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As athletes, we are most concerned with the "PT" part of the HPTA. "P" being the pituitary and "T" being the Testes. To review, our hormonal responses are based mainly on negative feedback. For example, supragenetic levels of Testosterone or any AAS will signal the pituitary to stop secreting LH and will signal the hypothalamus to stop secreting gonadotropin-releasing hormone (GRH). So, during an AAS cycle, we experience low, natural Testos levels, a reduction in testuclar mass, low LH, and low GRH. The goal of PCT (recovery) is to get the HPTA back to normal.
The most important aspect of recovery is getting testicular mass back to normal as quickly as possible. There is only one drug that will do this and do it quickly--HCG. HCG imitates LH (which is suppressed). HCG acts independently of the HPTA suppression and independent of the meds from the AAS cycle. In this situation, the only side effect we need to worry about is the return of estrogen to normal levels (estrogen rebound). Since estrogen is already at very low levels (the athlete used an aromatase inhibitor during his AAS cycle), Nolvadex is sufficient to block the onrush. By the time the athlete is using nolvadex-only, his testes are up to their normal size. And the pituitary begins to release its own LH.
The most important aspect of recovery is getting testicular mass back to normal as quickly as possible. There is only one drug that will do this and do it quickly--HCG. HCG imitates LH (which is suppressed). HCG acts independently of the HPTA suppression and independent of the meds from the AAS cycle. In this situation, the only side effect we need to worry about is the return of estrogen to normal levels (estrogen rebound). Since estrogen is already at very low levels (the athlete used an aromatase inhibitor during his AAS cycle), Nolvadex is sufficient to block the onrush. By the time the athlete is using nolvadex-only, his testes are up to their normal size. And the pituitary begins to release its own LH.
