SPATTS follow up on MU question

[wilson6]

SPATTS,

I checked with a colleague of mine who wrote part of his general exams in 94 on MUs and the neuromuscular junction, AChRs, etc.

He said at that time, and to his knowledge currently (no major new recent lit), that training per se in otherwise normal subjects has no effect on ACh receptors or ACh binding, etc. That part of the system appears to function optimally regardless of training status. Now, if there were nerve injury, disease, etc. there would be changes, but nothing related to training per se in healthy younger subjects.

W6

 

[wilson6]

Stress is more likely to play more of a central vs peripheral role. Never heard of stress per se limiting Ach release, binding or affecting any of receptor subunits or channel function.

Not sure what you're searching for here, but even if there was an issue, the only way to solve it would be to back off training as you would for generalized OT.

Everyone has their genetic limit and at some point further neurologic and hypertrophic adaptations in advance trainees is no longer possible. Then only anabolic agents will be of value. See Sale, DG. Neural adaptation to resistance exercise. MSSE 20: Suppl, S135-S145, 1988.

W6

 

[wilson6]

Neural adaptations occur in the first few weeks of training, then hypertrophy is the predominant mechanism for strength gains. Sure there are continued neuro adaptations (i.e., overcoming GTO inhibition and antagonist contractions, etc.), but to suggest that any strength sport is primarily CNS makes no sense to me.

If it were all CNS, then why is there muscle hypertrophy? and why does one lose strength with muscle mass loss if it is all CNS? If it were all CNS, then powerlifters would look like distance runners, but a lot stronger.

Most important factors; a well balanced diet that includes all necessary aminos, vitamins and minerals, enough energy and water.

There isn't anything that is going to supercharge your CNS long-term beyond a good diet, and adequate sleep and restoration.

W6

 

[wilson6]

There will always be some loss of contractile tissue with modest dieting. Some if you're natural, little if any if you're not. More serious dieting will increase muscle loss regardless of what you're doing.

Dropping 4% bodyfat should have little impact on strength as long as it is done slowly and the carbs (muscle and liver glycogen) are there for lifting days.

I might add some additional BCAAs into your supplement regiment as well as perhaps some tyrosine, although the latest research doesn't support a central ergogenic effect for tyrosine, under increased demand, it might help.

Give it try and see what happens.

W6

 

[spatts]

W6....CNS RECOVERY. This is the lastest topic.

Same rules as above? If a motor unit IS muscle and nerve, then wouldn't things like active recovery, and other recovery techniques for the muscles, aid in nerve recovery?

I'm just guessing here...kinda looking for your professional opinion on the matter.