here's a good article for you Retard. Next time don't post Shit you know nothing of.
OK, I've been meaning to post this up for a while and since it's a slow day in the library...
Since developing my understanding of physiology and pathology, and whilst having a lot of spare time on my hands whilst holidaying in Thailand over christmas, I came up with some theories regarding that phenomenon we juicers know as "The tren cough".
I wanted to basically try to rationalise and hence bust some of the dogma and incorrect thinking behind this, but my ideas are after all only theories (albeit backed up by sound physiological knowledge), so I would like input from other posters, to help refine/alter my 'definitive' answer to the tren cough issue.
I'll try and keep this to an easily understandable format, but my Spike-addled brain just wants to vomit words all over the screen in any order so please excuse me if my formatting is a bit crappy
OK, for those that have yet to experience it, the tren cough phenomenon is:
You inject a preparation containing trenbolone acetate and if unlucky, you experience a strong desire to cough repeatedly, with a strange taste in your throat. You may become dizzy, and feel the need to lie down. The urge passes after anywhere upto 10 minutes. No lasting symptoms.
The 'standard' theory states that this is due to the high levels of BA/BB required to dissolve the acetate-estered tren and hold it in solution. The theory is that you have injected this (unwittingly) into a vein and that the BA hits the lungs and makes you cough.
Most people seem to think that only tren will cause this phenomenon, and this is what got me thinking , after I experienced the cough several times, with gear that did not have any trenbolone in it. However the homebrew was of a rather high (425mg/ml) concentration. I have also experienced the cough with other gear, though I can't remembre what it was, other than that it contained no trenbolone.
SO, my modified/updated hypothesis on the 'tren' cough (I put tren in inverted commas because the cough is not limited to tren only) is this.
1. You inject gear with either sufficiently high total concentration, OR that contains tren ace. I.e. a preparation that has a high level of co-solvents (BA/BB).
2. Unluckily you hit a vein and so a proportion of the injection directy enters the venous circulation. Next stop: The vena cava.
3. Having entered the right side of the heart, the injected preparation now enters the pulmonary circulation, where it travels to the lungs. These are the first capillaries encountered by your injected 'accident' and they have a tiny lumen (diameter) and large surface area (for exchange of oxygen and CO2).
4. Because of this inherent design of the lungs to get rid of vapour and gas, and because the steroid hormone requires the BA/BB to hold it in solution, once the injected gear hits the lungs, the BA 'boils' away, and the crystals of steroid drop out of solution and remain (partially) within the lung tissue.
5. The sudden presence of crystals within the lungs is irritant to them and is what triggers the cough. The BA being expelled through the bronchioles is what causes the strange, chemically taste in the back of the throat.
6. The lungs adapt to the presence of the crystals, which causes the coughing to subside, plus macrophages within the lungs, combined with capilary blood flow, removes the crystals over time. With say tren ace, at perhaps 100mg/ml, this process happens quite quickly. However, on those occassions where I experienced the cough from injecting 425mg/ml gear, if I breathed in deeply, even several hours later, I would experience the sensation of 'tightness' and irritation within the lung tissue. In fact it would take up to 24 hours to feel completely 'normal' again.
So in summary, my additions/modifications to the 'tren' cough accepted theory, is that:1. It is not limited strictly to trenbolone preparations.
2. It is the presence of steroid crystals within the lungs that causes the cough.
Not exactly radical thinking I know, but I wanted to put this out there, in the hope of clearing up another piece of steroid-related dogma.
Hit me with yout thoughts gentlemen. Any physiologists, health professionals, medicos, doctors, scientists, whoever, are most welcome to add their thoughts/critiscise/correct my physiology, etc
Regards
bushy