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does anyone know the way smoking weed causes bitch tits?
how does smoking weed cause gyno
- Thread starter perfectspecimen
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rippedtrojan
New member
Beezers said:
true. weed isn't not really that bad. praise jah!
JUICESEEKER
New member
I am not sure but i believe that weed can lower testosterone and cause hormonal imbalance. Less testosterone means more estrogen therefore gyno.
Rémy!
New member
Marihuana and sex: a critical survey.Abel EL.
Marihuana usage is associated with a life-style that involves earlier and more frequent sexual activity. Marihuana usage does not affect human male testosterone levels significantly, but does adversely affect sperm production. Animal studies have not found consistent changes in weights of male sexual organs but have corroborated the adverse effects of cannabinoid compounds on sperm production. The biological significances of these effects on sperm production are unclear, however, since there is no evidence that human marihuana users or male animals given cannabinoid compounds are less fertile or are at risk for dominant lethal mutations. Cannabinoid compounds reliably inhibit ovulation in animals and are associated with depressed luteinizing hormone (LH) levels in both female and male animals. The decreased LH levels appear to be due to both hypothalamic and ovarian sites of action. Treatment with cannabinoid compounds is also associated with lower testosterone levels in male and lower prolactin levels in female animals. Effects on progesterone levels are inconclusive. Cannabinoid compounds do not possess estrogenic activity. Despite some consistencies in the data in virtually every study conducted with animals, there has been a basic confounding between direct drug action and secondary effects resulting from drug-induced decreases in food and water consumption and attendant weight loss. Almost all of the adverse effects of cannabinoid exposure on reproductive organs can be attributed to these secondary effects.
Marijuana: interaction with the estrogen receptor.
Sauer MA, Rifka SM, Hawks RL, Cutler GB Jr, Loriaux DL.
Crude marijuana extract competed with estradiol for binding to the estrogen receptor of rat uterine cytosol. Condensed marijuana smoke also competed with estradiol for its receptor. Pure delta 9-tetrahydrocannabinol, however, did not interact with the estrogen receptor. Ten delta 9-tetrahydrocannabinol metabolites also failed to compete with estradiol for its receptor. Of several other common cannabinoids tested, only cannabidiol showed any estrogen receptor binding. This was evident only at very high concentrations of cannabidiol. Apigenin, the aglycone of a flavinoid phytoestrogen found in cannabis, displayed high affinity for the estrogen receptor. To assess the biological significance of these receptor data, estrogen activity was measured in vivo with the uterine growth bioassay, using immature rats. Cannabis extract in large doses exhibited neither estrogenic nor antiestrogenic effects. Thus, although estrogen receptor binding activity was observed in crude marijuana extract, marijuana smoke condensate and several known components of cannabis, direct estrogenic activity of cannabis extract could not be demonstrated in vivo.
_______
It appears marijuana use does not elicit estrogenic effects.
Marihuana usage is associated with a life-style that involves earlier and more frequent sexual activity. Marihuana usage does not affect human male testosterone levels significantly, but does adversely affect sperm production. Animal studies have not found consistent changes in weights of male sexual organs but have corroborated the adverse effects of cannabinoid compounds on sperm production. The biological significances of these effects on sperm production are unclear, however, since there is no evidence that human marihuana users or male animals given cannabinoid compounds are less fertile or are at risk for dominant lethal mutations. Cannabinoid compounds reliably inhibit ovulation in animals and are associated with depressed luteinizing hormone (LH) levels in both female and male animals. The decreased LH levels appear to be due to both hypothalamic and ovarian sites of action. Treatment with cannabinoid compounds is also associated with lower testosterone levels in male and lower prolactin levels in female animals. Effects on progesterone levels are inconclusive. Cannabinoid compounds do not possess estrogenic activity. Despite some consistencies in the data in virtually every study conducted with animals, there has been a basic confounding between direct drug action and secondary effects resulting from drug-induced decreases in food and water consumption and attendant weight loss. Almost all of the adverse effects of cannabinoid exposure on reproductive organs can be attributed to these secondary effects.
Marijuana: interaction with the estrogen receptor.
Sauer MA, Rifka SM, Hawks RL, Cutler GB Jr, Loriaux DL.
Crude marijuana extract competed with estradiol for binding to the estrogen receptor of rat uterine cytosol. Condensed marijuana smoke also competed with estradiol for its receptor. Pure delta 9-tetrahydrocannabinol, however, did not interact with the estrogen receptor. Ten delta 9-tetrahydrocannabinol metabolites also failed to compete with estradiol for its receptor. Of several other common cannabinoids tested, only cannabidiol showed any estrogen receptor binding. This was evident only at very high concentrations of cannabidiol. Apigenin, the aglycone of a flavinoid phytoestrogen found in cannabis, displayed high affinity for the estrogen receptor. To assess the biological significance of these receptor data, estrogen activity was measured in vivo with the uterine growth bioassay, using immature rats. Cannabis extract in large doses exhibited neither estrogenic nor antiestrogenic effects. Thus, although estrogen receptor binding activity was observed in crude marijuana extract, marijuana smoke condensate and several known components of cannabis, direct estrogenic activity of cannabis extract could not be demonstrated in vivo.
_______
It appears marijuana use does not elicit estrogenic effects.
rippedtrojan
New member
Green said:
i'd probably be around that too.
good thing i'm not!
Stay off all rec drugs if you know what's good for
you...including alcohol, tobacco and weed. You
may think you're pretty freakin' cool now. Wait
till you are 40 and sneaking around your garage
with a bunch of other fat 40 year old losers hiding
your drug use from you kids. After a while you
won't even be getting high anymore. You'll be
smokin' just to feel normal. Find some other way
to get high that you won't regret for the rest of
your life.
you...including alcohol, tobacco and weed. You
may think you're pretty freakin' cool now. Wait
till you are 40 and sneaking around your garage
with a bunch of other fat 40 year old losers hiding
your drug use from you kids. After a while you
won't even be getting high anymore. You'll be
smokin' just to feel normal. Find some other way
to get high that you won't regret for the rest of
your life.
The often heard assertion that THC causes gyno is not related to estrogenic activity of the compound, but rather to the ability of THC to suppress luteinizing hormone (LH). A number of studies have shown this (1) (2) (3). Other studies have failed to show an effect (4).
As with evidence supporting the theory behind marijuana induced gyno, there are conflicting studies about whether there is even a correlation between marijuana use and gyno. There is a much stronger correlation between alcoholism and gyno than between weed and gyno.
(1) NIDA Res Monogr 1984;44:46-64
Endocrine effects of marijuana in the male: preclinical studies.
(2) Drug Alcohol Depend 1992 Apr;30(1):59-63
Chronic effects of marihuana smoking on luteinizing hormone, follicle-stimulating hormone and prolactin levels in human males.
(3) : J Pharmacol Exp Ther 1986 Jun;237(3):862-6
Marihuana smoking suppresses luteinizing hormone in women.
(4) 1: Drug Alcohol Depend 1991 Aug;28(2):121- 8
Effects of chronic marijuana use on testosterone, luteinizing hormone, follicle stimulating hormone, prolactin and cortisol in men and women.
As with evidence supporting the theory behind marijuana induced gyno, there are conflicting studies about whether there is even a correlation between marijuana use and gyno. There is a much stronger correlation between alcoholism and gyno than between weed and gyno.
(1) NIDA Res Monogr 1984;44:46-64
Endocrine effects of marijuana in the male: preclinical studies.
(2) Drug Alcohol Depend 1992 Apr;30(1):59-63
Chronic effects of marihuana smoking on luteinizing hormone, follicle-stimulating hormone and prolactin levels in human males.
(3) : J Pharmacol Exp Ther 1986 Jun;237(3):862-6
Marihuana smoking suppresses luteinizing hormone in women.
(4) 1: Drug Alcohol Depend 1991 Aug;28(2):121- 8
Effects of chronic marijuana use on testosterone, luteinizing hormone, follicle stimulating hormone, prolactin and cortisol in men and women.
I know a guy who got bitch tits from weed. Go figure. Who cares if there is no estrogen related evidence, the fact is that it does something to your body that induces gyno. Besides, sure weed is cool, but why are you even in the gym if you smoke weed. Any kind of smoke hinders your progress.
Further to my last, here is a good read in case you are interested. This is the link:
http://911papers.virtualave.net/classical_sciences/biology/Gynecomastia.htm
Furthermore this is a quote from the paper,
Many drugs can cause gynecomastia by several mechanisms. The drugs can either act directly as estrogens or cause and increase in plasma estrogen levels. "Boys and young men are particularly sensitive to estrogen, and can develop gynecomastia after the use of dermal ointments containing estrogen or after the ingestion of milk or meat from estrogen-treated animals."(Isselbacher, 2038) Some examples of drugs that may have cause gynecomastia include Cannabinoids (methane and marijuana), Psychotropics (pheno-thiazine, butyrophenone and reserpine), Antihypertensives (reserpine, alpha-methyldopa and spironolactone), Cardiac (digitalis), Gastrointestinal (cimetidine, metoclopramide and domperidone), Antituburculous (isoniazid), Cytoxic (cyclophospha-mide, mustine, vincristine and mitotane) and Hormonal (sex steroids, gonadotropins and antiandrogens). Use of these drugs, however, will rarely cause gynecomastia. In some instances, the feminization is due to effects of drugs on liver functions.
YOU WANT STUDIES, then use human studies. We are not rats.
http://911papers.virtualave.net/classical_sciences/biology/Gynecomastia.htm
Furthermore this is a quote from the paper,
Many drugs can cause gynecomastia by several mechanisms. The drugs can either act directly as estrogens or cause and increase in plasma estrogen levels. "Boys and young men are particularly sensitive to estrogen, and can develop gynecomastia after the use of dermal ointments containing estrogen or after the ingestion of milk or meat from estrogen-treated animals."(Isselbacher, 2038) Some examples of drugs that may have cause gynecomastia include Cannabinoids (methane and marijuana), Psychotropics (pheno-thiazine, butyrophenone and reserpine), Antihypertensives (reserpine, alpha-methyldopa and spironolactone), Cardiac (digitalis), Gastrointestinal (cimetidine, metoclopramide and domperidone), Antituburculous (isoniazid), Cytoxic (cyclophospha-mide, mustine, vincristine and mitotane) and Hormonal (sex steroids, gonadotropins and antiandrogens). Use of these drugs, however, will rarely cause gynecomastia. In some instances, the feminization is due to effects of drugs on liver functions.
YOU WANT STUDIES, then use human studies. We are not rats.
chanmanfoo
New member
ok buddy. thanks for the tip, and by the way is this from personal experience?Zeke_B said:
foo
chanmanfoo
New member
shrek1971 said:
what the fuck do you mean by this? why are you in the gym?
foo
Zeke_B said:
I love ignorant fuckers weed is not addictive meaning that its effects will not lessen overtime in fact most studies say they increase with time and you become more sensitive to it. And my teacher witha ph.d from princeton agrees what school did you goto to and what degree do you have his is in computer science
Iron Monger
New member
Although I don't smoke a lot, I do enjoy some herbage here and there. When I was younger I used to smoke the hell out of it and never got gyno. I think gyno comes from the combination of smoking, sitting on your ass for 12 hours playing video games, and eating 2 pizza's and drinking 4 liters of coke.
Personally, if I smoke a lot, I loose motivation and don't get important things done. I also have friends that smoke daily and are more motivated than me. Smoking weed can also aid bodybuilders eat more since it helps that dry-ass chicken breast taste like a big mac
Personally, if I smoke a lot, I loose motivation and don't get important things done. I also have friends that smoke daily and are more motivated than me. Smoking weed can also aid bodybuilders eat more since it helps that dry-ass chicken breast taste like a big mac
Speaking of pot that reminds me.................time to pack the bowl!!!!!!:havoc:
This discusion has been on this board for sooooo long, if you want to smoke, go ahead. Obviously it's not good for you. It burns brain cells(but so does caffine), damages your lungs, and a few other, but it's no where near as bad as anyother drug out there, especially roids.
So roll a fatty for this pimp daddy
This discusion has been on this board for sooooo long, if you want to smoke, go ahead. Obviously it's not good for you. It burns brain cells(but so does caffine), damages your lungs, and a few other, but it's no where near as bad as anyother drug out there, especially roids.
So roll a fatty for this pimp daddy
D
DaddyX
Guest
Zeke_B , shrek1971 - both of you might have possibly made the most ignorant replies i have ever seen on this board, there tons of guys that smoke a little weed and work out very intense and look great. And Zeke your reply was just horrible bro, go give your lectures to someone else.........i dont even know what to say but god damn i hope you were not serious Zeke_B
Marijuana: interaction with the estrogen receptor
Sauer MA, Rifka SM, Hawks RL, Cutler GB Jr, Loriaux DL.
Crude marijuana extract competed with estradiol for binding to the estrogen receptor of rat uterine cytosol. Pure delta 9-tetrahydrocannabinol, however, did not interact with the estrogen receptor. Ten delta 9-tetrahydrocannabinol metabolites also failed to compete with estradiol for its receptor. Of several other common cannabinoids tested, only cannabidiol showed any estrogen receptor binding. This was evident only at very high concentrations of cannabidiol. Apigenin, the aglycone of a flavinoid phytoestrogen found in cannabis, displayed high affinity for the estrogen receptor. To assess the biological significance of these receptor data, estrogen activity was measured in vivo with the uterine growth bioassay, using immature rats. Cannabis extract in large doses exhibited neither estrogenic nor antiestrogenic effects. Thus, although estrogen receptor binding activity was observed in crude marijuana extract, and several known components of cannabis, direct estrogenic activity of cannabis extract could not be demonstrated in vivo.
Treatment of Tourette Syndrome with Delta-9-Tetrahydrocannabinol (Delta(9)-THC): No Influence on Neuropsychological Performance.
Muller-Vahl KR, Prevedel H, Theloe K, Kolbe H, Emrich HM, Schneider U.
Previous studies provide evidence that marijuana (Cannabis sativa) and delta-9-tetrahydrocannabinol (Delta(9)-THC), the major psychoactive ingredient of marijuana, respectively, are effective in the treatment of tics and behavioral problems in Tourette syndrome (TS). It, therefore, has been speculated that the central cannabinoid receptor system might be involved in TS pathology. However, in healthy marijuana users there is an ongoing debate as to whether the use of cannabis causes acute and/or long-term cognitive deficits. In this randomized double-blind placebo-controlled study, we investigated the effect of a treatment with up to 10 mg Delta(9)-THC over a 6-week period on neuropsychological performance in 24 patients suffering from TS. During medication and immediately as well as 5-6 weeks after withdrawal of Delta(9)-THC treatment, no detrimental effect was seen on learning curve, interference, recall and recognition of word lists, immediate visual memory span, and divided attention. Measuring immediate verbal memory span, we even found a trend towards a significant improvement during and after treatment. Results from this study corroborate previous data suggesting that in patients suffering from TS, treatment with Delta(9)-THC causes neither acute nor long-term cognitive deficits. Larger and longer-duration controlled studies are recommended to provide more information on the adverse effect profile of THC in patients suffering from TS.Neuropsychopharmacology (2003) 28, 384-388. doi:10.1038/sj.npp.1300047
PMID: 12589392 [PubMed - in process]
Effects of acute smoked marijuana on complex cognitive performance.
Hart CL, van Gorp W, Haney M, Foltin RW, Fischman MW.
Division on Substance Abuse, New York State Psychiatric Institute, 1051 Riverside Drive, New York, NY 10032, USA.
Although the ability to perform complex cognitive operations is assumed to be impaired following acute marijuana smoking, complex cognitive performance after acute marijuana use has not been adequately assessed under experimental conditions. In the present study, we used a within-participant double-blind design to evaluate the effects acute marijuana smoking on complex cognitive performance in experienced marijuana smokers. Eighteen healthy research volunteers (8 females, 10 males), averaging 24 marijuana cigarettes per week, completed this three-session outpatient study; sessions were separated by at least 72-hrs. During sessions, participants completed baseline computerized cognitive tasks, smoked a single marijuana cigarette (0%, 1.8%, or 3.9% Delta(9)-THC w/w), and completed additional cognitive tasks. Blood pressure, heart rate, and subjective effects were also assessed throughout sessions. Marijuana cigarettes were administered in a double-blind fashion and the sequence of Delta(9)-THC concentration order was balanced across participants. Although marijuana significantly increased the number of premature responses and the time participants required to complete several tasks, it had no effect on accuracy on measures of cognitive flexibility, mental calculation, and reasoning. Additionally, heart rate and several subjective-effect ratings (e.g., "Good Drug Effect," "High," "Mellow") were significantly increased in a Delta(9)-THC concentration-dependent manner. These data demonstrate that acute marijuana smoking produced minimal effects on complex cognitive task performance in experienced marijuana users.
Publication Types:
Clinical Trial
PMID: 11682259 [PubMed - indexed for MEDLINE]
Neuropsychological performance in long-term cannabis users.
Pope HG Jr, Gruber AJ, Hudson JI, Huestis MA, Yurgelun-Todd D.
McLean Hospital, Harvard Medical School, 115 Mill St, Belmont, MA 02478, USA.
BACKGROUND: Although cannabis is the most widely used illicit drug in the United States, its long-term cognitive effects remain inadequately studied. METHODS: We recruited individuals aged 30 to 55 years in 3 groups: (1) 63 current heavy users who had smoked cannabis at least 5000 times in their lives and who were smoking daily at study entry; (2) 45 former heavy users who had also smoked at least 5000 times but fewer than 12 times in the last 3 months; and (3) 72 control subjects who had smoked no more than 50 times in their lives. Subjects underwent a 28-day washout from cannabis use, monitored by observed urine samples. On days 0, 1, 7, and 28, we administered a neuropsychological test battery to assess general intellectual function, abstraction ability, sustained attention, verbal fluency, and ability to learn and recall new verbal and visuospatial information. Test results were analyzed by repeated-measures regression analysis, adjusting for potentially confounding variables. RESULTS: At days 0, 1, and 7, current heavy users scored significantly below control subjects on recall of word lists, and this deficit was associated with users' urinary 11-nor-9-carboxy-Delta9-tetrahydrocannabinol concentrations at study entry. By day 28, however, there were virtually no significant differences among the groups on any of the test results, and no significant associations between cumulative lifetime cannabis use and test scores. CONCLUSION: Some cognitive deficits appear detectable at least 7 days after heavy cannabis use but appear reversible and related to recent cannabis exposure rather than irreversible and related to cumulative lifetime use.
Sauer MA, Rifka SM, Hawks RL, Cutler GB Jr, Loriaux DL.
Crude marijuana extract competed with estradiol for binding to the estrogen receptor of rat uterine cytosol. Pure delta 9-tetrahydrocannabinol, however, did not interact with the estrogen receptor. Ten delta 9-tetrahydrocannabinol metabolites also failed to compete with estradiol for its receptor. Of several other common cannabinoids tested, only cannabidiol showed any estrogen receptor binding. This was evident only at very high concentrations of cannabidiol. Apigenin, the aglycone of a flavinoid phytoestrogen found in cannabis, displayed high affinity for the estrogen receptor. To assess the biological significance of these receptor data, estrogen activity was measured in vivo with the uterine growth bioassay, using immature rats. Cannabis extract in large doses exhibited neither estrogenic nor antiestrogenic effects. Thus, although estrogen receptor binding activity was observed in crude marijuana extract, and several known components of cannabis, direct estrogenic activity of cannabis extract could not be demonstrated in vivo.
Treatment of Tourette Syndrome with Delta-9-Tetrahydrocannabinol (Delta(9)-THC): No Influence on Neuropsychological Performance.
Muller-Vahl KR, Prevedel H, Theloe K, Kolbe H, Emrich HM, Schneider U.
Previous studies provide evidence that marijuana (Cannabis sativa) and delta-9-tetrahydrocannabinol (Delta(9)-THC), the major psychoactive ingredient of marijuana, respectively, are effective in the treatment of tics and behavioral problems in Tourette syndrome (TS). It, therefore, has been speculated that the central cannabinoid receptor system might be involved in TS pathology. However, in healthy marijuana users there is an ongoing debate as to whether the use of cannabis causes acute and/or long-term cognitive deficits. In this randomized double-blind placebo-controlled study, we investigated the effect of a treatment with up to 10 mg Delta(9)-THC over a 6-week period on neuropsychological performance in 24 patients suffering from TS. During medication and immediately as well as 5-6 weeks after withdrawal of Delta(9)-THC treatment, no detrimental effect was seen on learning curve, interference, recall and recognition of word lists, immediate visual memory span, and divided attention. Measuring immediate verbal memory span, we even found a trend towards a significant improvement during and after treatment. Results from this study corroborate previous data suggesting that in patients suffering from TS, treatment with Delta(9)-THC causes neither acute nor long-term cognitive deficits. Larger and longer-duration controlled studies are recommended to provide more information on the adverse effect profile of THC in patients suffering from TS.Neuropsychopharmacology (2003) 28, 384-388. doi:10.1038/sj.npp.1300047
PMID: 12589392 [PubMed - in process]
Effects of acute smoked marijuana on complex cognitive performance.
Hart CL, van Gorp W, Haney M, Foltin RW, Fischman MW.
Division on Substance Abuse, New York State Psychiatric Institute, 1051 Riverside Drive, New York, NY 10032, USA.
Although the ability to perform complex cognitive operations is assumed to be impaired following acute marijuana smoking, complex cognitive performance after acute marijuana use has not been adequately assessed under experimental conditions. In the present study, we used a within-participant double-blind design to evaluate the effects acute marijuana smoking on complex cognitive performance in experienced marijuana smokers. Eighteen healthy research volunteers (8 females, 10 males), averaging 24 marijuana cigarettes per week, completed this three-session outpatient study; sessions were separated by at least 72-hrs. During sessions, participants completed baseline computerized cognitive tasks, smoked a single marijuana cigarette (0%, 1.8%, or 3.9% Delta(9)-THC w/w), and completed additional cognitive tasks. Blood pressure, heart rate, and subjective effects were also assessed throughout sessions. Marijuana cigarettes were administered in a double-blind fashion and the sequence of Delta(9)-THC concentration order was balanced across participants. Although marijuana significantly increased the number of premature responses and the time participants required to complete several tasks, it had no effect on accuracy on measures of cognitive flexibility, mental calculation, and reasoning. Additionally, heart rate and several subjective-effect ratings (e.g., "Good Drug Effect," "High," "Mellow") were significantly increased in a Delta(9)-THC concentration-dependent manner. These data demonstrate that acute marijuana smoking produced minimal effects on complex cognitive task performance in experienced marijuana users.
Publication Types:
Clinical Trial
PMID: 11682259 [PubMed - indexed for MEDLINE]
Neuropsychological performance in long-term cannabis users.
Pope HG Jr, Gruber AJ, Hudson JI, Huestis MA, Yurgelun-Todd D.
McLean Hospital, Harvard Medical School, 115 Mill St, Belmont, MA 02478, USA.
BACKGROUND: Although cannabis is the most widely used illicit drug in the United States, its long-term cognitive effects remain inadequately studied. METHODS: We recruited individuals aged 30 to 55 years in 3 groups: (1) 63 current heavy users who had smoked cannabis at least 5000 times in their lives and who were smoking daily at study entry; (2) 45 former heavy users who had also smoked at least 5000 times but fewer than 12 times in the last 3 months; and (3) 72 control subjects who had smoked no more than 50 times in their lives. Subjects underwent a 28-day washout from cannabis use, monitored by observed urine samples. On days 0, 1, 7, and 28, we administered a neuropsychological test battery to assess general intellectual function, abstraction ability, sustained attention, verbal fluency, and ability to learn and recall new verbal and visuospatial information. Test results were analyzed by repeated-measures regression analysis, adjusting for potentially confounding variables. RESULTS: At days 0, 1, and 7, current heavy users scored significantly below control subjects on recall of word lists, and this deficit was associated with users' urinary 11-nor-9-carboxy-Delta9-tetrahydrocannabinol concentrations at study entry. By day 28, however, there were virtually no significant differences among the groups on any of the test results, and no significant associations between cumulative lifetime cannabis use and test scores. CONCLUSION: Some cognitive deficits appear detectable at least 7 days after heavy cannabis use but appear reversible and related to recent cannabis exposure rather than irreversible and related to cumulative lifetime use.
variation
New member
pft
soy products get the same bullshit about estrogen-like phytohormones....... 'estrogen like' hormones don't grow boobs as far as I know, now I've read some documentation on a study done on mice... come on.. mice are not human, anyway.. these mice showed estrogen receptors to become stimulated? does this mean it's more likely to develop gyno if more estrogen is present in the system? I'm not sure, and the study wasn't done on humans so inconclusive, I know lots of people that smoke pot without gyno, and there is lots of people that use soy stuff that do not have gyno. about 40 year olds in garage comment, though I'm not saying that will happen, I have seen it.. pretty sad.
soy products get the same bullshit about estrogen-like phytohormones....... 'estrogen like' hormones don't grow boobs as far as I know, now I've read some documentation on a study done on mice... come on.. mice are not human, anyway.. these mice showed estrogen receptors to become stimulated? does this mean it's more likely to develop gyno if more estrogen is present in the system? I'm not sure, and the study wasn't done on humans so inconclusive, I know lots of people that smoke pot without gyno, and there is lots of people that use soy stuff that do not have gyno. about 40 year olds in garage comment, though I'm not saying that will happen, I have seen it.. pretty sad.
Weed is good. Sure I may be leaving a few pounds on the table, but you know what, who gives a fuck, cause when I'm old, its all gonna be gone anyway. The people I really worry about are those who go at everything to the extreme. While your discipline is certainly admirable, its misguided. Don't live to workout. Don't be the Meathead. Meatheads are miserable looking people. I workout because I enjoy it, I take roides because I reached my natural limit and want to be able to see some progress, I smoke weed because it feels good. Thats all I need to know.
SMOKING WEED DOES NOT CAUSE GYNO
The whole gyno/lowered sperm count is all just govt propaganda...the type of shit D.A.R.E. and cops tell people...
And unlike the "this is your brain on drugs" ads of the 80's/90's which millions bought into, the general reaction to the new anti-marijuana/anti-drug commercials is one of disgust or laughter. Many people see thru the blatant lies now, plus the internet has made much more information available.
Legalizing weed would be like letting someone innocent off death row. Marijuana never belonged being grouped with hard drugs anyway. Its more healthy for you than alcohol & tobacco by far, but which are legal? Could that be due to lobbying from big tobacco companies who know the legalization of weed would lead to a massive decrease in tobacco smoking??
Imagine going on a weed break at work or college instead of going out to have a cigarette...
The whole gyno/lowered sperm count is all just govt propaganda...the type of shit D.A.R.E. and cops tell people...
And unlike the "this is your brain on drugs" ads of the 80's/90's which millions bought into, the general reaction to the new anti-marijuana/anti-drug commercials is one of disgust or laughter. Many people see thru the blatant lies now, plus the internet has made much more information available.
Legalizing weed would be like letting someone innocent off death row. Marijuana never belonged being grouped with hard drugs anyway. Its more healthy for you than alcohol & tobacco by far, but which are legal? Could that be due to lobbying from big tobacco companies who know the legalization of weed would lead to a massive decrease in tobacco smoking??
Imagine going on a weed break at work or college instead of going out to have a cigarette...
what the fuck do you mean by this? why are you in the gym?
foo
HEY CHANMANFOO, I dont mean to pick a fight but All I am saying is that smoking weed as a chronic can, because I have seen it, cause gyno. I am not saying it causes gyno for everyone but yes it does. I am not against the shit, I just dont do it anymore because I end up getting a chronic cough and can not make as good progress. I used to be a 175 lbs person, average build, to 220 lbs 14% BF in less than 3 years. Try quitting smoking for a year and see how much real weight you can gain.
This is a dead discussion. If you like weed, then fucking smoke it. If you want to make as much progress in the gym then dont smoke it and be hard core. Isnt that what going to the gym is all about, for personal improvement?
And as far as calling me ignorant you dont have the right. But hey who knows you could be a fucking guru of smoking bowls and you know everything there is about human physiology and drug pathology. FUCK YOU!!!
foo
HEY CHANMANFOO, I dont mean to pick a fight but All I am saying is that smoking weed as a chronic can, because I have seen it, cause gyno. I am not saying it causes gyno for everyone but yes it does. I am not against the shit, I just dont do it anymore because I end up getting a chronic cough and can not make as good progress. I used to be a 175 lbs person, average build, to 220 lbs 14% BF in less than 3 years. Try quitting smoking for a year and see how much real weight you can gain.
This is a dead discussion. If you like weed, then fucking smoke it. If you want to make as much progress in the gym then dont smoke it and be hard core. Isnt that what going to the gym is all about, for personal improvement?
And as far as calling me ignorant you dont have the right. But hey who knows you could be a fucking guru of smoking bowls and you know everything there is about human physiology and drug pathology. FUCK YOU!!!
JungleRizla
New member
we know this we can't be talking about this on this site...not the place for it...to each his own on the subject. But don't call someone down for the love of blasting a blunt. We don't critique you on your alcohol consumption or steroid consumption. Once again to re-align anyone...WEED DOES NOT GIVE YOU GYNO...your predisposition and use of crank DOES !!! ...end of story, got it, get it , GOOD! BUMP FOR JAH!
S
satchboogie
Guest
nice6pac said:
no..
youre the ignorant one my friend!
weed is addictive.
i've been there. done it. 5 foot glass and the kindest herbs that cal produces. smoked weed like dorian shoots test.
smokers are in denial when they say what you claimed in the abovementioned.
Hahaha
First off you can thank MaryJ being illegal due to ignorant prejiduce.
Second you can thank almost all negative repor of pot on Ronald Reagan, who after taking office squashed Carters efforts to legalize pot and declare a 'War on drugs'!!
Thirdly studies are often flawed and left to interpretation by who is funding the fucking study. Just think about all the 'studies' cigarette company's did to show that nicotine was niether addictive or harmfull.
I would rather support pot smokers:
They don't start fights for no reason in bars
They don't steel to buy more due to addiction
And I would much rather share the roads with someone who is high rather than someone who's been drinkinig
First off you can thank MaryJ being illegal due to ignorant prejiduce.
Second you can thank almost all negative repor of pot on Ronald Reagan, who after taking office squashed Carters efforts to legalize pot and declare a 'War on drugs'!!
Thirdly studies are often flawed and left to interpretation by who is funding the fucking study. Just think about all the 'studies' cigarette company's did to show that nicotine was niether addictive or harmfull.
I would rather support pot smokers:
They don't start fights for no reason in bars
They don't steel to buy more due to addiction
And I would much rather share the roads with someone who is high rather than someone who's been drinkinig
Tired of bitch ass non-smokers who spout bullshit and govt. propaganda about weed simply b/c they dont do it themselves.
- Marijuana is not physically addictive
- You cant OD from weed
- Marijuana doesnt affect driving
- Marijuana has many medical uses (for AIDS, MS, Glaucoma, Nausea, Anorexia, stress reduction, pain killing, etc.)
- No permanent effects on the brain
Pussies like Satch really need to get their facts straight. The positive evidence far outweighs the tiny negatives it has.
Aside from its legality, there is virtually nothing wrong with blowing a little herb.
- Marijuana is not physically addictive
- You cant OD from weed
- Marijuana doesnt affect driving
- Marijuana has many medical uses (for AIDS, MS, Glaucoma, Nausea, Anorexia, stress reduction, pain killing, etc.)
- No permanent effects on the brain
Pussies like Satch really need to get their facts straight. The positive evidence far outweighs the tiny negatives it has.
Aside from its legality, there is virtually nothing wrong with blowing a little herb.
ok beefy bull you are so right, I cant believe I even doubted you. Oh and jungle rizla, dont accuse someone of being a mass consumer of alcohol if you dont know them. This whole thread is stupid. I just thought I would share some of my personal experience and what I know from others that actually ARE chronics. Nothing wrong with smoking a little here and there but the fact of the matter is that too much of anything is bad for you. Even MARY JANE.
So beefy bull go light a nice fatty and then go to the gym.
So beefy bull go light a nice fatty and then go to the gym.
chanmanfoo
New member
shrek1971 said:
i think what you mean shrek is that weed gave YOU gyno. also i never called you ignorant, your statements say it all. your correct about one thing though, this thread is dumb.
foo
Dark_Stalker
New member
Weed doesn't cause gyno and ISN'T physically addictive period.
Enough said.
Enough said.
BeefyBull said:
Dunno about that. In my exp, you can get pretty adept at driving when high but that doesn't mean you aren't distracted or have the same concentration and quick wittedness you'd have when sober. Sometimes it's that little edge that keeps you out of an accident. That said, driving dead tired is probably worse than driving high. Driving drunk is worst of all. That's the farthest extreme of the 'stupid' scale.
ArmaniMark
New member
Sinistar said:
Didn't Arnold smoke weed...... He had a pretty decent physique...
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