Clenbuterol:

Brand Names: Broncodil, Broncoterol, Cesbron, Clenasma, Clenbuter.Pharmachim, Contrasmina,
Contraspasmina, Monores, Novegam, Oxyflux, Prontovent, Spiropent, Ventolase, Ventapulmin,

Description: Is available in 10 - 20 mcg tablets or in the .016 mg/gram Ventapulmin Vet variety.
Clenbuterol is known as a sympathomimetic. These hormones are taken to mimic adrenaline and
noradrenaline in the human body. Clenbuterol is a selective beta-2 agonist that is used to stimulate
the beta-receptors in fat and muscle tissue in the body. Clenbuterol exhibits most of its effects on
the stimulation of both type 2 and 3 beta-receptors. Clenbuterol is really one of bodybuilding's most
misunderstood performance enhancement drugs. It is true that it is effective in helping to burn
bodyfat but it is often been stated that clenbuterol is effective in causing anabolic gains and has in
times even been compared to some of the weaker anabolic *******s. Books such as the World
Anabolic Review, 1996, by P. Grunding and M. Bachmann state incorrectly that, "its effects,
however, can by all means be compared to those of *******s. Similar to a combination of Winstrol
Depot and Oxandrolone...." These statements are inaccurate and misleading to say the least. A lot
of these claims as to the anabolic effects of clenbuterol are derived from studying the effects of
clenbuterol on livestock. Clenbuterol is effective in increasing muscle mass and decreasing fat loss
in animals.

The problem with the variation in anabolic effects between humans and livestock is that livestock
have an abundance of the type 3 beta receptors whereas humans have little if any of the type 3 beta
receptors. These beta-3 receptors increases insulin secretion and sensitivity, causing more glucose
and amino acids to be transported into skeletal muscle thus causing the anabolic effects that we,
humans, just aren't seeing. As Dan Duchaine stated in his Muscle Media article on clenbuterol, "In
those animal research studies showing an anabolic effect from clenbuterol, it's my guess the
anabolism happens specifically when the beta2 receptor stops working. At that point, the beta3
increases and causes the anabolic effect through insulin mechanisms." Since humans, again, have
either very little or no beta-3 receptors, there is no chance of this anabolic effect. Just another of
the studies where everyone assumed that what works in animals must work in humans. This is just
simply not the case with clenbuterol.

Clenbuterol does work effectively as a fat burner though. It does this by slight increases in the body
temperature. With each degree that the temperature in your body is raised from the use of
clenbuterol, you will burn up approximately an extra 5% of maintenance calories. This makes it
effective as a fat burner. Your body will fight this by cutting down on the amount of active thyroid in
the body as well as through beta-receptor down regulation, which explains why you only have a
limited effective period to take clenbuterol. While I am on the subject of beta-receptor down
regulation, I would like to dispose of another myth. This involves the two on/two off cycling theory
that I believe was originated by Bill Phillips in the Anabolic Reference Guide and has somehow
made it's was into every other ******* book since then including the WAR and Physical
Enhancement with an Edge. The two on-two off theory simply will not work because of one main
reason: the half life of clenbuterol. This 2-on/2-off idea was a THEORY ONLY, not by a doctor or
scientist, and not based on specific knowledge of clenbuterol, but derived by imitation from other
drug's with shorter half lives.

Clenbuterol has been reported as having a half life of about 2 days, but that is not actually correct,
since it has biphasic elimination, with the half-life of the rapid phase being about 10 hours, and the
slower phase being several days. Supposedly, this is one of the reasons the FDA never approved
clenbuterol as an anti-asthmatic drug...the FDA frowns on drugs with long half-lives if drugs with
more normal half-lives are available. So with a 2-on/2-off cycle you never have time to get enough of
the clenbuterol out of your system for this theory to be reasonable. In actuality, it probably hasn't
even dropped to 50% of your peak concentration before you are taking the drug again. With this all
taken into account, there is no reason to think that this cycling would significantly reduce the
problem of receptor desensitization. A more reasonable approach would be either one week on, one
week off, or alternately, two weeks on two weeks off. The two week cycle has the disadvantage of a
"crash" period afterwards. This crash period can be helped with the use of ephedrine to lessen the
lethargy that you will experience.

If you are interested in taking clenbuterol for anything other than fat loss then you might as well
stay away from this compound. There is a lot of talk as to how clenbuterol compares to ephedrine
as well. Most "experts" feel that clen gives a better bang for the buck than the ECA stack. It should
be noted that clenbuterol's results and effects are much shorter lived. They work through very
similar mechanisms. Both products stimulate the beta-receptors but clenbuterol seems to be a
more refined version, called a second generation beta-agonist drug, than ephedrine. Clenbuterol
targets the proper receptors, being the beta-2 and 3 receptors than ephedrine more specifically
which should in theory make clenbuterol more effective of a fat burner. Again, most of the so called
"experts" say that clenbuterol is more effective than ephedrine. I, personally, get worse results with
clen vs. the good old ECA stack. Clenbuterol also didn't blunt my hunger either and I ate more while
taking it as well. I also seem to get much better effects out of cytomel as a fat burner as well. Even
better than the ECA stack or clenbuterol. But, again, that is my personal opinion.

Effective Dose: 80-140 mcgs. / day in split doses throughout the day. Anything over 140 mcg a day
is overkill since the beta receptors can only take so much of a product and then more is just
wasteful.

Street Price: $.50 - 1.00 / tab. Fairly inexpensive in Mexico though. Spiropent is currently going for
about $7.50/box, Novegam for $5.25/box, and Oxyflux for about $3.30/box.

Stacking Info: One week on, one week off might make sense, or alternately, two weeks on two
weeks off makes sense but has the disadvantage of a "crash" period afterwards. You can take
ephedrine after the clen to help reduce this "crash" period or at least make it more bearable for you.
The two on/two off theory is absolute bullshit and can't work; read above.