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I will not quote a study since you want scientific proof:

"progestagenic activity was detected and measured in T, DHT and three synthetic androgens: nandrolone (19-nortestosterone). 7 alpha-methyl 19-nortestosterone (MENT) and mibolerone (7 alpha, 17 alpha-dimethyl 19-nortestosterone) (DMNT). While progestagenic effects of T and DHT required relatively high concentrations (microM levels), the synthetic androgens stimulated AP activity at nM or pM levels. These effects seem to be mediated by the progesterone receptor (PR), since they are completely abolished by the antiprogestins RU-486, ZK-98299 and ZK-112993, but not by the antiandrogen OHFl"

That just said T & DHT are no where near as progestagenic as 19-nortest which is nandrolone. Deca is nandrolone deconate.

SECOND--THIS IS WHAT I GOT FROM THAT. ITS THEIR BUT IS NOT ACTIVE, IF THIS IS SO WHAT MAKES IT ACTIVE IF EVEN POSSIBLE.

ONE LAST QUESTION??

Nolvadex is an anti-estrogen, so by itself it will not de-activate the progestagenic activity of nandrolone, since it is unlikely that it binds to the prog receptor. However, the complex interaction between nandrolone and estrogen metabolism in the breast is unknown. Therefore, anti-estrogens may have some effect in reducing feminization symptoms caused by the use of nandrolone or other progestagens. Also, it would be a very wise choice if estrogens or aromatizable substances are also being used.

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Next, just a few of the HUNDREDS of research papers showing Nortestosterone and it's derivatives are progestenic:

Abstract
To explore the regulatory mechanism at the critical period of the luteal-placental shift, the effects of various steroids and peptides on the production of progesterone by placental explants at 7-10 weeks were studied..������19-Nortestosterone (1-18 mumol) stimulated progesterone production 10- to 100-fold������������������� Thus, of all the compounds tested, only 19-nortestosterone stimulated progesterone production ������.. 19-Nortestosterone was found to be less efficiently aromatized compared to other androgens. These observations suggest that C18 and C19 steroids may be important in the regulation of progesterone ���������..

And why the bros tell you to also take some winny:

Abstract
������������Binding of stanozolol to both cell types could be partially displaced by progesterone, indicating that stanozolol binds to the progesterone receptor������������ Nortestosterone, had no effect on binding of stanozolol to either cell type�����These results indicate that the inhibition of DNA synthesis by stanozolol is elicited through the progesterone receptor���������

Abstract
Testosterone and 19-nortestosterone derivatives were evaluated in a developmental scheme designed to identify
competitive progesterone antagonists������� Compounds that displayed significant binding to the rabbit uterine progesterone receptor were followed in biologic tests for progestational, antiprogestational, and
abortifacient activities. Of the seven compounds tested for both progestational and antiprogestational activity, only
one, 5 alpha-dihydronorethindrone, behaved exclusively as an antagonist. Five other 19-nortestosterones
(19-nortestosterone, 17 beta-hydroxyestra-4, 9(10)-dien-3-one, norethindrone, norethindrone acetate, and R 2323)
proved to be mixed agonists/antagonists������������

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Those studies that MS posted were done on female rabbits and other female animals.

Finally the dosage used on these animals exceeds 1000mg of Deca per week for 7-10 weeks so you of course adverse sid effects appear when you abuse it.

Use your heads guys!!