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testicular shrinkage

mcgirkz

New member
i am just finishing up a light cycle. i have noticed some testicular atrophy. how much nolvadex shuold i be taking? or will this even help get this back to normal?

i was thinking 40mg ED for a week and then 20mg ED after that for another week. is there anything else i could try besides HCG. i don;t have access to this.

any thoughts
 
Maybe I read it wrong I am asuiming you are done with your cycle and trying to restore natural function/size to your balls.......
 
champion said:
Maybe I read it wrong I am asuiming you are done with your cycle and trying to restore natural function/size to your balls.......

you are ssuming correct. i still have a few days of orals left. but will be done early next week.
and isn't clomid and nolvadex similar in there actions? why wouldn't my nolvadex be good enough.
i've also read about "nettles". or is that more for prostate stuff?

i have some trib, which i plan on taking too

thanks for everybodies help so far
 
If it was a light cycle like you said, then do the CLOMID.

200mgs the first day. Then 100mgs for the next 13 days. then 50 for the final 7. THat should help out a lot.

Nolv is an ant-e blocker.
 
Steak Helmet said:
If it was a light cycle like you said, then do the CLOMID.

200mgs the first day. Then 100mgs for the next 13 days. then 50 for the final 7. THat should help out a lot.

Nolv is an ant-e blocker.

according to what i have read. they are both similar drugs in effect.

here's a quote: "So which one should you use? Well personally, I'd have to say Nolvadex. Both as an on-cycle anti-estrogen and a post-cycle therapy. As an anti-estrogen its simply much stronger, demonstrated by the fact that better results are obtained with 20-40 mg than with 100-150 mg of clomid. For post-cycle, this plays a key role as well. It deactivates rebound estrogen much faster and more effective. But most importantly, Nolvadex has a direct influence on bringing back natural testosterone, where as clomid may actually have a slight negative influence. The reason being that Tamoxifen (as in Nolvadex) seems to increase the responsiveness of LH (luteinizing hormone) to GnRH (gonadtropin releasing hormone), whereas clomid seems to decrease the responsiveness a bit"

taken from here: http://www.bodybuilding.com/fun/catnolv.htm


with that said, neither one will help with my nut size and i need hcg for that.

can i get this from a doctor? or do i need to go black market?
 
well actually once they come back "online" they should regain there size....but in reality I am not versed in this subject as i have never really been off. I use nolva fior anti e and arimidex for heavy cycles I have used clomid while i went off for no more than 6 weeks a couple times and felt that it helped restore me somewhat..........as for ball size dont care never used hcg.
 
That article you posted by Big Cat is right out of Bill Llewellyn's mouth. That's pretty funny, I never saw that. Nice theory but clomid is much better than nolva for recovery because CLOMID will start your LH faster. But on your testes. Yes you need H C G. But if I were you I wouldn't worry about it. Most likely they'll be fine in a month or two anyways.


by Bill Roberts - Clomid is the anti-estrogen of choice for improving recovery of natural testosterone production after a cycle, improving testosterone production of endurance athletes, and is also effective in reducing risk of gynecomastia during a cycle employing aromatizable steroids.

While it has been claimed that Clomid "stimulates" production of LH and therefore of testosterone, in fact Clomid’s activity is achieved not by stimulation of the hypothalamus and pituitary, but by blocking their inhibition by estrogen.

Clomid is a mixed estrogen agonist/antagonist (activator/blocker) which, when bound to the estrogen receptor, puts it in a somewhat different conformation (shape) than does estradiol. The estrogen receptor requires binding of an estrogen or drug at its binding site and also the binding of any of several cofactors at different sites. Without the binding of the cofactor, the estrogen receptor is inactive. Different tissues use different cofactors. Some of these cofactors are able to bind to the estrogen receptor/Clomid complex, but others are blocked due to the change in shape. The result is that in some tissues Clomid acts as an antagonist -- the cofactor used in that tissue cannot bind and so the receptor remains inactive -- and in others Clomid acts as an agonist (activator), because the cofactors used in that tissue are able to bind.

Clomid is an effective antagonist in the hypothalamus and in breast tissue. It is an effective agonist in bone tissue, and for improving blood cholesterol.

Clomid also has the property of reducing the adverse effect of exercise-induced damage of muscle tissue. This is very significant for endurance athletes but is not very significant, if at all significant, with reasonable weight training. Clomid does not perceptibly affect gains of the weight trainer either favorably or adversely in my experience.

The drug seems to have estrogenic effects on mood, which can be beneficial (improving relationships with women by improving empathy) or can yield depression or PMS-like symptoms, but for most users there is no significant effect either way.

The claim that duration of intake should not exceed 10-14 days is incorrect. Clinical studies with male patients have been for periods of a year or longer. This error probably originates from the fact that, for use in women, due to the menstrual cycle there would obviously be no point in trying to stimulate ovulation all four weeks of the month. Thus, use in women is limited to 10-14 days. That limitation is not because of toxicity.

Clomid is in fact useful throughout a cycle if aromatizable drugs are being used. I do think however that to be conservative, one should use it no more than 2/3 of the time throughout the year or a little less.
 
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