What I could not find is if Nandrolone has only progestogenic effect or if it can also convert into estrogens.

Only one post said that a steroid can not have progestogenic effects and at the same time convert to estrogens, is it true?

Oxymetholone estro and progesto
Nandrolones estro and progesto
Trenbolones progesto
Nilevar estro and progesto
Methandienone estro
Boldenone estro
DHT progesto? (not sure)
Testosterones estro

NO PROBLEMS WITH: Stanozolol, Oxandrolone and Primobolan.

quote:

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Originally posted by HUCKLEBERRY FINNaplex:
WINSTROL HAS BEEN SHOWN TO BIND AGGRESSIVELY TO THE P/R IN MANY STUDIES,YET IT IS NOT PROGESTOGENIC...THEREFORE,IF IT'S OCCUPYING P/R'S,BUT NOT ACTIVATING THEM,WHAT ON EARTH DO YOU THINK IT'S DOING?IT'S PREVENTING PROGESTIN'S FROM BINDING TO AND ACTIVATING THEM("BLOCKING THEM")...

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Nandrolone, popularly known as Deca, is a classified as a progestin. Deca derives many of its benefits from its progestenic nature: including, but not limited to, increase IM fat storage and increased fluid retention in the joints from glucocorticoid(GC) stimulation.

Deca is the most widely used form of prescription contraception in the first world. Deca is superior to testosterone as a form of birth control because its progestenic effects which result in rapid onset of azoospermia. Progestins are used similarly in women, progestins given to women in birth control pills and other drugs such as norgestrel and norethidrone are classified as 19-nor-testosterone or 19 nor- progesterone derivatives. Natural progesterone plays an important role in sexual arousal- affecting GABA to a considerable extent. The addition of progestins like deca which compete with progesterone and decrease its production may result in drastically reduced sexual arousal. Interestingly enough, the chemical castration of sex offenders, is acheived through the use of a 19-nortestosterone derivative.

This brings us to the second most common problem with the use of progestenic drugs like Deca, the breast tissue has both PR(progesterone receptors) and ER(estrogen receptors) and stimulation of either will result in new tissue formation and growth. This will vary considerably from individual to individuals based on the numbers and ratio of receptors in the tissue. Some individuals have more PR, which will make them more susceptable to Gyno. Another suspected factor is that there are slightly physiologically different PR, as well as ER and AR, which may effect binding and expression of synthetic progestins either positively or negatively.

The use of Anti-estrogens and Aromatase-inhibitors will help by reducing stimulation of the ER in the breast tissue. However, those with high concentrations of PR or PR whose physiology allows for greater binding or expression of progestins will be faced with developing Gynomacastia.

In short

1. DECA dick is real

2. DECA does cause Gyno

3. DECA is progestin it must be fought with anti-progestins

4. Use of Nolvadex and Arimidex will help, but only by reducing ER stimulation.

Oxymetholone does not aromatize. Both Bill Roberts and Pat Arnold have stated that they can find no mechanism by which it could aromatize...I have to take their educated statements as fact, and also, i could find no research showing estrogen formation with oxy use.

"Nandrolones estro and progesto"

Correct

"Trenbolones progesto"

Plausable, although one study shows no progestagenic activity.

"Nilevar estro and progesto
Methandienone estro
Boldenone estro"

Correct

"DHT progesto? (not sure)"

No, no aromatization and no progestagenic activity.

"Testosterones estro"

Correct