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Author
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Topic: ? for LALA
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Vanguard
Pro Bodybuilder
(Total posts: 232)
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posted August 10, 2000 02:32 PM
  
Where would I find info on the relation of ketosis and aspartame? ThanksIP: Logged |
NYyankee
Amateur Bodybuilder
(Total posts: 91)
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posted August 10, 2000 07:22 PM
asparteme raises insulin levels and kicks you out of ketosis. ------------------
" I only train when I'm insane,love to feel the pains and see the gains " IP: Logged |
NYyankee
Amateur Bodybuilder
(Total posts: 91)
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posted August 10, 2000 07:29 PM
Hope that is the info you were looking for Vanguard, besides that I really dont know where else to lead you. ------------------
" I only train when I'm insane,love to feel the pains and see the gains " IP: Logged |
MS
Pro Bodybuilder
(Total posts: 675)
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posted August 10, 2000 08:49 PM
I don't know where some of these urban myths come from, but every medline abstract I've looked at so far concludes that aspartame does not have a significant effect on insulin levels.Here's 3 abstracts from the top of the pile....
1)
BACKGROUND: Neurobehavioral symptoms have been reported
anecdotally with aspartame. OBJECTIVE: This study sought to determine
whether aspartame can disrupt cognitive, neurophysiologic, or behavioral
functioning in normal individuals. DESIGN: Forty-eight healthy volunteers
completed a randomized, double-blind, placebo-controlled, crossover study.
The first month was aspartame free. Subjects then consumed sodas and
capsules with placebo, aspartame, or sucrose for 20 d each. Order was
randomized and subjects were assigned to either a high- (45 mg x kg body
wt(-1) x d(-1)) or low- (15 mg x kg body wt(-1) x d(-1)) dose aspartame
group. Neuropsychologic and laboratory testing was done on day 10 of each
treatment period to determine possible acute effects and on day 20 for
possible chronic effects. RESULTS: Plasma phenylalanine concentrations
increased significantly during aspartame treatment. Neuropsychologic
results; adverse experiences; amino acid, insulin, and glucose values; and
electroencephalograms were compared by sex and by treatment. No
significant differences were found for any dependent measure.
CONCLUSION: Large daily doses of aspartame had no effect on
neuropsychologic, neurophysiologic, or behavioral functioning in healthy
young adults
2) The sweet taste of nonnutritive sweeteners has been reported to increase
hunger and food intake through the mechanism of cephalic-phase insulin
release (CPIR). We investigated the effect of oral sensation of sweetness
on CPIR and other indexes associated with glucose metabolism using
nutritive and nonnutritive sweetened tablets as stimuli. At lunchtime, 12
normal-weight men sucked for 5 min a sucrose, an aspartame-polydextrose,
or an unsweetened polydextrose tablet (3 g) with no added flavor. The three
stimuli were administered in a counterbalanced order, each on a separate
day at 1-wk intervals. Blood was drawn continuously for 45 min before and
25 min after the beginning of sucking and samples were collected at 1-min
intervals. Spontaneous oscillations in glucose, insulin, and glucagon
concentrations were assessed as were increments (slopes) of fatty acid
concentrations during the baseline period. The nature of the baseline
(oscillations: glucose, insulin, and glucagon; and slopes: fatty acids) was
taken into account in the analyses of postexposure events. No CPIR and no
significant effect on plasma glucagon or fatty acid concentrations were
observed after the three stimuli. However, there was a significant decrease
in plasma glucose and insulin after all three stimuli. Only the consumption
of the sucrose tablet was followed by a postabsorptive increase in plasma
glucose and insulin concentrations starting 17 and 19 min, respectively,
after the beginning of sucking. In conclusion, this study suggested that oral
stimulation provided by sweet nonflavored tablets is not sufficient for
inducing CPIR. 3) To determine whether sweet-tasting solutions are effective elicitors of
cephalic phase insulin release (CPIR) in humans, two studies were
conducted using nutritive and nonnutritive sweeteners as stimuli. Normal
weight men sipped and spit four different solutions: water, aspartame,
saccharin, and sucrose. A fifth condition involved a modified sham-feed with
apple pie. The five stimuli were administered in counterbalanced order,
each on a separate day. In study 1, subjects tasted the stimuli for 1 min (n =
15) and in study 2 (n = 16), they tasted the stimuli for 3 min. Arterialized
venous blood was drawn to establish a baseline and then at 1 min
poststimulus, followed by every 2 min for 15 min and then every 5 min for
15 min. In both study 1 and study 2, no significant increases in plasma
insulin were observed after subjects tasted the sweetened solutions. In
contrast, significant increases in plasma insulin occurred after the modified
sham-feed with both the 1 min and 3 min exposure. These results suggest
that nutritive and nonnutritive sweeteners in solution are not adequate
stimuli for the elicitation of CPIR.
IP: Logged |
NYyankee
Amateur Bodybuilder
(Total posts: 91)
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posted August 10, 2000 09:38 PM
When I was a ketogenic diet and had a few cans of diet coke, it did kick me out of ketosis. Every person is different, which of course my sensitive body is.------------------
" I only train when I'm insane,love to feel the pains and see the gains " IP: Logged |
MR. BMJ
Pro Bodybuilder
(Total posts: 123)
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posted August 11, 2000 01:15 AM
I am one of those people too that believe aspartame in diet cola does not raise insulin levels. There was a post a little down the board named "Creatine during the week on a CKD" and Cockdezl gave a reply explaining that he feels it may be the "hydration" effect of the drinks which may be effecting the readings on the ketostix. This may be something to think about. As far as sweetness (by artificial sweetners/any food) being a factor for raising insulin, I have never seen any study relating to this assumption. Therefore, I agree with MS on her reply also.IP: Logged |
Vanguard
Pro Bodybuilder
(Total posts: 232)
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posted August 11, 2000 06:01 AM
Thanks for the help.IP: Logged |
Decaman
Pro Bodybuilder
(Total posts: 862)
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posted August 11, 2000 10:54 AM
Hey ms , your alive Bro? I emailed you awhile back, asking for your vast knowledge, never heard back though.------------------
DECA-IT'S WHAT'S FOR DINNER IP: Logged |
MS
Pro Bodybuilder
(Total posts: 675)
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posted August 12, 2000 07:23 PM
Decaman, email me again. I don't think I got it (might have been when Hushmail was messing around with everything).Anyway, I find this an interesting topic, and it's possible that a state of ketosis could change one's response to Asparatame. But I feel, especially since diet coke is a good diuretic, that any lessening of ketosis as measured by ketostix is more likely to be related to diuresis diluting the measurable ketones in urine. You would probably get a similar result if you tripled your intake of water over a 24 hour period too. Just an opinion. A humble one at that IP: Logged |
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