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Bromocriptine and Prolactin levels while using Tren

blaster220

New member
Now, it has been established that bromocriptine can lower prolactin and GH in some cases... Is the possible reduction in GH directly related to the below normal levels of prolactin present?

I can understand how taking bromocriptine alone, without a compound that increases prolactin levels such as Tren (due to decreased T3 levels) could cause less than normal prolactin levels to cause a reduction in GH prodution.

However, wouldn't it be sufficient to say that since Tren increases prolactin levels and bromocriptine decreases prolactin levels, that one could avoid any GH suppression AND prolactin induced gyno with the correct dosage of bromo due to the fact prolactin levels would be close to normal for a male?

Any thoughts?
 
Last edited:
http://boards.elitefitness.com/forum/showthread.php?threadid=41453&highlight=bromocriptine

A couple of replies in the above thread note that bromocriptine alone could lower prolactin levels below normal to the point of supressing GH. To combat, they recommend a combo of GHB + Bromo for increased GH production without prolactin sides because the "up prolactin" of GHB and the "down prolactin" of bromo basically negate eachothers prolactin effects to keep good GH levels with a normal level of prolactin....

So, I was wondering that if you used Tren (which will raise prolactin due to reduced free t3/t4) and bromocriptine (which will lower the release of prolactin) that you might find your happy medium of normal prolactin levels therefore eliminating prolactin induced gyno and suppressed GH levels. Any thoughts?
 
You shouldn't need to throw GHB into the mix- too much of an unknown as far as prolactin. The prolactin lowering effect will last for 24 hours after a dose, but the GH lowering effect only last 4-8 hours. So take it no less than 8 hours before you go to bed like around noon to avoid decreasing the GH increase you get naturally when you go to sleep. If you get insomnia, take it earlier in the day. One more thing, I hope you have a girlfriend. This stuff really makes it hard to get enough action.
 
Sadist, that's what I was getting at... I wasn't thinking of using GHB + Fina + Bromo... I was just saying if GHB and Bromo are suggested as a combo, then why not Fina + Bromo to bring prolactin levels to normal?

To clarify... am I correct when I presume:

*Fina - lowers free t3/t4 - lowered t3/t4 = increased prolactin
*Bromocriptine - lowers prolactin production

*increase in prolactin + decrease in prolactin = normal prolactin levels

*normal prolactin levels = no gyno and no altered GH levels...

is it that simple or am I just wishing? Fonz?
 
good question. fonz?

would this be ideal

t3 cycle while on fina
bromocriptine while on fina
arimidex to lower igf-1 levels
winstrol to occupy progesterone receptors
fina at 75mg a day

cranberry extract and ala
 
animal studies show a decrease in serum thyroxin but no increase in prolactin.
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Growth hormone, insulin, prolactin and total thyroxine in the plasma of sheep implanted with the anabolic steroid trenbolone acetate alone or with oestradiol.

Donaldson IA, Hart IC, Heitzman RJ.

The mode of action of the anabolic steroid trenbolone acetate (19-norandrost-4,9,11-trien-3-one-17-acetate) was studied through the endogenous hormonal response of castrated male sheep to subcutaneous implantation of 140 mg of trenbolone acetate and 20 mg of oestradiol both separately and in combination. Radioimmunoassay of delta-4,9,11-trienic steroids and oestradiol-17 beta in plasma confirmed that simultaneous administration of trenbolone acetate with oestradiol led to a significantly greater persistence of oestradiol-17 beta residues in plasma (P less than 0.05) than with implantation of oestradiol alone. Oestradiol treatment increased concentrations of growth hormone and insulin (P less than 0.05; P less than 0.001 respectively) in plasma samples collected weekly. Trenbolone acetate by itself had no significant effect and the oestrogenic response was blocked on the simultaneous implantation of trenbolone acetate and oestradiol (despite higher plasma levels of oestradiol-17 beta with this treatment). Plasma total thyroxine was markedly depressed to 45 per cent of its basal level by trenbolone acetate, alone or with oestradiol (P less than 0.001) and depressed to 80 per cent of basal by oestradiol treatment alone (P less than 0.001). Plasma prolactin was unaltered by the above treatments.
 
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