The natural decline in DHEA levels accompany a number of events related to both disease and aging which effect virtually every system in our body. A partial list of such processes includes:

decreased bone density and the development of osteoporosis
decreased muscle mass and strength losses
atherosclerosis and cardiovascular disease
decreased insulin sensitivity and the development of diabetes
declines in mental acuity and organic brain deterioration including senile dementia, Alzheimer's Disease and Parkinson's Disease
metabolic slowing with accompanying decreases in energy levels
increased percentage body fat
decreased immune function and the development of malignancy, autoimmune disorders, and increased susceptibility to both bacterial and viral illnesses
decreased libido
psychiatric problems including depression.
The biggest concern surrounding over-the-counter hormone replacement therapy is that consumers will enjoy the effects of small doses and self-medicate with more than what is recommended, eventually crippling their body's natural capacity to produce the hormone. This is of particular importance to individuals in their 20's and 30's who are presumably producing adequate quantities of the hormone without supplementation. In addition, DHEA can be converted to testosterone in women and could theoretically promote virilizing effects like acne, increased facial hair, male-pattern baldness, and voice deepening.

Despite 2500 scientific studies spanning the past two decades, no one really knows what constitutes the proper age-related replacement dose, or how this dose should differ between men and women, or simply between physiologically divergent individuals. Most health professionals feel that more scientific studies are required to establish better replacement guidelines and advise taking the supplement only under the supervision of a physician.

FROM http://www.naples.net/~nfn03605/testslee.htm

Testosterone causes the genes of "testosterone target tissues" to absorb extra DHEA for their functions. For example, this is why men have larger, more powerful muscles than women. So, the increased testosterone of men reduces the availability of DHEA, therefore, I suggest sleep apneas occur more in men because of reduced availability of DHEA for proper nervous stimulation. According to my theory of DHEA function, all tissues compete for DHEA. Therefore, in a man of low DHEA, administering testosterone as in the report above, will cause some tissues to absorb extra DHEA and reduce already low DHEA. This may cause either the nervous stimulation of the airways to decline, resulting in a collapsed airway, or it may reduce the DHEA so low that the brainstem ceases to function momentarily, as in SIDS. This situation would produce the apnea in which the airways do not collapse. Of course, the brainstem malfunction and airway collapse could occur simultaneously. This could explain the variability of effects of administration of testosterone in the quotation, above. Some of the men may be hypogonadal because of a poorly functioning pituitary-adrenal-gonadal axis, caused by low DHEA with low testosterone as a secondary consequence. Some of the men may have simply not produced much testosterone along with ample amounts of DHEA. This second group would represent those individuals in whom testosterone administration had no effect on sleep at all.

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