If how harsh an oral is to the liver is based on it's mg contents then ...

[Athernigy]

... how come people don't shit out their livers when taking 500mg tylenols? Maybe I am misunderstanding something by thinking this?

-sk

 

[nice6pac]

tylenol doesn not hurt he liver per se, it inhibits enzyme s that help the body. also everytthing is specific that is some work differntly and have other ffectg swhihc contribute aka winstrol has neurological effects whihc cna cause depression

 

[Athernigy]

tylenol doesn not hurt he liver per se, it inhibits enzyme s that help the body. also everytthing is specific that is some work differntly and have other ffectg swhihc contribute aka winstrol has neurological effects whihc cna cause depression

Tylenol gets processed through the liver, it is mildly liver toxic ... I don't get why it isn't more toxic then anadrol for ex. since it has more mg contents.

-sk

 

[thx9000]

Tylenol is hepatoxic at 10 - 15 grams. That's 30 of those 500mg pills. 17-AA's are hepatoxic at somewhat lower mg doseages....

 

[ZeroCool]

... how come people don't shit out their livers when taking 500mg tylenols? Maybe I am misunderstanding something by thinking this?

-sk

Not a bad question to ask. Tylenol has an active ingredient named Acetaminophen. That is what is in a 500mg Tylenol. Tylenol is actually not to hard on the liver although it does pass through the liver. However think about if you took 500 mg of D-bol. You would probably start suffering from coughing up blood, having trouble urinating. It probably would even cause internal bleeding of the stomach. the difference is the chemical structure and balance of each ingredient. Certain pharm. drugs that are prescribed or even over the counter have diferent chemical structure that make the actual compound. When we buy products we buy the trade name. I could be wrong and would like to see a few more posts on this subject but what I do know is there are several different trade names for amny different types of gear.

 

[BullGod]

I think the answer is due to the fact that in order for oral anabolics to get into the blood - they must pass thru the liver. Scientitst added a particular molecule (17-AA) to the drug in order to make sure the drug is able to make it through this "first pass" of the liver without being rendered ineffective. The problem is this 17-AA molecule creates a very heavy burden on the liver. The liver must keep grinding through this 17-AA molecule in order to eventually clear the drug.

On the other hand, acetomeniphine (Tylenol) does not require this molecule. However, the drug itsef has proven to be mildy hepatoxic.

Can't really compare the two because they are hepatoxic for different reasons - bottom line 17-AA orals are highly hepatoxic, where as Tylenol is very mildly hepatoxic.

 

[Mike P.T.]

I think the answer is due to the fact that in order for oral anabolics to get into the blood - they must pass thru the liver. Scientitst added a particular molecule (17-AA) to the drug in order to make sure the drug is able to make it through this "first pass" of the liver without being rendered ineffective. The problem is this 17-AA molecule creates a very heavy burden on the liver. The liver must keep grinding through this 17-AA molecule in order to eventually clear the drug.

On the other hand, acetomeniphine (Tylenol) does not require this molecule. However, the drug itsef has proven to be mildy hepatoxic.

Can't really compare the two because they are hepatoxic for different reasons - bottom line 17-AA orals are highly hepatoxic, where as Tylenol is very mildly hepatoxic.

Agreed. Same thing with why d-bol is milder than even halotestin for example. Not just for it's mg content but because of other things present that make halo more harsh than d-bol.

 

[Mike P.T.]

Agreed. Same thing with why d-bol is milder than even halotestin for example. Not just due to it's mg content but because of other things present that make halo more harsh than d-bol.