORDS
[R]
--------------------------------------------------------------------------------
Last edited:
