How to use Growth Hormone????

[Smokescreen]

It's been 2 years since I last used gear. The reason why I don't use it anymore is because I think I'm fairly big enough for my height. Not only that but I have very small joints which means I don't need to pack on that much to look big. Not only that but I haven't been able to get my hands on anything good anymore. The stuff that I get is either underdosed, fake or just plain makes me sick! What I do use now is growth hormone. Not fake stuff...but the real deal from the pharmacy. I've been taking 6iu's everyday for the last year and a half. I take 3iu's before bed and upon waking up. I don't know if this is the correct way but a lot of people have told me to do it like this. From taking nothing but the GH, I have slept better, been happier, and have had a bit more energy. The only problem is that I'm not losing fat like the way GH is all hyped on! When I started taking it late last year, I was at 10%bf. Now after over a year I'm at 8%bf. I can't seem to lose anymore! I'm eating clean, lifting for 90 minutes at the gym and then jogging for 30minutes. Then I work at textile factory that requires more strenous physical work. I'm not getting fatter, but I'm just not getting leaner at all! I've heard of people taking GH and eating everything in site and shedding fat. How come it's not happening to me?? What I'm I doing wrong?? Any suggestion s and/or advice would be greatly appreciated. Thank you!

 

[ANOINTED]

how old are u???

u shouldnt be takin it b4 bed...

ur body will natually be producing growth once u get into REM and u dnt want to fuck that up

 

[gilly6993]

eating everything in site AND shedding fat.....ain't gonna happen bro....

 

[Smokescreen]

gilly...I never said I was going to eat everything in site. This is just what I have seen and heard of others that have been taking GH. Anointed....I am 29 years old.

 

[thx9000]

You've been on 6iu ED for a year and a half? I sure hope you have been avoiding carbs. I also remember DrJMW stating that continued use of exo-GH will result in hypothyroidism so you need to use Armour or T3 along with it. I'm not sure what the mechanism would be though. Thyroid hormone does stimulate GH secretion, and at the same time GH replacement is known to increase the rate of T4 to T3 conversion so maybe the increased T3 results in the eventual decline in TSH?

 

[gilly6993]

I sure hope you have been avoiding carbs

huh

 

[thx9000]

huh

Use of rHGH results in insuline resistance as a result of fatty acid mobilization:
http://www.ncbi.nlm.nih.gov/entrez/...ve&db=PubMed&list_uids=12679422&dopt=Abstract

That some times, at least in sick little kids, doesn't reverse post-therapy:
http://www.ncbi.nlm.nih.gov/entrez/...ve&db=PubMed&list_uids=12584529&dopt=Abstract

So, I would think one would want to avoid carbs when on longterm GH doses that are something like 3X normal levels.

 

[gilly6993]

well....I've been on GH for about a year now and sure don't avoid carbs.....are those studies on healthy 30 year old adults....probably not

 

[thx9000]

well....I've been on GH for about a year now and sure don't avoid carbs.....are those studies on healthy 30 year old adults....probably not

No, the first was on a GH deficient adults that were put on low dose GH therapy and the second was on not well children. There are other studies, but I doubt many of them address 30 healthy adults or people using 3x what a normal human would produce.

If the mode of action is as the study states, ie: the mobilization of fatty acids and a shift towards insulin-resistant type II muscle fibers, then I don't see what being healty a healthy 30yo adult is going to accomplish.

 

[thx9000]

Not sure if this helps much (just confuses me):

http://diabetes.diabetesjournals.org/cgi/content/abstract/50/8/1891

These results indicate that cellular insulin resistance induced by chronic GH treatment in 3T3-L1 adipocytes is caused by uncoupling between activation of PI 3-kinase and its downstream signals, which is specific to the insulin-stimulated PI 3-kinase pathway. This effect of GH might result from the altered subcellular distribution of IRS-1–associated PI 3-kinase.

and...

http://www.ebmonline.org/cgi/content/abstract/227/3/149

long-term exposure to GH is, in general, associated with hyperinsulinemia, which leads to a reduction of IR levels and an impairment of its tyrosine kinase activity. The signals of GH and insulin may converge at post-receptor levels. The signaling pathway leading to activation of PI 3-kinase appears to be an important site of convergence between the signals of these two hormones and seems to be mediated principally by IRS-1. Rodent models of chronic GH excess have been useful tools to investigate the mechanism by which GH induces insulin resistance. Decreased IR, IRS-1, and IRS-2 tyrosyl phosphorylation in response to insulin was found in skeletal muscle, whereas a chronic activation of the IRS-PI 3-kinase pathway was found in liver. The induction of the expression of proteins that inhibit IR signaling such as suppressors of cytokine signaling (SOCS)-1 and -6 may also be involved in this alteration.