There could be many more interrelated bits to the stress-cortisol story than meets the eye. The amounts of binding protein for cortisol can be altered along with the amounts of blood binding proteins for sex hormones. Expect alterations in either one or the other to have some cross-talk with each other and the rest of the system. Consider anorexia: we might find that very low E can lead to less of both binding proteins for both E and C. So the 'total' cortisol is low, while the 'free' cortisol is surprisingly high. It isn't so clear what net effect this has at the time, nor if/when the person starts to re-feed. And the low binding protein levels for E result in some other subtle mess-ups that tend to drive the endocrinologists crazy with a bit of a chicken-or-the-egg situation. It is difficult to sort out cause versus effect. But in such cases the hypothalamus often sorts itself out and symptoms go away after a few days/months of rest, recuperation, and possibly a winning lottery ticket. Either way though, the body is then stuck with some extra fat cells that grew/divided during the dysfuntional time-period, yet don't go away afterwards. Nasty.
On a more practical level: How many have noticed that students enter college healthy and lithe, go through several years of wasted artificial stress, all-nighters etc. and then graduate with assorted bumps, bulges and lovehandles etc.? This effect seems to happen even if the kid stays at home and eats the same stuff that has been apparently okay so far. The stressed-out body seems to handle food and partition it quite differently than a calm well-rested body. Could wacked-out cortisol levels (among other things) be a contributor here?