I had read an article that talked about how Var was 72% less liver toxic than other oral steroids even though it had the same structure to it...is this false also? Just wondering, I plan to run full liver support, actually erring on the side of caution. TUDCA, NAC, Chaga Root Extract, Glutathione, Water with fresh squeezed lemon juice every morning upon waking, Milk Thistle, Hawthorn, probably a liver support supplement, Matcha tea times a day, Dandelion root, Tumeric, etc...probably will also do oil pulling which is an Ayurvedic method of detoxifying the body and one which I used for many months with good results.
I have never done any AAS before and am petrified of the liver sides, so I plan on doing way more than what is considered necessary to protect my liver. I don't drink at all and try and eat tons of veggies and other high anti-oxidant foods.
Here is the reference to Anavar being less toxic to the liver based on 1/3 of it being unprocessed by the liver and excreted in the urine:
"Anavar is also a 17alpha alkylated oral steroid, carrying an alteration that is noted for putting stress on the liver. It is important to point out however that to spite this alteration oxandrolone is generally very well tolerated. While liver enzyme tests will occasionally show elevated values, actual damage due to this steroid is not a statistical problem.
Bio-Technology General states that oxandrolone is not as extensively metabolized by the liver as other l7aa orals are; evidenced by the fact that nearly a third of the compound is still intact when excreted in the urine. This may have to do with the understood milder nature of this agent (compared to other l7aa orals) in terms of hepatotoxicity. One study comparing the effects of oxandrolone to other agents including methyltestosterone, norethandrolone, fluoxymesterone and methAndriol clearly supports this notion. Here it was demonstrated that oxandrolone causes the lowest sulfobromophthalein (BSP; a marker of liver stress) retention among all the alkylated orals tested.
20mg of oxandrolone in fact produced 72% less BSP retention than an equal dosage of fluoxyrnesterone, which is a considerable difference being that they possess the same liver-toxic alteration. With such findings, combined with the fact that athletes rarely report trouble with this drug, most feel comfortable believing it to be much safer to use during longer cycles than most of other orals with this distinction. Although this may very well be true, the chance of liver damage still cannot be excluded however."
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